Atan Gross

9.6k total citations · 1 hit paper
31 papers, 2.5k citations indexed

About

Atan Gross is a scholar working on Molecular Biology, Hematology and Physiology. According to data from OpenAlex, Atan Gross has authored 31 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 6 papers in Hematology and 6 papers in Physiology. Recurrent topics in Atan Gross's work include Mitochondrial Function and Pathology (20 papers), Cell death mechanisms and regulation (10 papers) and Cancer, Hypoxia, and Metabolism (5 papers). Atan Gross is often cited by papers focused on Mitochondrial Function and Pathology (20 papers), Cell death mechanisms and regulation (10 papers) and Cancer, Hypoxia, and Metabolism (5 papers). Atan Gross collaborates with scholars based in Israel, United States and Canada. Atan Gross's co-authors include Stanley J. Korsmeyer, Vamsi K. Mootha, Tullia Lindsten, Michael C. Wei, Solly Weiler, Mona Ashiya, Amir Bahat, Yehudit Zaltsman, Maria Maryanovich and Tsvee Lapidot and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Genes & Development.

In The Last Decade

Atan Gross

31 papers receiving 2.5k citations

Hit Papers

tBID, a membrane-targeted death ligand, oligomerizes BAK ... 2000 2026 2008 2017 2000 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Atan Gross Israel 19 2.0k 326 324 302 288 31 2.5k
Helin Vakifahmetoglu-Norberg Sweden 15 1.6k 0.8× 348 1.1× 366 1.1× 491 1.6× 257 0.9× 21 2.2k
Jonathan L. Coloff United States 24 1.5k 0.8× 386 1.2× 746 2.3× 391 1.3× 459 1.6× 33 2.4k
Sun‐Il Hwang United States 22 1.8k 0.9× 161 0.5× 304 0.9× 416 1.4× 354 1.2× 44 2.7k
Simone Kühnle Germany 13 1.7k 0.8× 280 0.9× 175 0.5× 307 1.0× 324 1.1× 14 2.5k
Melissa J. Parsons United States 11 1.8k 0.9× 393 1.2× 233 0.7× 418 1.4× 381 1.3× 18 2.3k
Beat Nyfeler Switzerland 15 1.7k 0.9× 523 1.6× 479 1.5× 304 1.0× 176 0.6× 19 2.6k
Yasuyuki Kitaura Japan 24 1.2k 0.6× 179 0.5× 323 1.0× 286 0.9× 258 0.9× 62 2.0k
David Sumpton United Kingdom 22 1.3k 0.6× 143 0.4× 402 1.2× 235 0.8× 374 1.3× 45 2.0k
Justin Kale Canada 13 1.6k 0.8× 262 0.8× 225 0.7× 267 0.9× 329 1.1× 15 2.2k
Joslyn K. Brunelle United States 12 1.4k 0.7× 185 0.6× 646 2.0× 212 0.7× 251 0.9× 13 2.1k

Countries citing papers authored by Atan Gross

Since Specialization
Citations

This map shows the geographic impact of Atan Gross's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Atan Gross with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Atan Gross more than expected).

Fields of papers citing papers by Atan Gross

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Atan Gross. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Atan Gross. The network helps show where Atan Gross may publish in the future.

