Anne‐Laurence Boutillier

4.2k total citations · 1 hit paper
54 papers, 2.7k citations indexed

About

Anne‐Laurence Boutillier is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Anne‐Laurence Boutillier has authored 54 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Molecular Biology, 17 papers in Cellular and Molecular Neuroscience and 13 papers in Physiology. Recurrent topics in Anne‐Laurence Boutillier's work include Histone Deacetylase Inhibitors Research (9 papers), Neurogenesis and neuroplasticity mechanisms (8 papers) and Genetic Neurodegenerative Diseases (7 papers). Anne‐Laurence Boutillier is often cited by papers focused on Histone Deacetylase Inhibitors Research (9 papers), Neurogenesis and neuroplasticity mechanisms (8 papers) and Genetic Neurodegenerative Diseases (7 papers). Anne‐Laurence Boutillier collaborates with scholars based in France, United States and Germany. Anne‐Laurence Boutillier's co-authors include Jean‐Philippe Loeffler, Caroline Rouaux, Jean‐Christophe Cassel, Jean‐Philippe Loeffler, Emmanuelle Trinh, Olivier Bousiges, Andoni Echaniz‐Laguna, Jean-Philippe Loeffler, Frédérique René and Tapas K. Kundu and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Anne‐Laurence Boutillier

53 papers receiving 2.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Polycystic ovary syndrome is transmitted via a transgenerational epigenetic process

2021 161 citations Isabel Paiva, Damien Plassard et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Anne‐Laurence Boutillier France	29	1.5k	762	396	383	330	54	2.7k
Eiichiro Nagata Japan	30	2.1k 1.4×	1.2k 1.6×	588 1.5×	485 1.3×	208 0.6×	117	4.0k
Juan José Toledo‐Aral Spain	26	2.1k 1.4×	1.2k 1.6×	479 1.2×	395 1.0×	432 1.3×	50	3.3k
Susan C. Su United States	14	1.8k 1.2×	640 0.8×	594 1.5×	250 0.7×	430 1.3×	15	2.7k
Eugene P. Brandon United States	29	2.0k 1.4×	1.4k 1.8×	563 1.4×	244 0.6×	606 1.8×	32	3.7k
Jasper J. Anink Netherlands	30	1.1k 0.7×	657 0.9×	517 1.3×	430 1.1×	331 1.0×	68	2.5k
Esther B. E. Becker United Kingdom	29	2.2k 1.5×	885 1.2×	191 0.5×	250 0.7×	264 0.8×	47	3.4k
Rosanna Parlato Germany	28	1.5k 1.0×	879 1.2×	155 0.4×	283 0.7×	428 1.3×	63	2.4k
Donald Pizzo United States	31	1.7k 1.1×	643 0.8×	451 1.1×	279 0.7×	74 0.2×	80	3.1k
Yun‐Li Ma Taiwan	31	1.2k 0.8×	798 1.0×	303 0.8×	123 0.3×	405 1.2×	68	2.4k
Ignacio Muñoz-Sanjuán United States	29	2.4k 1.6×	1.2k 1.6×	208 0.5×	470 1.2×	275 0.8×	82	3.3k

Countries citing papers authored by Anne‐Laurence Boutillier

Since Specialization

Citations

This map shows the geographic impact of Anne‐Laurence Boutillier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anne‐Laurence Boutillier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anne‐Laurence Boutillier more than expected).

Fields of papers citing papers by Anne‐Laurence Boutillier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anne‐Laurence Boutillier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anne‐Laurence Boutillier. The network helps show where Anne‐Laurence Boutillier may publish in the future.

Co-authorship network of co-authors of Anne‐Laurence Boutillier

This figure shows the co-authorship network connecting the top 25 collaborators of Anne‐Laurence Boutillier. A scholar is included among the top collaborators of Anne‐Laurence Boutillier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anne‐Laurence Boutillier. Anne‐Laurence Boutillier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Séguin, Jonathan, Brigitte Cosquer, Étienne Birmelé, et al.. (2025). Accelerated epigenetic aging in Huntington’s disease involves polycomb repressive complex 1. Nature Communications. 16(1). 1550–1550. 3 indexed citations

Kemmel, Véronique, et al.. (2025). Passive immunization against amyloid peptide restores pattern separation deficits in early stage of amyloid pathology but not in normal aging. SPIRE - Sciences Po Institutional REpository. 5.

