Beate Schlotter

1.1k total citations
12 papers, 598 citations indexed

About

Beate Schlotter is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Beate Schlotter has authored 12 papers receiving a total of 598 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 4 papers in Neurology. Recurrent topics in Beate Schlotter's work include Muscle Physiology and Disorders (3 papers), Hereditary Neurological Disorders (3 papers) and Neurological diseases and metabolism (2 papers). Beate Schlotter is often cited by papers focused on Muscle Physiology and Disorders (3 papers), Hereditary Neurological Disorders (3 papers) and Neurological diseases and metabolism (2 papers). Beate Schlotter collaborates with scholars based in Germany, Australia and United Kingdom. Beate Schlotter's co-authors include D. Pongratz, Hanns Lochmüller, Peter Reilich, Maggie C. Walter, Wolfgang Müller‐Felber, Carl D. Reimers, B. Martin Eicke, Thomas N. Witt, M. Toepfer and Teepu Siddique and has published in prestigious journals such as Neurology, Clinical Chemistry and Journal of the Neurological Sciences.

In The Last Decade

Beate Schlotter

12 papers receiving 576 citations

Peers

Beate Schlotter

NEURO

EPIDE

GENET

CMN

Shiro Matsubara Japan

Keiji Chida Japan

James Miller United Kingdom

Keivan Basiri Iran

T. E. Bertoríni United States

Shlomo Dotan Israel

Robert T. Shebert United States

Cláudia Suemi Kamoi Kay Brazil

N P Cavanagh United Kingdom

S Riku Japan

Shiro Matsubara Japan

Beate Schlotter

239 ×0.9

NEURO

263 ×1.3

201 ×1.5

EPIDE

99 ×0.8

GENET

110 ×1.0

CMN

Citations per year, relative to Beate Schlotter Beate Schlotter (= 1×) peers Shiro Matsubara

Countries citing papers authored by Beate Schlotter

Since Specialization

Citations

This map shows the geographic impact of Beate Schlotter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beate Schlotter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beate Schlotter more than expected).

Fields of papers citing papers by Beate Schlotter

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beate Schlotter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beate Schlotter. The network helps show where Beate Schlotter may publish in the future.

Co-authorship network of co-authors of Beate Schlotter

This figure shows the co-authorship network connecting the top 25 collaborators of Beate Schlotter. A scholar is included among the top collaborators of Beate Schlotter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beate Schlotter. Beate Schlotter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Reilich, Peter, Beate Schlotter, Federica Montagnese, et al.. (2020). Location matters – Genotype-phenotype correlation in LRSAM1 mutations associated with rare Charcot-Marie-Tooth neuropathy CMT2P. Neuromuscular Disorders. 31(2). 123–133. 1 indexed citations

Reimers, C. D., et al.. (2008). Der Einfluß von Muskelarbeit auf das Myosonogramm. Ultraschall in der Medizin - European Journal of Ultrasound. 16(2). 79–83. 3 indexed citations

Hunter, Michael, Rafaëlle Bernard, Elizabeth Freitas, et al.. (2003). Mutation screening of the N-myc downstream-regulated gene 1 (NDRG1) in patients with Charcot-Marie-Tooth Disease. Human Mutation. 22(2). 129–135. 41 indexed citations

Walter, Maggie C., Peter Reilich, R. Kohnen, et al.. (2002). Creatine monohydrate in myotonic dystrophy. Journal of Neurology. 249(12). 1717–1722. 44 indexed citations

Walter, Maggie C., Hanns Lochmüller, Beate Schlotter, et al.. (2001). Neue Gesichtspunkte zur Pathogenese und Therapie der sporadischen Einschlusskörpermyositis (s-IBM). Der Nervenarzt. 72(2). 117–121. 1 indexed citations

Walter, Maggie C., Hanns Lochmüller, M. Toepfer, et al.. (2000). High-dose immunoglobulin therapy in sporadic inclusion body myositis: a double-blind, placebo-controlled study. Journal of Neurology. 247(1). 22–28. 164 indexed citations

Abicht, Angela, Rolf Stucka, Veronika Karcagi, et al.. (1999). A common mutation (ε1267delG) in congenital myasthenic patients of Gypsy ethnic origin. Neurology. 53(7). 1564–1564. 77 indexed citations

Hentati, Afif, Karim Ouahchi, Margaret A. Pericak‐Vance, et al.. (1998). Linkage of a commoner form of recessive amyotrophic lateral sclerosis to chromosome 15q15-q22 markers. Neurogenetics. 2(1). 55–60. 126 indexed citations

Karg, O, et al.. (1998). Nichtinvasive intermittierende Selbstbeatmung (ISB) als Palliativmaßnahme bei amyotropher Lateralsklerose. Der Nervenarzt. 69(12). 1074–1082. 12 indexed citations

10.

Reimers, Carl D., Beate Schlotter, B. Martin Eicke, & Thomas N. Witt. (1996). Calf enlargement in neuromuscular diseases: a quantitative ultrasound study in 350 patients and review of the literature. Journal of the Neurological Sciences. 143(1-2). 46–56. 90 indexed citations

11.

Braun, S., et al.. (1996). Discrepant results for cardiac troponin T and troponin I in chronic myopathy, depending on instrument and assay generation. Clinical Chemistry. 42(12). 2039–2041. 19 indexed citations

12.

Braun, S., et al.. (1996). Discrepant results for cardiac troponin T and troponin I in chronic myopathy, depending on instrument and assay generation.. PubMed. 42(12). 2039–41. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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