Alexander J.R. Bishop
- Molecular Biology top 5%
- Oncology top 5%
- Cancer Research top 5%
- Epidemiology top 10%
- Cell Biology top 10%
- Co-authors
- Robert H. SchiestlAparna GorthiYidong ChenMarı́a M. FergussonLiu CaoYoshichika KawaiIlsa I. RoviraNoboru Motoyama
- Topics
- DNA Repair Mechanisms (30 papers)CRISPR and Genetic Engineering (11 papers)Cancer-related Molecular Pathways (10 papers)
- Partner nations
- United StatesBrazilUnited Kingdom
In The Last Decade
Alexander J.R. Bishop
83 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 122
- Molecular Biology 1.7k
- Oncology 475
- Cancer Research 453
- Epidemiology 327
- Cell Biology 172
Countries citing papers authored by Alexander J.R. Bishop
This map shows the geographic impact of Alexander J.R. Bishop's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexander J.R. Bishop with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexander J.R. Bishop more than expected).
Fields of papers citing papers by Alexander J.R. Bishop
This network shows the impact of papers produced by Alexander J.R. Bishop. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexander J.R. Bishop. The network helps show where Alexander J.R. Bishop may publish in the future.
Co-authorship network of co-authors of Alexander J.R. Bishop
This figure shows the co-authorship network connecting the top 25 collaborators of Alexander J.R. Bishop. A scholar is included among the top collaborators of Alexander J.R. Bishop based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexander J.R. Bishop. Alexander J.R. Bishop is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 10 | |
| 3 | 1 | |
| 4 | 1 | |
| 5 | 11 | |
| 6 | 12 | |
| 7 | 37 | |
| 8 | 62 | |
| 9 | 30 | |
| 10 | 52 | |
| 11 | 55 | |
| 12 | 49 | |
| 13 | 274 | |
| 14 | 33 | |
| 15 | 24 | |
| 16 | 41 | |
| 17 | Atm-, p53-, and Gadd45a-deficient mice show an increased frequency of homologous recombination at different stages during development. | 44 |
| 18 | 95 | |
| 19 | 65 | |
| 20 | 29 |
About Alexander J.R. Bishop
Alexander J.R. Bishop is a scholar working on Molecular Biology, Cancer Research and Oncology, having authored 86 papers that have together received 2.4k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (30 papers), CRISPR and Genetic Engineering (11 papers) and Cancer-related Molecular Pathways (10 papers). The work is most often cited by research in Cancer Research (453 citations), Molecular Biology (1.7k citations) and Oncology (475 citations). Alexander J.R. Bishop has collaborated with scholars based in United States, Brazil and United Kingdom. Frequent co-authors include Robert H. Schiestl, Aparna Gorthi, Yidong Chen, Marı́a M. Fergusson, Liu Cao, Yoshichika Kawai, Ilsa I. Rovira, Noboru Motoyama, Toren Finkel and In Hye Lee. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.