Paiva, Isabel, Jonathan Séguin, Brigitte Cosquer, et al.. (2024). Dysregulated expression of cholesterol biosynthetic genes in Alzheimer's disease alters epigenomic signatures of hippocampal neurons. Neurobiology of Disease. 198. 106538–106538. 4 indexed citations

Paiva, Isabel, et al.. (2023). Circadian functioning of Locus Cœruleus of the nocturnal rat and diurnal rodent Arvicanthis. Neuroscience Letters. 799. 137091–137091. 2 indexed citations

Séguin, Jonathan, Anne Molitor, Ibai Irastorza-Azcárate, et al.. (2021). Age-related and disease locus-specific mechanisms contribute to early remodelling of chromatin structure in Huntington’s disease mice. Nature Communications. 12(1). 364–364. 20 indexed citations

Wang, Xiaolan, Sander Kooijman, Yuanqing Gao, et al.. (2021). Microglia-specific knock-down of Bmal1 improves memory and protects mice from high fat diet-induced obesity. Molecular Psychiatry. 26(11). 6336–6349. 52 indexed citations

Boutillier, Anne‐Laurence, et al.. (2020). Epigenetic mechanisms underlying enhancer modulation of neuronal identity, neuronal activity and neurodegeneration. Neurobiology of Disease. 147. 105155–105155. 14 indexed citations

Wang, Xiaolan, Nikita L. Korpel, Irina V. Milanova, et al.. (2020). Deficiency of the Circadian Clock Gene Bmal1 Reduces Microglial Immunometabolism. Frontiers in Immunology. 11. 586399–586399. 55 indexed citations

Swaminathan, Amrutha, Hélène Delage, Snehajyoti Chatterjee, et al.. (2016). Transcriptional Coactivator and Chromatin Protein PC4 Is Involved in Hippocampal Neurogenesis and Spatial Memory Extinction. Journal of Biological Chemistry. 291(39). 20303–20314. 17 indexed citations

10.

Puttagunta, Radhika, Andrea Tedeschi, Marília Grando Sória, et al.. (2014). PCAF-dependent epigenetic changes promote axonal regeneration in the central nervous system. Nature Communications. 5(1). 3527–3527. 129 indexed citations

11.

Rouaux, Caroline, Frédérique René, José‐Luis González de Aguilar, et al.. (2007). Sodium Valproate Exerts Neuroprotective EffectsIn Vivothrough CREB-Binding Protein-Dependent Mechanisms But Does Not Improve Survival in an Amyotrophic Lateral Sclerosis Mouse Model. Journal of Neuroscience. 27(21). 5535–5545. 174 indexed citations

12.

Lannes, Béatrice, Luc Buée, André Delacourte, et al.. (2007). Sp3 and Sp4 Transcription Factor Levels Are Increased in Brains of Patients with Alzheimer’s Disease. Neurodegenerative Diseases. 4(6). 413–423. 27 indexed citations

13.

Sée, Violaine, David G. Spiller, Caroline Rouaux, et al.. (2007). HP1α guides neuronal fate by timing E2F‐targeted genes silencing during terminal differentiation. The EMBO Journal. 26(15). 3616–3628. 40 indexed citations

14.

Rouaux, Caroline, et al.. (2004). HDAC‐3 Participates in the Repression of e2f‐Dependent Gene Transcription in Primary Differentiated Neurons. Annals of the New York Academy of Sciences. 1030(1). 656–660. 18 indexed citations

15.

Boutillier, Anne‐Laurence, Emmanuelle Trinh, & Jean‐Philippe Loeffler. (2003). Selective E2F‐dependent gene transcription is controlled by histone deacetylase activity during neuronal apoptosis. Journal of Neurochemistry. 84(4). 814–828. 84 indexed citations

16.

Trinh, Emmanuelle, Anne‐Laurence Boutillier, & Jean‐Philippe Loeffler. (2001). Regulation of the Retinoblastoma-Dependent Mdm2 and E2F-1 Signaling Pathways during Neuronal Apoptosis. Molecular and Cellular Neuroscience. 17(2). 342–353. 32 indexed citations

17.

Boutillier, Anne‐Laurence, Pascal Kienlen‐Campard, & Jean‐Philippe Loeffler. (1999). Depolarization regulates cyclin D1 degradation and neuronal apoptosis: a hypothesis about the role of the ubiquitin/proteasome signalling pathway. European Journal of Neuroscience. 11(2). 441–448. 60 indexed citations

18.

Gaiddon, Christian, Yves Larmet, Emmanuelle Trinh, et al.. (1999). Brain‐Derived Neurotrophic Factor Exerts Opposing Effects on β₂‐Adrenergic Receptor According to Depolarization Status of Cerebellar Neurons. Journal of Neurochemistry. 73(4). 1467–1476. 7 indexed citations

19.

Boutillier, Anne‐Laurence, D. Monnier, Dominique Lorang, et al.. (1995). Corticotropin-releasing hormone stimulates proopiomelanocortin transcription by cFos-dependent and -independent pathways: characterization of an AP1 site in exon 1.. Molecular Endocrinology. 9(6). 745–755. 81 indexed citations

20.

Monnier, D., Anne‐Laurence Boutillier, P. Giraud, et al.. (1994). Insulin-like growth factor-I stimulates c-fos and c-jun transcription in PC12 cells. Molecular and Cellular Endocrinology. 104(2). 139–145. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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