Co-authorship network of co-authors of Atan Gross

This figure shows the co-authorship network connecting the top 25 collaborators of Atan Gross. A scholar is included among the top collaborators of Atan Gross based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Atan Gross. Atan Gross is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Petucci, Christopher, Hu Wang, Xianlin Han, et al.. (2025). MTCH2 controls energy demand and expenditure to fuel anabolism during adipogenesis. The EMBO Journal. 44(4). 1007–1038. 8 indexed citations
2.
Furer, Victoria, et al.. (2024). Mitochondrial structural alterations in fibromyalgia: a pilot electron microscopy study. Clinical and Experimental Rheumatology. 42(6). 1215–1223. 3 indexed citations
3.
Mullokandov, Michael, et al.. (2023). MTCH2 cooperates with MFN2 and lysophosphatidic acid synthesis to sustain mitochondrial fusion. EMBO Reports. 25(1). 45–67. 13 indexed citations
4.
Filanovsky, Kalman, Michal Haran, Andrei Braester, et al.. (2020). PERPHERAL BLOOD CELL MITOCHONDRIAL DYSFUNCTION IN MYELODYSPLASTIC SYNDROMECAN BE IMPROVED BY A COMBINATION OF COENZYME Q10 AND CARNITINE. Mediterranean Journal of Hematology and Infectious Diseases. 12(1). e2020072–e2020072. 5 indexed citations
5.
Bahat, Amir & Atan Gross. (2019). Mitochondrial plasticity in cell fate regulation. Journal of Biological Chemistry. 294(38). 13852–13863. 106 indexed citations
6.
Jenkins, Alicia J., Imre Lengyel, Guy A. Rutter, et al.. (2018). Obesity, diabetes and zinc: A workshop promoting knowledge and collaboration between the UK and Israel, november 28–30, 2016 – Israel. Journal of Trace Elements in Medicine and Biology. 49. 79–85. 1 indexed citations
7.
Bahat, Amir, Yehudit Zaltsman, Dilshad H. Khan, et al.. (2018). MTCH2-mediated mitochondrial fusion drives exit from naïve pluripotency in embryonic stem cells. Nature Communications. 9(1). 5132–5132. 64 indexed citations
8.
Aloni, Etay, Antonella Ruggiero, Atan Gross, & Menahem Segal. (2018). Learning Deficits in Adult Mitochondria Carrier Homolog 2 Forebrain Knockout Mouse. Neuroscience. 394. 156–163. 5 indexed citations
9.
Filanovsky, Kalman, Michal Haran, Andrei Braester, et al.. (2017). Clinical Benefit and Improvement of Mitochondrial Function in Low Risk Myelodysplastic Syndrome Treated By Combination Ultra Coenzyme Q10 and L-Carnitine. Blood. 130. 1704–1704. 2 indexed citations
11.
Ruggiero, Antonella, Etay Aloni, Eduard Korkotian, et al.. (2017). Loss of forebrain MTCH2 decreases mitochondria motility and calcium handling and impairs hippocampal-dependent cognitive functions. Scientific Reports. 7(1). 44401–44401. 34 indexed citations
12.
Gross, Atan. (2016). BCL-2 family proteins as regulators of mitochondria metabolism. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1857(8). 1243–1246. 116 indexed citations
13.
Kuperman, Yael, Michael Tsoory, Yehudit Zaltsman, et al.. (2016). Loss of Muscle MTCH2 Increases Whole-Body Energy Utilization and Protects from Diet-Induced Obesity. Cell Reports. 14(7). 1602–1610. 43 indexed citations
14.
Weaver, David T., Verónica Eisner, Xingguo Liu, et al.. (2014). Distribution and Apoptotic Function of Outer Membrane Proteins Depend on Mitochondrial Fusion. Molecular Cell. 54(5). 870–878. 48 indexed citations
15.
Zinkel, Sandra S., Xiaoming Yin, & Atan Gross. (2012). Rejuvenating Bi(d)ology. Oncogene. 32(27). 3213–3219. 11 indexed citations
16.
Robinson, Alan J., Edmund R.S. Kunji, & Atan Gross. (2012). Mitochondrial carrier homolog 2 (MTCH2): The recruitment and evolution of a mitochondrial carrier protein to a critical player in apoptosis. Experimental Cell Research. 318(11). 1316–1323. 33 indexed citations
17.
Zaltsman, Yehudit, Maria Maryanovich, Riekelt H. Houtkooper, et al.. (2010). MTCH2/MIMP is a major facilitator of tBID recruitment to mitochondria. Nature Cell Biology. 12(6). 553–562. 162 indexed citations
18.
Tsafriri, Alex, et al.. (2007). Luteinizing Hormone-Induced Caspase Activation in Rat Preovulatory Follicles Is Coupled to Mitochondrial Steroidogenesis. Endocrinology. 148(4). 1717–1726. 35 indexed citations
19.
Gross, Atan. (2001). BCL‐2 Proteins: Regulators of the Mitochondrial Apoptotic Program. IUBMB Life. 52(3-5). 231–236. 67 indexed citations
20.
Gross, Atan, Elizabeth Blachly‐Dyson, Emy Basso, et al.. (2000). Biochemical and Genetic Analysis of the Mitochondrial Response of Yeast to BAX and BCL-X L. Molecular and Cellular Biology. 20(9). 3125–3136. 139 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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