Anthony J. Coyle

39.2k total citations · 12 hit papers
209 papers, 30.3k citations indexed

About

Anthony J. Coyle is a scholar working on Immunology, Physiology and Immunology and Allergy. According to data from OpenAlex, Anthony J. Coyle has authored 209 papers receiving a total of 30.3k indexed citations (citations by other indexed papers that have themselves been cited), including 136 papers in Immunology, 73 papers in Physiology and 35 papers in Immunology and Allergy. Recurrent topics in Anthony J. Coyle's work include Asthma and respiratory diseases (70 papers), Immune Cell Function and Interaction (65 papers) and T-cell and B-cell Immunology (44 papers). Anthony J. Coyle is often cited by papers focused on Asthma and respiratory diseases (70 papers), Immune Cell Function and Interaction (65 papers) and T-cell and B-cell Immunology (44 papers). Anthony J. Coyle collaborates with scholars based in United States, Canada and United Kingdom. Anthony J. Coyle's co-authors include José-Carlos Gutierrez-Ramos, Daniel C. Rowe, John Bertin, Ethan Grant, Eicke Latz, Gary P. Sims, Katherine A. Fitzgerald, Jane Tian, Sha-Mei Liao and Ronald Herbst and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

Anthony J. Coyle

208 papers receiving 29.7k citations

Hit Papers

IKKε and TBK1 are essential components of the IRF3 signal... 1998 2026 2007 2016 2003 2002 2003 2010 2004 500 1000 1.5k 2.0k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. IKKε and TBK1 are essential components of the IRF3 signaling pathway
    2003 2.2k citations Anthony J. Coyle et al. Nature Immunology profile →
  2. Th1-specific cell surface protein Tim-3 regulates macrophage activation and severity of an autoimmune disease
    2002 1.4k citations Anthony J. Coyle et al. profile →
  3. Nod1 Detects a Unique Muropeptide from Gram-Negative Bacterial Peptidoglycan
    2003 1.2k citations Stephen E. Girardin, Ivo G. Boneca et al. Science profile →
  4. HMGB1 and RAGE in Inflammation and Cancer
    2010 1.2k citations Gary P. Sims, Anthony J. Coyle et al. profile →
  5. Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island
    2004 971 citations Jérôme Viala, Ivo G. Boneca et al. Nature Immunology profile →
  6. Cytosolic flagellin requires Ipaf for activation of caspase-1 and interleukin 1β in salmonella-infected macrophages
    2006 949 citations Luigi Franchi, Amal O. Amer et al. Nature Immunology profile →
  7. Bacterial RNA and small antiviral compounds activate caspase-1 through cryopyrin/Nalp3
    2006 935 citations Thirumala‐Devi Kanneganti, Nesrin Özören et al. profile →
  8. Critical Role for NALP3/CIAS1/Cryopyrin in Innate and Adaptive Immunity through Its Regulation of Caspase-1
    2006 799 citations Ethan Grant, John Bertin et al. profile →
  9. Tim-3 inhibits T helper type 1–mediated auto- and alloimmune responses and promotes immunological tolerance
    2003 601 citations Jane Tian, José-Carlos Gutierrez-Ramos et al. Nature Immunology profile →
  10. Mast cells are essential intermediaries in regulatory T-cell tolerance
    2006 590 citations Anthony J. Coyle et al. profile →
  11. Interaction of Tim-3 and Tim-3 ligand regulates T helper type 1 responses and induction of peripheral tolerance
    2003 538 citations Anthony J. Coyle et al. Nature Immunology profile →
  12. T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function
    1998 516 citations Anthony J. Coyle, José-Carlos Gutierrez-Ramos et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anthony J. Coyle United States 84 19.8k 7.7k 6.6k 4.2k 3.1k 209 30.3k
Craig Gérard United States 86 16.2k 0.8× 6.3k 0.8× 4.5k 0.7× 5.8k 1.4× 2.2k 0.7× 223 29.5k
Sergio Romagnani Italy 95 20.6k 1.0× 4.7k 0.6× 6.2k 0.9× 4.9k 1.2× 3.2k 1.0× 350 36.0k
René de Waal Malefyt United States 70 25.5k 1.3× 4.5k 0.6× 4.8k 0.7× 5.8k 1.4× 2.2k 0.7× 150 38.3k
Charles R. Mackay Australia 104 25.5k 1.3× 12.5k 1.6× 7.8k 1.2× 9.1k 2.2× 2.9k 0.9× 255 47.0k
Raif S. Geha United States 99 19.4k 1.0× 5.3k 0.7× 4.6k 0.7× 2.7k 0.6× 1.6k 0.5× 501 31.8k
Steven F. Ziegler United States 85 20.4k 1.0× 4.2k 0.5× 5.6k 0.9× 4.3k 1.0× 2.0k 0.6× 240 30.7k
Enrico Maggi Italy 83 15.6k 0.8× 3.6k 0.5× 4.5k 0.7× 3.9k 0.9× 2.1k 0.7× 367 27.2k
Sharon M. Wahl United States 87 15.0k 0.8× 7.9k 1.0× 2.7k 0.4× 4.4k 1.1× 2.5k 0.8× 277 31.6k
William E. Paul United States 98 30.2k 1.5× 8.2k 1.1× 4.7k 0.7× 6.1k 1.5× 2.8k 0.9× 344 42.5k
Volker Brinkmann Germany 91 23.2k 1.2× 19.4k 2.5× 3.5k 0.5× 3.4k 0.8× 3.6k 1.2× 250 45.3k

