Alfons Navarro
- Molecular Biology top 2%
- Cancer Research top 0.5%
- Oncology top 5%
- Pathology and Forensic Medicine top 5%
- Immunology top 10%
- Co-authors
- Mariano MonzóJesús García‐FoncillasTania DíazEva BandrésBernat GelNúria ViñolasRamón M. MarradesI. Moreno
- Topics
- Cancer-related molecular mechanisms research (40 papers)MicroRNA in disease regulation (35 papers)Circular RNAs in diseases (24 papers)
- Partner nations
- SpainUnited StatesChina
In The Last Decade
Alfons Navarro
101 papers receiving 4.0k citations
Hit Papers
Peers
Comparison fields: 5 of 107
- Molecular Biology 3.1k
- Cancer Research 2.7k
- Oncology 522
- Pathology and Forensic Medicine 311
- Immunology 292
Countries citing papers authored by Alfons Navarro
This map shows the geographic impact of Alfons Navarro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alfons Navarro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alfons Navarro more than expected).
Fields of papers citing papers by Alfons Navarro
This network shows the impact of papers produced by Alfons Navarro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alfons Navarro. The network helps show where Alfons Navarro may publish in the future.
Co-authorship network of co-authors of Alfons Navarro
This figure shows the co-authorship network connecting the top 25 collaborators of Alfons Navarro. A scholar is included among the top collaborators of Alfons Navarro based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alfons Navarro. Alfons Navarro is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 9 | |
| 4 | 4 | |
| 5 | 6 | |
| 6 | 8 | |
| 7 | 41 | |
| 8 | 1 | |
| 9 | 20 | |
| 10 | 16 | |
| 11 | 47 | |
| 12 | 64 | |
| 13 | 62 | |
| 14 | 6 | |
| 15 | 50 | |
| 16 | 79 | |
| 17 | 28 | |
| 18 | 50 | |
| 19 | Effect of NOD2/CARD15 variants in T-cell depleted allogeneic stem cell transplantation. | 51 |
| 20 | Identification by Real-time PCR of 13 mature microRNAs differentially expressed in colorectal cancer and non-tumoral tissues.breakdown → | 706 |
About Alfons Navarro
Alfons Navarro is a scholar working on Cancer Research, Hematology and Molecular Biology, having authored 102 papers that have together received 4.0k indexed citations. Recurring topics across this work include Cancer-related molecular mechanisms research (40 papers), MicroRNA in disease regulation (35 papers) and Circular RNAs in diseases (24 papers). The work is most often cited by research in Cancer Research (2.7k citations), Molecular Biology (3.1k citations) and Hematology (273 citations). Alfons Navarro has collaborated with scholars based in Spain, United States and China. Frequent co-authors include Mariano Monzó, Jesús García‐Foncillas, Tania Díaz, Eva Bandrés, Bernat Gel, Núria Viñolas, Ramón M. Marrades, I. Moreno, Ruth Zárate and Natalia Ramírez. Their work appears in journals such as Journal of Clinical Oncology, Blood and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.