S. M. Forrest

2.4k total citations
39 papers, 1.7k citations indexed

About

S. M. Forrest is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, S. M. Forrest has authored 39 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 11 papers in Cellular and Molecular Neuroscience and 8 papers in Genetics. Recurrent topics in S. M. Forrest's work include Muscle Physiology and Disorders (11 papers), Genetic Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (8 papers). S. M. Forrest is often cited by papers focused on Muscle Physiology and Disorders (11 papers), Genetic Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (8 papers). S. M. Forrest collaborates with scholars based in Australia, United Kingdom and United States. S. M. Forrest's co-authors include K. E. Davies, Gareth Cross, Andreas Speer, Kay E. Davies, Donald R. Love, Sarah England, Mark Johnson, Dennis E. Bulman, Elizabeth E. Zubrzycka‐Gaarn and J.B. Harris and has published in prestigious journals such as Nature, The Lancet and Nucleic Acids Research.

In The Last Decade

S. M. Forrest

38 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
S. M. Forrest Australia 23 1.3k 499 283 263 237 39 1.7k
Mariko Yagi Japan 21 1.1k 0.9× 226 0.5× 99 0.3× 225 0.9× 190 0.8× 66 1.5k
Behzad Moghadaszadeh United States 21 1.5k 1.1× 289 0.6× 213 0.8× 515 2.0× 274 1.2× 29 2.1k
Hiroyuki Awano Japan 21 1.2k 0.9× 191 0.4× 142 0.5× 306 1.2× 320 1.4× 125 1.5k
Alison J. Coffey United Kingdom 17 699 0.5× 242 0.5× 85 0.3× 169 0.6× 79 0.3× 27 1.0k
Tomasa Barrientos United States 17 1.2k 1.0× 146 0.3× 127 0.4× 249 0.9× 243 1.0× 20 2.0k
W. Schmidt Germany 6 1.5k 1.2× 293 0.6× 124 0.4× 45 0.2× 118 0.5× 9 1.8k
Barbara Illi Italy 23 1.2k 1.0× 150 0.3× 114 0.4× 82 0.3× 100 0.4× 42 1.6k
Shu Kachi Japan 28 1.2k 0.9× 185 0.4× 199 0.7× 55 0.2× 34 0.1× 78 2.2k
Partha S. Sarkar United States 31 1.8k 1.4× 258 0.5× 893 3.2× 128 0.5× 94 0.4× 40 2.2k
Bulmaro Cisneros Mexico 25 1.4k 1.1× 117 0.2× 575 2.0× 112 0.4× 80 0.3× 123 1.9k

Countries citing papers authored by S. M. Forrest

Since Specialization
Citations

This map shows the geographic impact of S. M. Forrest's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. M. Forrest with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. M. Forrest more than expected).

Fields of papers citing papers by S. M. Forrest

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. M. Forrest. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. M. Forrest. The network helps show where S. M. Forrest may publish in the future.

