Alison H. Hammond

536 total citations
27 papers, 463 citations indexed

About

Alison H. Hammond is a scholar working on Pharmacology, Plant Science and Biochemistry. According to data from OpenAlex, Alison H. Hammond has authored 27 papers receiving a total of 463 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Pharmacology, 8 papers in Plant Science and 7 papers in Biochemistry. Recurrent topics in Alison H. Hammond's work include Pharmacogenetics and Drug Metabolism (16 papers), Drug-Induced Hepatotoxicity and Protection (9 papers) and Pesticide Exposure and Toxicity (7 papers). Alison H. Hammond is often cited by papers focused on Pharmacogenetics and Drug Metabolism (16 papers), Drug-Induced Hepatotoxicity and Protection (9 papers) and Pesticide Exposure and Toxicity (7 papers). Alison H. Hammond collaborates with scholars based in United Kingdom, Türkiye and Ukraine. Alison H. Hammond's co-authors include Jeffrey R. Fry, Michael J. Garle, Andrew L. Lewis, Rena Bhandari, M.J. Garle, Kevin M. Shakesheff, Saul J. B. Tendler, Julia H. Fentem, Alan K. Hatfield and Norman W. Thomas and has published in prestigious journals such as Biochemical Pharmacology, Toxicology and Applied Pharmacology and Toxicology.

In The Last Decade

Alison H. Hammond

26 papers receiving 449 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Alison H. Hammond United Kingdom 12 142 102 95 94 89 27 463
Michiel W. den Braver Netherlands 9 131 0.9× 129 1.3× 53 0.6× 209 2.2× 156 1.8× 12 508
Rozenn Jossé France 13 129 0.9× 56 0.5× 21 0.2× 50 0.5× 332 3.7× 18 698
Steven Wink Netherlands 11 136 1.0× 48 0.5× 17 0.2× 37 0.4× 278 3.1× 20 518
Yukinori Mera Japan 11 77 0.5× 37 0.4× 75 0.8× 9 0.1× 341 3.8× 26 599
Hans-Juergen Ahr Germany 17 73 0.5× 9 0.1× 18 0.2× 55 0.6× 245 2.8× 23 595
WJ Angerson United Kingdom 7 23 0.2× 52 0.5× 19 0.2× 25 0.3× 133 1.5× 10 446
Masako Muguruma Japan 12 83 0.6× 7 0.1× 19 0.2× 23 0.2× 237 2.7× 28 425
Ji‐Eun Seo United States 13 33 0.2× 14 0.1× 11 0.1× 28 0.3× 160 1.8× 28 349
Somaya Z. Mansour Egypt 10 35 0.2× 18 0.2× 15 0.2× 33 0.4× 121 1.4× 32 331
Mohammad Akbar Hossain South Korea 10 38 0.3× 29 0.3× 24 0.3× 9 0.1× 192 2.2× 12 417

Countries citing papers authored by Alison H. Hammond

Since Specialization
Citations

This map shows the geographic impact of Alison H. Hammond's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alison H. Hammond with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alison H. Hammond more than expected).

Fields of papers citing papers by Alison H. Hammond

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alison H. Hammond. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alison H. Hammond. The network helps show where Alison H. Hammond may publish in the future.

