Anniina Virtanen
- Oncology top 10%
- Immunology
- Rheumatology top 10%
- Genetics top 10%
- Molecular Biology
- Co-authors
- Olli SilvennoinenJuuli RaivolaT. HaikarainenHenrik M. HammarénPia IsomäkiKrista KuulialaMassimo GadinaMarjatta Leirisalo‐Repo
- Topics
- Cytokine Signaling Pathways and Interactions (15 papers)Myeloproliferative Neoplasms: Diagnosis and Treatment (9 papers)Rheumatoid Arthritis Research and Therapies (5 papers)
- Cited by
- GeneticsRheumatologyOncology
- Journals
- Nature Reviews Drug DiscoveryJournal of Allergy and Clinical ImmunologyJournal of Medicinal Chemistry
- Partner nations
- FinlandUnited StatesGermany
In The Last Decade
Anniina Virtanen
16 papers receiving 556 citations
Peers
Comparison fields: 5 of 71
- Oncology 250
- Immunology 151
- Rheumatology 147
- Genetics 128
- Molecular Biology 125
Countries citing papers authored by Anniina Virtanen
This map shows the geographic impact of Anniina Virtanen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anniina Virtanen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anniina Virtanen more than expected).
Fields of papers citing papers by Anniina Virtanen
This network shows the impact of papers produced by Anniina Virtanen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anniina Virtanen. The network helps show where Anniina Virtanen may publish in the future.
Co-authorship network of co-authors of Anniina Virtanen
This figure shows the co-authorship network connecting the top 25 collaborators of Anniina Virtanen. A scholar is included among the top collaborators of Anniina Virtanen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anniina Virtanen. Anniina Virtanen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 32 | |
| 4 | 7 | |
| 5 | 8 | |
| 6 | 13 | |
| 7 | 15 | |
| 8 | 2 | |
| 9 | 15 | |
| 10 | 1 | |
| 11 | 39 | |
| 12 | 7 | |
| 13 | 204 | |
| 14 | 22 | |
| 15 | 15 | |
| 16 | 156 | |
| 17 | 31 |
About Anniina Virtanen
Anniina Virtanen is a scholar working on Genetics, Anatomy and Oncology, having authored 17 papers that have together received 570 indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (15 papers), Myeloproliferative Neoplasms: Diagnosis and Treatment (9 papers) and Rheumatoid Arthritis Research and Therapies (5 papers). The work is most often cited by research in Genetics (128 citations), Rheumatology (147 citations) and Oncology (250 citations). Anniina Virtanen has collaborated with scholars based in Finland, United States and Germany. Frequent co-authors include Olli Silvennoinen, Juuli Raivola, T. Haikarainen, Henrik M. Hammarén, Pia Isomäki, Krista Kuuliala, Massimo Gadina, Marjatta Leirisalo‐Repo, Francesca Romana Spinelli and Ritva Peltomaa. Their work appears in journals such as Nature Reviews Drug Discovery, Journal of Allergy and Clinical Immunology and Journal of Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.