Alicia Salcedo

739 citations
8 papers · 604 indexed · h-index 7
Topics
Receptor Mechanisms and Signaling (5 papers)Protein Kinase Regulation and GTPase Signaling (4 papers)Neuropeptides and Animal Physiology (2 papers)

In The Last Decade

Alicia Salcedo

8 papers receiving 601 citations

Peers

Alicia Salcedo
Comparison fields: 5 of 72
  • Molecular Biology 487
  • Cellular and Molecular Neuroscience 169
  • Oncology 98
  • Physiology 67
  • Immunology 59
Replace А. Г. Бабич with:
А. Г. Бабич Germany
Guillermo Romero United States
Etrusca D’Urbano Italy
Yumiko Moritani Japan
Kevin M. Kaltenbronn United States
Maria Zoudilova United States
Jonathan M. Dermott United States
Елена Жукова United States
Michelle Connell United Kingdom
Francis Amara Canada
Alicia Salcedo relative to А. Г. Бабич Germany А. Г. Бабич's profile →
Citations per field
00.5×3.3×
А. Г. Бабич · 1×
Citations per year

Countries citing papers authored by Alicia Salcedo

Since Specialization
Citations

This map shows the geographic impact of Alicia Salcedo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alicia Salcedo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alicia Salcedo more than expected).

Fields of papers citing papers by Alicia Salcedo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alicia Salcedo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alicia Salcedo. The network helps show where Alicia Salcedo may publish in the future.

Co-authorship network of co-authors of Alicia Salcedo

This figure shows the co-authorship network connecting the top 25 collaborators of Alicia Salcedo. A scholar is included among the top collaborators of Alicia Salcedo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alicia Salcedo. Alicia Salcedo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
#WorkIndexed citations
1 56
2 2
3 26
4 68
5 15
6 17
7 105
8 315

About Alicia Salcedo

Alicia Salcedo is a scholar working on Physiology, Immunology and Allergy and Cellular and Molecular Neuroscience, having authored 8 papers that have together received 604 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (5 papers), Protein Kinase Regulation and GTPase Signaling (4 papers) and Neuropeptides and Animal Physiology (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (169 citations), Immunology and Allergy (43 citations) and Molecular Biology (487 citations). Alicia Salcedo has collaborated with scholars based in Spain, United States and Germany. Frequent co-authors include Petronila Penela, Federico Mayor, Cristina Murga, María Jurado-Pueyo, Ivette Aymerich, Catalina Ribas, Carlota García‐Hoz, Verónica Rivas, Laura Nogués and Vanesa Lafarga. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The EMBO Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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