Countries citing papers authored by Anthony J. Coyle

Since Specialization
Citations

This map shows the geographic impact of Anthony J. Coyle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anthony J. Coyle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anthony J. Coyle more than expected).

Fields of papers citing papers by Anthony J. Coyle

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anthony J. Coyle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anthony J. Coyle. The network helps show where Anthony J. Coyle may publish in the future.

Co-authorship network of co-authors of Anthony J. Coyle

This figure shows the co-authorship network connecting the top 25 collaborators of Anthony J. Coyle. A scholar is included among the top collaborators of Anthony J. Coyle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anthony J. Coyle. Anthony J. Coyle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
McInnis, Christine, Shilpa Bhatia, Brinda Vijaykumar, et al.. (2023). Identification of HPV16 E1 and E2-specific T cells in the oropharyngeal cancer tumor microenvironment. Journal for ImmunoTherapy of Cancer. 11(3). e006721–e006721. 5 indexed citations
2.
Delaney, Tracy, Chris Morehouse, Philip Z. Brohawn, et al.. (2016). Type I IFNs Regulate Inflammation, Vasculopathy, and Fibrosis in Chronic Cutaneous Graft-versus-Host Disease. The Journal of Immunology. 197(1). 42–50. 27 indexed citations
3.
Bara, Imane, A. Ozier, Pierre‐Olivier Girodet, et al.. (2012). Role of YKL-40 in Bronchial Smooth Muscle Remodeling in Asthma. American Journal of Respiratory and Critical Care Medicine. 185(7). 715–722. 98 indexed citations
4.
Nikota, Jake K., Fernando Botelho, Carla M. T. Bauer, et al.. (2011). Differential expression and function of breast regression protein 39 (BRP-39) in murine models of subacute cigarette smoke exposure and allergic airway inflammation. Respiratory Research. 12(1). 39–39. 22 indexed citations
5.
Stephens, Geoffrey L., Ebony Benjamin, Anthony J. Coyle, et al.. (2011). IL‐9 is a Th17‐derived cytokine that limits pathogenic activity in organ‐specific autoimmune disease. European Journal of Immunology. 41(4). 952–962. 48 indexed citations
6.
Kearley, Jennifer, Jonas S. Erjefält, Cecilia Andersson, et al.. (2010). IL-9 Governs Allergen-induced Mast Cell Numbers in the Lung and Chronic Remodeling of the Airways. American Journal of Respiratory and Critical Care Medicine. 183(7). 865–875. 161 indexed citations
7.
Fattouh, Ramzi, Amal Al‐Garawi, Katherine Arias, et al.. (2010). Eosinophils Are Dispensable for Allergic Remodeling and Immunity in a Model of House Dust Mite–induced Airway Disease. American Journal of Respiratory and Critical Care Medicine. 183(2). 179–188. 64 indexed citations
8.
Létuvé, S., Alexander Kozhich, Olivier Thibaudeau, et al.. (2010). Expression of High-Mobility Group Box 1 and of Receptor for Advanced Glycation End Products in Chronic Obstructive Pulmonary Disease. American Journal of Respiratory and Critical Care Medicine. 181(9). 917–927. 184 indexed citations
9.
