Andrew B. Singleton
Impact in
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- Genetic Neurodegenerative Diseases
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- Neurological diseases and metabolism
- Parkinson's Disease Mechanisms and Treatments
- Neurological disorders and treatments
Papers in
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- Genetic Neurodegenerative Diseases 4
- Hereditary Neurological Disorders 1
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- Mitochondrial Function and Pathology 2
- DNA Repair Mechanisms 1
- Co-authors
- Joyce van de Leemput (2 shared papers)Dena G. Hernandez (2 shared papers)Elsdon Storey (1 shared paper)Scott J. Diede (1 shared paper)G.J.B. van Ommen (1 shared paper)Dena Hernández (1 shared paper)Ian Rafferty (1 shared paper)S. M. Forrest (1 shared paper)
- Journals
- European Journal of Neurology (1 paper)Human Molecular Genetics (1 paper)Movement Disorders (1 paper)Neuroscience Letters (1 paper)
- Partner nations
- United StatesUnited KingdomAustralia
In The Last Decade
Andrew B. Singleton
4 papers receiving 102 citations
Peers
Comparison fields: 5 of 20
- Cellular and Molecular Neuroscience 77
- Neurology 21
- Neurology 32
- Genetics 38
- Molecular Biology 78
Countries citing papers authored by Andrew B. Singleton
This map shows the geographic impact of Andrew B. Singleton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew B. Singleton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew B. Singleton more than expected).
Fields of papers citing papers by Andrew B. Singleton
This network shows the impact of papers produced by Andrew B. Singleton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew B. Singleton. The network helps show where Andrew B. Singleton may publish in the future.
Co-authors
The 25 scholars most cited alongside Andrew B. Singleton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 51 | |
| 2 | 2005 | 32 | |
| 3 | 2012 | 22 | |
| 4 | 2014 | 22 |
About Andrew B. Singleton
Andrew B. Singleton is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Neurology, Neurology and Genetics, having authored 4 papers that have together received 127 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (4 papers), Mitochondrial Function and Pathology (2 papers), Neurological diseases and metabolism (1 paper), Hereditary Neurological Disorders (1 paper), Parkinson's Disease Mechanisms and Treatments (1 paper), DNA Repair Mechanisms (1 paper), Genetics and Neurodevelopmental Disorders (1 paper) and Genomic variations and chromosomal abnormalities (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (77 citations), Neurology (21 citations), Neurology (32 citations), Genetics (38 citations) and Molecular Biology (78 citations). Andrew B. Singleton has collaborated with scholars based in United States, United Kingdom and Australia. Frequent co-authors include Joyce van de Leemput, Dena G. Hernandez, Elsdon Storey, Scott J. Diede, G.J.B. van Ommen, Dena Hernández, Ian Rafferty, S. M. Forrest, Hans G. Dauwerse and R. J. M. Gardner. Their work appears in journals such as European Journal of Neurology, Human Molecular Genetics, Movement Disorders and Neuroscience Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.