Balwinder Sambi

645 citations
10 papers · 559 indexed · h-index 9
Topics
Protein Kinase Regulation and GTPase Signaling (7 papers)Receptor Mechanisms and Signaling (6 papers)Sphingolipid Metabolism and Signaling (4 papers)

In The Last Decade

Balwinder Sambi

10 papers receiving 548 citations

Peers

Balwinder Sambi
Comparison fields: 5 of 56
  • Molecular Biology 499
  • Cell Biology 168
  • Cellular and Molecular Neuroscience 82
  • Physiology 60
  • Immunology 36
Replace Ann-Marie Conway with:
Ann-Marie Conway United Kingdom
Chiharu Yamada Japan
Martin Houle Canada
Brahim Houssa Netherlands
Ruth Zeidman Sweden
Jasmina Profirovic United States
Julie Radeff‐Huang United States
Ursula Brandt Germany
Joseph Hwang United States
Naoto Kakinuma Japan
Balwinder Sambi relative to Ann-Marie Conway United Kingdom Ann-Marie Conway's profile →
Citations per field
00.5×1.5×2.5×
Ann-Marie Conway · 1×
Citations per year

Countries citing papers authored by Balwinder Sambi

Since Specialization
Citations

This map shows the geographic impact of Balwinder Sambi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Balwinder Sambi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Balwinder Sambi more than expected).

Fields of papers citing papers by Balwinder Sambi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Balwinder Sambi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Balwinder Sambi. The network helps show where Balwinder Sambi may publish in the future.

Co-authorship network of co-authors of Balwinder Sambi

This figure shows the co-authorship network connecting the top 25 collaborators of Balwinder Sambi. A scholar is included among the top collaborators of Balwinder Sambi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Balwinder Sambi. Balwinder Sambi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
#WorkIndexed citations
1 34
2 6
3 24
4 118
5 65
6 44
7 29
8 79
9 134
10 26

About Balwinder Sambi

Balwinder Sambi is a scholar working on Cell Biology, Molecular Biology and Hematology, having authored 10 papers that have together received 559 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (7 papers), Receptor Mechanisms and Signaling (6 papers) and Sphingolipid Metabolism and Signaling (4 papers). The work is most often cited by research in Cell Biology (168 citations), Molecular Biology (499 citations) and Cellular and Molecular Neuroscience (82 citations). Balwinder Sambi has collaborated with scholars based in United Kingdom, Australia and United States. Frequent co-authors include Nigel J. Pyne, Susan Pyne, Catherine Waters, Forbes Alderton, Timothy M. Palmer, Kok Choi Kong, Soma Rakhit, Dawn Thompson, Stuart M. Pitson and Rothwelle J. Tate. Their work appears in journals such as Journal of Biological Chemistry, Methods in enzymology on CD-ROM/Methods in enzymology and British Journal of Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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