Yamasaki, Kenshi, Jun Muto, Kristen R. Taylor, et al.. (2009). NLRP3/Cryopyrin Is Necessary for Interleukin-1β (IL-1β) Release in Response to Hyaluronan, an Endogenous Trigger of Inflammation in Response to Injury. Journal of Biological Chemistry. 284(19). 12762–12771. 225 indexed citations
10.
Létuvé, S., Alexander Kozhich, Alison A. Humbles, et al.. (2009). Lung Chitinolytic Activity and Chitotriosidase Are Elevated in Chronic Obstructive Pulmonary Disease and Contribute to Lung Inflammation. American Journal Of Pathology. 176(2). 638–649. 64 indexed citations
11.
Franchi, Luigi, Amal O. Amer, Mathilde Body–Malapel, et al.. (2006). Cytosolic flagellin requires Ipaf for activation of caspase-1 and interleukin 1β in salmonella-infected macrophages. Nature Immunology. 7(6). 576–582. 949 indexed citations breakdown →
12.
Simmons, William J., Mythili Koneru, Mani Mohindru, et al.. (2005). Tim-3+ T-bet+ Tumor-Specific Th1 Cells Colocalize with and Inhibit Development and Growth of Murine Neoplasms. The Journal of Immunology. 174(3). 1405–1415. 25 indexed citations
13.
Schmidt‐Supprian, Marc, Jane Tian, Ethan Grant, et al.. (2004). Differential dependence of CD4 + CD25 + regulatory and natural killer-like T cells on signals leading to NF-κB activation. Proceedings of the National Academy of Sciences. 101(13). 4566–4571. 202 indexed citations
14.
Nanji, Sulaiman, Wayne W. Hancock, Colin C. Anderson, et al.. (2004). Multiple Combination Therapies Involving Blockade of ICOS/B7RP-1 Costimulation Facilitate Long-Term Islet Allograft Survival. American Journal of Transplantation. 4(4). 526–536. 60 indexed citations
15.
Cates, Elizabeth C., Ramzi Fattouh, Jennifer Wattie, et al.. (2004). Intranasal Exposure of Mice to House Dust Mite Elicits Allergic Airway Inflammation via a GM-CSF-Mediated Mechanism. The Journal of Immunology. 173(10). 6384–6392. 227 indexed citations
16.
Girardin, Stephen E., Ivo G. Boneca, Letícia A. M. Carneiro, et al.. (2003). Nod1 Detects a Unique Muropeptide from Gram-Negative Bacterial Peptidoglycan. Science. 300(5625). 1584–1587. 1220 indexed citations breakdown →
17.
Johnson, Jill R., Ryan E. Wiley, Ramzi Fattouh, et al.. (2003). Continuous Exposure to House Dust Mite Elicits Chronic Airway Inflammation and Structural Remodeling. American Journal of Respiratory and Critical Care Medicine. 169(3). 378–385. 346 indexed citations
18.
Lloyd, Clare M., José-Ángel Gonzalo, Trang Nguyen, et al.. (2001). Resolution of Bronchial Hyperresponsiveness and Pulmonary Inflammation Is Associated with IL-3 and Tissue Leukocyte Apoptosis. The Journal of Immunology. 166(3). 2033–2040. 49 indexed citations
19.
Meyer, Alexandra, Anthony J. Coyle, Amanda E. I. Proudfoot, Timothy N. C. Wells, & Christine A. Power. (1996). Cloning and characterization of a novel murine macrophage inflammatory protein-1α receptor. Journal of Biological Chemistry. 271(38). 23601–23601. 2 indexed citations
20.
Coyle, Anthony J., et al.. (1995). Human eosinophil-granule major basic protein and synthetic polycations induce airway hyperresponsiveness in vivo dependent on bradykinin generation.. Journal of Clinical Investigation. 95(4). 1735–1740. 86 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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