Co-authorship network of co-authors of Alison H. Hammond

This figure shows the co-authorship network connecting the top 25 collaborators of Alison H. Hammond. A scholar is included among the top collaborators of Alison H. Hammond based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alison H. Hammond. Alison H. Hammond is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Hammond, Alison H., et al.. (2002). Modulation of hepatocyte thiol content by medium composition: implications for toxicity studies. Toxicology in Vitro. 16(3). 259–265. 9 indexed citations
3.
Bhandari, Rena, Andrew L. Lewis, Jeffrey R. Fry, et al.. (2001). Liver Tissue Engineering: A Role for Co-culture Systems in Modifying Hepatocyte Function and Viability. Tissue Engineering. 7(3). 345–357. 134 indexed citations
4.
Hammond, Alison H., Michael J. Garle, & Jeffrey R. Fry. (1999). The Nature of Halogen Substitution Determines the Mode of Cytotoxicity of Halopropanols. Toxicology and Applied Pharmacology. 155(3). 287–291. 10 indexed citations
5.
Hammond, Alison H. & Jeffrey R. Fry. (1999). Effect of cyanamide on toxicity and glutathione depletion in rat hepatocyte cultures: differences between two dichloropropanol isomers. Chemico-Biological Interactions. 122(2). 107–115. 26 indexed citations
6.
Hammond, Alison H. & Jeffrey R. Fry. (1997). Involvement of cytochrome P4502E1 in the toxicity of dichloropropanol to rat hepatocyte cultures. Toxicology. 118(2-3). 171–179. 24 indexed citations
7.
Hammond, Alison H., Michael J. Garle, & Jeffrey R. Fry. (1996). Toxicity of dichloropropanols in rat hepatocyte cultues. Environmental Toxicology and Pharmacology. 1(1). 39–43. 28 indexed citations
8.
Hammond, Alison H. & Jeffrey R. Fry. (1996). Effects of culture duration, cytochrome P-450 inhibition and glutathione depletion on toxicity of diverse xenobiotics. Toxicology in Vitro. 10(3). 315–321. 10 indexed citations
9.
Hammond, Alison H., Michael J. Garle, & Jeffrey R. Fry. (1995). Mechanism of toxicity of precocene II in rat hepatocyte cultures. Journal of Biochemical Toxicology. 10(5). 265–273. 17 indexed citations
10.
Hammond, Alison H. & Jeffrey R. Fry. (1994). Toxicity of precocene II in rat hepatocyte cultures: effects of serum and culture time. Toxicology Letters. 70(3). 337–342. 11 indexed citations
12.
Hammond, Alison H. & Jeffrey R. Fry. (1993). Maintenance of xenobiotic metabolism and toxicity in rat hepatocyte cultures after cell preservation at 4°. Biochemical Pharmacology. 46(2). 333–335. 15 indexed citations
14.
Fentem, Julia H., Alison H. Hammond, M.J. Garle, & Jeffrey R. Fry. (1992). Toxicity of coumarin and various methyl derivatives in cultures of rat hepatocytes and V79 cells. Toxicology in Vitro. 6(1). 21–25. 32 indexed citations
15.
Hammond, Alison H. & Jeffrey R. Fry. (1992). Effect of serum-free medium on cytochrome P450-dependent metabolism and toxicity in rat cultured hepatocytes. Biochemical Pharmacology. 44(7). 1461–1464. 24 indexed citations
16.
Hammond, Alison H. & Jeffrey R. Fry. (1991). The use of hepatocytes cultured from inducer-treated rats in the detection of cytochrome P-450-mediated cytotoxicity. Toxicology in Vitro. 5(2). 133–137. 9 indexed citations
17.
Fentem, Julia H., Alison H. Hammond, & Jeffrey R. Fry. (1991). Maintenance of monooxygenase activities and detection of cytochrome P-450-mediated cytotoxicity in Mongolian gerbil hepatocyte cultures. Xenobiotica. 21(10). 1363–1370. 3 indexed citations
18.
Fry, Jeffrey R., M.J. Garle, Alison H. Hammond, & Alan K. Hatfield. (1990). Correlation of acute lethal potency with in vitro cytotoxicity. Toxicology in Vitro. 4(3). 175–178. 27 indexed citations
19.
Hammond, Alison H. & Jeffrey R. Fry. (1990). The in vivo induction of rat hepatic cytochrome P450-dependent enzyme activities and their maintenance in culture. Biochemical Pharmacology. 40(3). 637–642. 19 indexed citations
20.
Fry, Jeffrey R., M.J. Garle, & Alison H. Hammond. (1990). Competing pathways in metabolism-mediated cytotoxicity in vitro. Toxicology in Vitro. 4(4-5). 467–473. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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