Almudena Albillos
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Cell Biology top 2%
- Physiology top 10%
- Cardiology and Cardiovascular Medicine top 10%
- Co-authors
- Antonio G. Garcı́aLuis Gandı́aInmaculada Cuchillo‐IbáñezW. AlmersManfred LindauGregor DernickGuillermo Álvarez de ToledoHeinz Horstmann
- Topics
- Ion channel regulation and function (36 papers)Nicotinic Acetylcholine Receptors Study (23 papers)Neuroscience and Neuropharmacology Research (19 papers)
- Partner nations
- SpainUnited StatesGermany
In The Last Decade
Almudena Albillos
51 papers receiving 2.2k citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 1.9k
- Cellular and Molecular Neuroscience 1.2k
- Cell Biology 591
- Physiology 221
- Cardiology and Cardiovascular Medicine 174
Countries citing papers authored by Almudena Albillos
This map shows the geographic impact of Almudena Albillos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Almudena Albillos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Almudena Albillos more than expected).
Fields of papers citing papers by Almudena Albillos
This network shows the impact of papers produced by Almudena Albillos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Almudena Albillos. The network helps show where Almudena Albillos may publish in the future.
Co-authorship network of co-authors of Almudena Albillos
This figure shows the co-authorship network connecting the top 25 collaborators of Almudena Albillos. A scholar is included among the top collaborators of Almudena Albillos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Almudena Albillos. Almudena Albillos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 4 | |
| 3 | 27 | |
| 4 | 5 | |
| 5 | 1 | |
| 6 | 18 | |
| 7 | 27 | |
| 8 | 11 | |
| 9 | 13 | |
| 10 | 23 | |
| 11 | 6 | |
| 12 | 17 | |
| 13 | 462 | |
| 14 | 7 | |
| 15 | 16 | |
| 16 | 58 | |
| 17 | 63 | |
| 18 | 58 | |
| 19 | 70 | |
| 20 | 5 |
About Almudena Albillos
Almudena Albillos is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cell Biology, having authored 51 papers that have together received 2.2k indexed citations. Recurring topics across this work include Ion channel regulation and function (36 papers), Nicotinic Acetylcholine Receptors Study (23 papers) and Neuroscience and Neuropharmacology Research (19 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.2k citations), Cell Biology (591 citations) and Physiology (135 citations). Almudena Albillos has collaborated with scholars based in Spain, United States and Germany. Frequent co-authors include Antonio G. Garcı́a, Luis Gandı́a, Inmaculada Cuchillo‐Ibáñez, W. Almers, Manfred Lindau, Gregor Dernick, Guillermo Álvarez de Toledo, Heinz Horstmann, Mayte Montero and Javier Álvarez. Their work appears in journals such as Nature, Journal of Neuroscience and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.