Askale Mathias

614 citations
12 papers · 494 indexed · h-index 11

Impact in

Papers in

    • TGF-β signaling in diseases 3
    • Bone Metabolism and Diseases 2
    • Ion channel regulation and function 1
    • Toxin Mechanisms and Immunotoxins 4

Askale Mathias

12 papers receiving 481 citations

Peers

Askale Mathias
Comparison fields: 5 of 65
  • Biotechnology 117
  • Immunology 161
  • Radiology, Nuclear Medicine and Imaging 115
  • Oncology 136
  • Molecular Biology 246
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F Ghani United States
Ning-Hsing Yeh Taiwan
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Richard Priest United Kingdom
Jennifer L. Bromberg-White United States
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Citations per field
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Citations per year

Countries citing papers authored by Askale Mathias

Since Specialization
Citations

This map shows the geographic impact of Askale Mathias's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Askale Mathias with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Askale Mathias more than expected).

Fields of papers citing papers by Askale Mathias

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Askale Mathias. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Askale Mathias. The network helps show where Askale Mathias may publish in the future.

Co-authors

The 25 scholars most cited alongside Askale Mathias, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Askale Mathias Line = papers co-authored together Askale Mathias links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 201887
2 200283
3 200556
4 200149
5
Expression of transforming growth factor beta ligand and receptor messenger RNAs in lung cancer cell lines.
199546
6 199741
7 199938
8 200030
9 200727
10 199723
11 202111
12 19893

About Askale Mathias

Askale Mathias is a scholar working on Molecular Biology, Immunology, Biotechnology, Oncology and Radiology, Nuclear Medicine and Imaging, having authored 12 papers that have together received 494 indexed citations. Recurring topics across this work include Toxin Mechanisms and Immunotoxins (4 papers), Transgenic Plants and Applications (4 papers), TGF-β signaling in diseases (3 papers), Bone Metabolism and Diseases (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Microbial Inactivation Methods (2 papers), Neurobiology and Insect Physiology Research (1 paper) and Ion channel regulation and function (1 paper). The work is most often cited by research in Biotechnology (117 citations), Immunology (161 citations), Radiology, Nuclear Medicine and Imaging (115 citations), Oncology (136 citations) and Molecular Biology (246 citations). Askale Mathias has collaborated with scholars based in United States. Frequent co-authors include David M. Neville, Scott Stavrou, Sonia B. Jakowlew, Jung Hee Woo, Hyun S. Lillehoj, John A. Cook, Rajani Choudhuri, Sarwat Naz, Terry W. Moody and Philip E.D. Chung. Their work appears in journals such as Protein Engineering Design and Selection, Protein Expression and Purification, Clinical Cancer Research, International Journal of Radiation Oncology*Biology*Physics and Biology of Reproduction.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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