Alexander K. Haas
- Molecular Biology top 10%
- Cell Biology top 1%
- Genetics top 10%
- Physiology
- Surgery
- Co-authors
- Francis A. BarrEvelyn FuchsShin‐ichiro YoshimuraBenjamin ShortRobert KopajtichJohannes EgererChristian PreisingerDavid Stephens
- Topics
- Cellular transport and secretion (7 papers)Glycosylation and Glycoproteins Research (6 papers)Monoclonal and Polyclonal Antibodies Research (6 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryThe Journal of Cell Biology
- Partner nations
- GermanySwitzerlandUnited Kingdom
In The Last Decade
Alexander K. Haas
23 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 98
- Molecular Biology 1.1k
- Cell Biology 965
- Genetics 282
- Physiology 148
- Surgery 143
Countries citing papers authored by Alexander K. Haas
This map shows the geographic impact of Alexander K. Haas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexander K. Haas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexander K. Haas more than expected).
Fields of papers citing papers by Alexander K. Haas
This network shows the impact of papers produced by Alexander K. Haas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexander K. Haas. The network helps show where Alexander K. Haas may publish in the future.
Co-authorship network of co-authors of Alexander K. Haas
This figure shows the co-authorship network connecting the top 25 collaborators of Alexander K. Haas. A scholar is included among the top collaborators of Alexander K. Haas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexander K. Haas. Alexander K. Haas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 14 | |
| 2 | 10 | |
| 3 | 31 | |
| 4 | 17 | |
| 5 | 2 | |
| 6 | 16 | |
| 7 | 28 | |
| 8 | 15 | |
| 9 | 11 | |
| 10 | 82 | |
| 11 | 8 | |
| 12 | 117 | |
| 13 | 293 | |
| 14 | 3 | |
| 15 | 172 | |
| 16 | 191 | |
| 17 | 182 | |
| 18 | 221 | |
| 19 | 3 | |
| 20 | 1 |
About Alexander K. Haas
Alexander K. Haas is a scholar working on Aging, Cell Biology and Molecular Biology, having authored 23 papers that have together received 1.7k indexed citations. Recurring topics across this work include Cellular transport and secretion (7 papers), Glycosylation and Glycoproteins Research (6 papers) and Monoclonal and Polyclonal Antibodies Research (6 papers). The work is most often cited by research in Cell Biology (965 citations), Physiology (138 citations) and Molecular Biology (1.1k citations). Alexander K. Haas has collaborated with scholars based in Germany, Switzerland and United Kingdom. Frequent co-authors include Francis A. Barr, Evelyn Fuchs, Shin‐ichiro Yoshimura, Benjamin Short, Robert Kopajtich, Johannes Egerer, Christian Preisinger, David Stephens, C. Bogedain and Erik Bruyneel. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.