Amy M. Kadell

1.3k total citations
21 papers, 1.1k citations indexed

About

Amy M. Kadell is a scholar working on Internal Medicine, Hematology and Surgery. According to data from OpenAlex, Amy M. Kadell has authored 21 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Internal Medicine, 10 papers in Hematology and 9 papers in Surgery. Recurrent topics in Amy M. Kadell's work include Venous Thromboembolism Diagnosis and Management (16 papers), Blood Coagulation and Thrombosis Mechanisms (10 papers) and Atrial Fibrillation Management and Outcomes (5 papers). Amy M. Kadell is often cited by papers focused on Venous Thromboembolism Diagnosis and Management (16 papers), Blood Coagulation and Thrombosis Mechanisms (10 papers) and Atrial Fibrillation Management and Outcomes (5 papers). Amy M. Kadell collaborates with scholars based in United States, United Kingdom and South Korea. Amy M. Kadell's co-authors include Thomas W. Wakefield, Robert M. Strieter, Lazar J. Greenfield, Carol A. Wilke, Shirley K. Wrobleski, Marie D. Burdick, L. J. Downing, Peter K. Henke, Steven L. Kunkel and J. Brian Fowlkes and has published in prestigious journals such as The Journal of Immunology, Arteriosclerosis Thrombosis and Vascular Biology and Journal of Vascular Surgery.

In The Last Decade

Amy M. Kadell

21 papers receiving 1.0k citations

Peers

Amy M. Kadell
Diana Farris United States
Dorothée Kling Switzerland
A. Smith United Kingdom
AK Kakkar United Kingdom
T. Tschopp Switzerland
S I Rapaport United States
Édith Fressinaud France
C Korninger Austria
Gian Paolo Visentin United States
Roxane Darbousset France
Diana Farris United States
Amy M. Kadell
Citations per year, relative to Amy M. Kadell Amy M. Kadell (= 1×) peers Diana Farris

Countries citing papers authored by Amy M. Kadell

Since Specialization
Citations

This map shows the geographic impact of Amy M. Kadell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amy M. Kadell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amy M. Kadell more than expected).

Fields of papers citing papers by Amy M. Kadell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amy M. Kadell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amy M. Kadell. The network helps show where Amy M. Kadell may publish in the future.

