Anzhi Dai

574 total citations
10 papers, 477 citations indexed

About

Anzhi Dai is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Anzhi Dai has authored 10 papers receiving a total of 477 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Physiology and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Anzhi Dai's work include Mitochondrial Function and Pathology (4 papers), Glycosylation and Glycoproteins Research (3 papers) and Nitric Oxide and Endothelin Effects (3 papers). Anzhi Dai is often cited by papers focused on Mitochondrial Function and Pathology (4 papers), Glycosylation and Glycoproteins Research (3 papers) and Nitric Oxide and Endothelin Effects (3 papers). Anzhi Dai collaborates with scholars based in United States, Mexico and China. Anzhi Dai's co-authors include Brian T. Scott, Ayako Makino, Wolfgang Dillmann, Jorge Suárez, Tanja Diemer, Julieta Anabell Díaz-Juárez, Ying Han, Aninda Basu, Jason X.‐J. Yuan and Minglin Pan and has published in prestigious journals such as Journal of Biological Chemistry, Diabetes and The FASEB Journal.

In The Last Decade

Anzhi Dai

10 papers receiving 466 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anzhi Dai United States 9 332 156 106 80 61 10 477
Roselle Gélinas Canada 15 321 1.0× 218 1.4× 166 1.6× 73 0.9× 39 0.6× 22 579
Rakhee S. Gupte United States 10 246 0.7× 149 1.0× 192 1.8× 71 0.9× 52 0.9× 13 593
Daniel Franco United States 14 282 0.8× 70 0.4× 102 1.0× 115 1.4× 50 0.8× 24 510
Ke‐Xue Li China 12 207 0.6× 95 0.6× 63 0.6× 43 0.5× 59 1.0× 17 465
Dajun Zhao China 8 193 0.6× 143 0.9× 98 0.9× 35 0.4× 39 0.6× 15 493
Tadashi Seno Japan 6 136 0.4× 68 0.4× 95 0.9× 26 0.3× 93 1.5× 6 430
Norbert Fülöp United States 10 555 1.7× 94 0.6× 80 0.8× 73 0.9× 267 4.4× 14 724
Saori Takeshita Japan 6 161 0.5× 155 1.0× 204 1.9× 35 0.4× 155 2.5× 9 531
Nicolas Godin Canada 7 185 0.6× 81 0.5× 48 0.5× 74 0.9× 34 0.6× 8 437

Countries citing papers authored by Anzhi Dai

Since Specialization
Citations

This map shows the geographic impact of Anzhi Dai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anzhi Dai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anzhi Dai more than expected).

Fields of papers citing papers by Anzhi Dai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anzhi Dai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anzhi Dai. The network helps show where Anzhi Dai may publish in the future.

Co-authorship network of co-authors of Anzhi Dai

This figure shows the co-authorship network connecting the top 25 collaborators of Anzhi Dai. A scholar is included among the top collaborators of Anzhi Dai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anzhi Dai. Anzhi Dai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Scott, Brian T., Jorge Suárez, Darren E. Casteel, et al.. (2020). Ncor2/PPARα-Dependent Upregulation of MCUb in the Type 2 Diabetic Heart Impacts Cardiac Metabolic Flexibility and Function. Diabetes. 70(3). 665–679. 30 indexed citations
2.
Si, Rui, Atsumi Tsuji‐Hosokawa, Makiko Watanabe, et al.. (2019). Overexpression of p53 due to excess protein O-GlcNAcylation is associated with coronary microvascular disease in type 2 diabetes. Cardiovascular Research. 116(6). 1186–1198. 34 indexed citations
3.
Suárez, Jorge, Brian T. Scott, Julieta Anabell Díaz-Juárez, et al.. (2018). Restoring mitochondrial calcium uniporter expression in diabetic mouse heart improves mitochondrial calcium handling and cardiac function. Journal of Biological Chemistry. 293(21). 8182–8195. 75 indexed citations
4.
Pan, Minglin, Ying Han, Aninda Basu, et al.. (2018). Overexpression of hexokinase 2 reduces mitochondrial calcium overload in coronary endothelial cells of type 2 diabetic mice. American Journal of Physiology-Cell Physiology. 314(6). C732–C740. 26 indexed citations
5.
Scott, Brian T., Anzhi Dai, Wenlong Han, et al.. (2016). O-GlcNAcylation of 8-Oxoguanine DNA Glycosylase (Ogg1) Impairs Oxidative Mitochondrial DNA Lesion Repair in Diabetic Hearts. Journal of Biological Chemistry. 291(51). 26515–26528. 57 indexed citations
6.
Díaz-Juárez, Julieta Anabell, Jorge Suárez, Brian T. Scott, et al.. (2016). Expression of the mitochondrial calcium uniporter in cardiac myocytes improves impaired mitochondrial calcium handling and metabolism in simulated hyperglycemia. American Journal of Physiology-Cell Physiology. 311(6). C1005–C1013. 54 indexed citations
7.
Han, Ying, Ramon J. Ayon, Rui Guo, et al.. (2015). SGLT inhibitors attenuate NO-dependent vascular relaxation in the pulmonary artery but not in the coronary artery. American Journal of Physiology-Lung Cellular and Molecular Physiology. 309(9). L1027–L1036. 86 indexed citations
8.
Makino, Ayako, Anzhi Dai, Ying Han, et al.. (2015). O-GlcNAcase overexpression reverses coronary endothelial cell dysfunction in type 1 diabetic mice. American Journal of Physiology-Cell Physiology. 309(9). C593–C599. 46 indexed citations
9.
Han, Ying, Young‐Eun Cho, Ramon J. Ayon, et al.. (2014). SGLT inhibitors attenuate NO‐dependent vascular relaxation in mouse pulmonary artery (677.3). The FASEB Journal. 28(S1). 1 indexed citations
10.
Basu, Aninda, et al.. (2013). Coronary endothelial dysfunction and mitochondrial reactive oxygen species in type 2 diabetic mice. American Journal of Physiology-Cell Physiology. 305(10). C1033–C1040. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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