Battistina Asproni
- Organic Chemistry top 10%
- Molecular Biology
- Pharmacology top 10%
- Cellular and Molecular Neuroscience
- Computational Theory and Mathematics top 10%
- Co-authors
- Gérard A. PinnaGabriele MurinedduAmedeo PauMorten LanggårdAntonio DoreClaus T. ChristoffersenJan KehlerGianpiero Boatto
- Topics
- Pharmacological Receptor Mechanisms and Effects (19 papers)Cannabis and Cannabinoid Research (14 papers)Neurotransmitter Receptor Influence on Behavior (9 papers)
- Partner nations
- ItalySpainUnited States
In The Last Decade
Battistina Asproni
42 papers receiving 484 citations
Peers
Comparison fields: 5 of 71
- Organic Chemistry 244
- Molecular Biology 205
- Pharmacology 163
- Cellular and Molecular Neuroscience 103
- Computational Theory and Mathematics 36
Countries citing papers authored by Battistina Asproni
This map shows the geographic impact of Battistina Asproni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Battistina Asproni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Battistina Asproni more than expected).
Fields of papers citing papers by Battistina Asproni
This network shows the impact of papers produced by Battistina Asproni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Battistina Asproni. The network helps show where Battistina Asproni may publish in the future.
Co-authorship network of co-authors of Battistina Asproni
This figure shows the co-authorship network connecting the top 25 collaborators of Battistina Asproni. A scholar is included among the top collaborators of Battistina Asproni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Battistina Asproni. Battistina Asproni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 7 | |
| 3 | 17 | |
| 4 | 3 | |
| 5 | 3 | |
| 6 | 4 | |
| 7 | 3 | |
| 8 | 16 | |
| 9 | 7 | |
| 10 | 18 | |
| 11 | 6 | |
| 12 | 17 | |
| 13 | 10 | |
| 14 | 11 | |
| 15 | 8 | |
| 16 | 16 | |
| 17 | 18 | |
| 18 | 5 | |
| 19 | 9 | |
| 20 | 7 |
About Battistina Asproni
Battistina Asproni is a scholar working on Pharmacology, Cellular and Molecular Neuroscience and Toxicology, having authored 44 papers that have together received 495 indexed citations. Recurring topics across this work include Pharmacological Receptor Mechanisms and Effects (19 papers), Cannabis and Cannabinoid Research (14 papers) and Neurotransmitter Receptor Influence on Behavior (9 papers). The work is most often cited by research in Pharmacology (163 citations), Organic Chemistry (244 citations) and Toxicology (22 citations). Battistina Asproni has collaborated with scholars based in Italy, Spain and United States. Frequent co-authors include Gérard A. Pinna, Gabriele Murineddu, Amedeo Pau, Morten Langgård, Antonio Dore, Claus T. Christoffersen, Jan Kehler, Gianpiero Boatto, Riccardo Cerri and Jacob Nielsen. Their work appears in journals such as International Journal of Molecular Sciences, Journal of Medicinal Chemistry and Molecules.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.