Co-authorship network of co-authors of Amy M. Kadell

This figure shows the co-authorship network connecting the top 25 collaborators of Amy M. Kadell. A scholar is included among the top collaborators of Amy M. Kadell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amy M. Kadell. Amy M. Kadell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Henke, Peter K., Thomas W. Wakefield, Amy M. Kadell, et al.. (2001). Interleukin-8 Administration Enhances Venous Thrombosis Resolution in a Rat Model. Journal of Surgical Research. 99(1). 84–91. 89 indexed citations
2.
Henke, Peter K., Robert M. Strieter, Jonathan S. Bromberg, et al.. (2000). Viral IL-10 Gene Transfer Decreases Inflammation and Cell Adhesion Molecule Expression in a Rat Model of Venous Thrombosis,. The Journal of Immunology. 164(4). 2131–2141. 55 indexed citations
3.
Wakefield, Thomas W., Robert M. Strieter, Martin R. Prince, et al.. (2000). Venous thrombosis prophylaxis by inflammatory inhibition without anticoagulation therapy. Journal of Vascular Surgery. 31(2). 309–324. 75 indexed citations
4.
Wakefield, Thomas W., Marisa J. Linn, Peter K. Henke, et al.. (1999). Neovascularization during venous thrombosis organization: A preliminary study. Journal of Vascular Surgery. 30(5). 885–893. 94 indexed citations
5.
Fowlkes, J. Brian, Robert M. Strieter, L. J. Downing, et al.. (1998). Ultrasound echogenicity in experimental venous thrombosis. Ultrasound in Medicine & Biology. 24(8). 1175–1182. 30 indexed citations
6.
Downing, L. J., Robert M. Strieter, Amy M. Kadell, et al.. (1998). Low-dose low–molecular-weight heparin is anti-inflammatory during venous thrombosis. Journal of Vascular Surgery. 28(5). 848–854. 82 indexed citations
7.
Downing, L. J., Robert M. Strieter, Amy M. Kadell, et al.. (1998). IL-10 Regulates Thrombus-Induced Vein Wall Inflammation and Thrombosis,. The Journal of Immunology. 161(3). 1471–1476. 80 indexed citations
8.
Downing, L. J., Thomas W. Wakefield, Robert M. Strieter, et al.. (1997). Anti-P-selectin antibody decreases inflammation and thrombus formation in venous thrombosis. Journal of Vascular Surgery. 25(5). 816–828. 91 indexed citations
9.
Wakefield, Thomas W., Robert M. Strieter, L. J. Downing, et al.. (1996). P-Selectin and TNF Inhibition Reduce Venous Thrombosis Inflammation. Journal of Surgical Research. 64(1). 26–31. 60 indexed citations
10.
Downing, L. J., Robert M. Strieter, Amy M. Kadell, et al.. (1996). Neutrophils Are the Initial Cell Type Identified in Deep Venous Thrombosis Induced Vein Wall Inflammation. ASAIO Journal. 42(5). M677–681. 46 indexed citations
11.
Wakefield, Thomas W., et al.. (1996). A [+18RGD] Protamine Variant for Nontoxic and Effective Reversal of Conventional Heparin and Low-Molecular- Weight Heparin Anticoagulation. Journal of Surgical Research. 63(1). 280–286. 27 indexed citations
12.
Downing, L. J., Robert M. Strieter, Amy M. Kadell, et al.. (1996). PMNs ARE THE INITIAL CELL TYPE IN VENOUS THROMBOSIS INDUCED VEIN WALL INFLAMMATION. ASAIO Journal. 42(2). 109–109. 1 indexed citations
13.
Wakefield, Thomas W., et al.. (1995). Effective and less toxic reversal of low-molecular weight heparin anticoagulation by a designer variant of protamine. Journal of Vascular Surgery. 21(5). 839–850. 11 indexed citations
14.
Wakefield, Thomas W., Robert M. Strieter, Carol A. Wilke, et al.. (1995). Venous Thrombosis–Associated Inflammation and Attenuation With Neutralizing Antibodies to Cytokines and Adhesion Molecules. Arteriosclerosis Thrombosis and Vascular Biology. 15(2). 258–268. 155 indexed citations
15.
Wakefield, Thomas W., et al.. (1994). Reversal of Low-Molecular-Weight Heparin Anticoagulation by Synthetic Protamine Analogues. Journal of Surgical Research. 56(6). 586–593. 18 indexed citations
16.
DeLucia, Alphonse, Thomas W. Wakefield, Philip Andrews, et al.. (1993). Efficacy and toxicity of differently charged polycationic protamine-like peptides for heparin anticoagulation reversal. Journal of Vascular Surgery. 18(1). 49–60. 26 indexed citations
17.
DeLucia, Alphonse, et al.. (1993). Tissue Distribution, Circulating Half-Life, and Excretion of Intravenously Administered Protamine Sulfate. ASAIO Journal. 39(3). M715–M718. 18 indexed citations
18.
DeLucia, Alphonse, et al.. (1993). Tissue Distribution, Circulating Half-Life, and Excretion of Intravenously Administered Protamine Sulfate. ASAIO Journal. 39(3). M715–M718. 9 indexed citations
19.
Wakefield, Thomas W., Lazar J. Greenfield, Mark W. Rolfe, et al.. (1993). Inflammatory and Procoagulant Mediator Interactions in an Experimental Baboon Model of Venous Thrombosis. Thrombosis and Haemostasis. 69(2). 164–172. 78 indexed citations
20.
Smith, Philip, et al.. (1992). Heparin-mediated reductions of the toxic effects of protamine sulfate on rabbit myocardium. Journal of Vascular Surgery. 16(1). 47–53. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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