Co-authorship network of co-authors of S. M. Forrest

This figure shows the co-authorship network connecting the top 25 collaborators of S. M. Forrest. A scholar is included among the top collaborators of S. M. Forrest based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. M. Forrest. S. M. Forrest is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fitzpatrick, Emer, Matthew P. Johnson, Thomas D. Dyer, et al.. (2009). Genetic association of the activin A receptor gene (ACVR2A) and pre-eclampsia. Molecular Human Reproduction. 15(3). 195–204. 34 indexed citations
2.
Knight, Melanie A., Dena Hernández, Scott J. Diede, et al.. (2008). A duplication at chromosome 11q12.2-11q12.3 is associated with spinocerebellar ataxia type 20. Human Molecular Genetics. 17(24). 3847–3853. 51 indexed citations
3.
Fitzpatrick, Emer, H.H.H. Göring, Anthony J. Borg, et al.. (2004). Fine Mapping and SNP Analysis of Positional Candidates at the Preeclampsia Susceptibility Locus (PREG1) on Chromosome 2. Human Biology. 76(6). 849–862. 33 indexed citations
4.
Cashman, John R., Beverly R. Akerman, S. M. Forrest, & Eileen P. Treacy. (2000). Population-Specific Polymorphisms of the Human FMO3 Gene: Significance for Detoxication. Drug Metabolism and Disposition. 28(2). 169–173. 62 indexed citations
5.
Delatycki, Martin B., Melanie A. Knight, Michel Kœnig, et al.. (1999). G130V, a common FRDA point mutation, appears to have arisen from a common founder. Human Genetics. 105(4). 343–346. 33 indexed citations
6.
Akerman, Beverly R., S. M. Forrest, L. H. Chow, et al.. (1999). Two novel mutations of theFMO3 gene in a proband with trimethylaminuria. Human Mutation. 13(5). 376–379. 35 indexed citations
7.
Delatycki, Martin B., Damien B.B.P. Paris, Garth A. Nicholson, et al.. (1998). Sperm DNA analysis in a Friedreich ataxia premutation carrier suggests both meiotic and mitotic expansion in the FRDA gene.. Journal of Medical Genetics. 35(9). 713–716. 31 indexed citations
8.
Lagerström‐Fermér, Maria, Mats Sundvall, Garry L. Warne, et al.. (1997). X-linked recessive panhypopituitarism associated with a regional duplication in Xq25-q26.. PubMed. 60(4). 910–6. 46 indexed citations
9.
Forrest, S. M., et al.. (1994). Mutation detection in X-linked hydrocephalus. The American Journal of Human Genetics. 55. 2 indexed citations
10.
Cotton, Richard G.H., H H Dahl, S. M. Forrest, et al.. (1993). Analysis of Sequence Contexts Flanking T·G Mismatches Leads to Predictions about Reactivity of the Mismatched T to Osmium Tetroxide. DNA and Cell Biology. 12(10). 945–949. 13 indexed citations
11.
Mackey, David A., Steven Nasioulas, & S. M. Forrest. (1993). Finger prick blood testing in Leber hereditary optic neuropathy.. British Journal of Ophthalmology. 77(5). 311–312. 3 indexed citations
12.
Forrest, S. M., et al.. (1992). Use of the chemical cleavage of mismatch method for prenatal deficiency diagnosis of alpha‐1‐antitrypsin. Prenatal Diagnosis. 12(2). 133–137. 5 indexed citations
13.
Ramus, Susan J., et al.. (1992). CpG hotspot causes second mutation in codon 408 of the phenylalanine hydroxylase gene. Human Genetics. 90(1-2). 147–8. 4 indexed citations
14.
Nasioulas, Steven, et al.. (1991). Direct PCR from CVS and blood lysates for detection of cystic fibrosis and Duchenne muscular dystrophy deletions. Nucleic Acids Research. 19(5). 1155–1155. 34 indexed citations
15.
Speer, Andreas, et al.. (1989). DNA amplification of a further exon of Duchenne muscular dystrophy locus increase possibilities for deletion screening. Nucleic Acids Research. 17(12). 4892–4892. 11 indexed citations
16.
Forrest, S. M., Terry Smith, Gareth Cross, et al.. (1988). Molecular Analysis and Diagnosis of Duchenne Muscular Dystrophy. Journal of the Royal College of Physicians of London. 22(2). 65–67.
17.
Read, Andrew, R. Mountford, S. M. Forrest, et al.. (1988). Patterns of exon deletions in Duchenne and Becker muscular dystrophy. Human Genetics. 80(2). 152–156. 36 indexed citations
18.
Forrest, S. M., Gareth Cross, Tracey Flint, et al.. (1988). Further studies of gene deletions that cause Duchenne and Becker muscular dystrophies. Genomics. 2(2). 109–114. 147 indexed citations
19.
Forrest, S. M., Gareth Cross, Andreas Speer, et al.. (1987). Preferential deletion of exons in Duchenne and Becker muscular dystrophies. Nature. 329(6140). 638–640. 164 indexed citations
20.
Davies, Kay E., Sarah Ball, Huw Dorkins, et al.. (1986). Molecular analysis of X-linked diseases.. PubMed. 18(5-6). 231–3. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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