Atsushi Watanabe
- Molecular Biology top 10%
- Genetics top 5%
- Cell Biology top 5%
- Endocrinology, Diabetes and Metabolism top 5%
- Rheumatology top 5%
- Co-authors
- Takashi ShimadaBarbara PloplisKazuhisa TakedaMasayoshi TachibanaHiko HyakusokuRei OgawaMakoto MigitaHiroshi Mizuno
- Topics
- Alkaline Phosphatase Research Studies (18 papers)Connective tissue disorders research (16 papers)Heterotopic Ossification and Related Conditions (12 papers)
- Cited by
- Sensory SystemsNeurologyCell Biology
- Partner nations
- JapanUnited StatesPoland
In The Last Decade
Atsushi Watanabe
131 papers receiving 2.9k citations
Peers
Comparison fields: 5 of 126
- Molecular Biology 1.1k
- Genetics 438
- Cell Biology 414
- Endocrinology, Diabetes and Metabolism 369
- Rheumatology 349
Countries citing papers authored by Atsushi Watanabe
This map shows the geographic impact of Atsushi Watanabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Atsushi Watanabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Atsushi Watanabe more than expected).
Fields of papers citing papers by Atsushi Watanabe
This network shows the impact of papers produced by Atsushi Watanabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Atsushi Watanabe. The network helps show where Atsushi Watanabe may publish in the future.
Co-authorship network of co-authors of Atsushi Watanabe
This figure shows the co-authorship network connecting the top 25 collaborators of Atsushi Watanabe. A scholar is included among the top collaborators of Atsushi Watanabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Atsushi Watanabe. Atsushi Watanabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 1 | |
| 4 | 6 | |
| 5 | 3 | |
| 6 | 2 | |
| 7 | 2 | |
| 8 | 1 | |
| 9 | 19 | |
| 10 | 3 | |
| 11 | 63 | |
| 12 | 14 | |
| 13 | 8 | |
| 14 | 17 | |
| 15 | 56 | |
| 16 | 6 | |
| 17 | 2 | |
| 18 | 6 | |
| 19 | Analyses of loss-of-function mutations of the MITF gene suggest that haploinsufficiency is a cause of Waardenburg syndrome type 2A. | 86 |
| 20 | 8 |
About Atsushi Watanabe
Atsushi Watanabe is a scholar working on Neurology, Sensory Systems and Rheumatology, having authored 137 papers that have together received 3.0k indexed citations. Recurring topics across this work include Alkaline Phosphatase Research Studies (18 papers), Connective tissue disorders research (16 papers) and Heterotopic Ossification and Related Conditions (12 papers). The work is most often cited by research in Sensory Systems (243 citations), Neurology (320 citations) and Cell Biology (414 citations). Atsushi Watanabe has collaborated with scholars based in Japan, United States and Poland. Frequent co-authors include Takashi Shimada, Barbara Ploplis, Kazuhisa Takeda, Masayoshi Tachibana, Hiko Hyakusoku, Rei Ogawa, Makoto Migita, Hiroshi Mizuno, Hideo Orimo and Tetsuo Ikezono. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Lancet and Nature Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.