Alan Mackay

19.7k total citations · 3 hit papers
134 papers, 10.1k citations indexed

About

Alan Mackay is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Alan Mackay has authored 134 papers receiving a total of 10.1k indexed citations (citations by other indexed papers that have themselves been cited), including 78 papers in Molecular Biology, 50 papers in Cancer Research and 43 papers in Oncology. Recurrent topics in Alan Mackay's work include Cancer Genomics and Diagnostics (29 papers), Breast Cancer Treatment Studies (23 papers) and Glioma Diagnosis and Treatment (20 papers). Alan Mackay is often cited by papers focused on Cancer Genomics and Diagnostics (29 papers), Breast Cancer Treatment Studies (23 papers) and Glioma Diagnosis and Treatment (20 papers). Alan Mackay collaborates with scholars based in United Kingdom, United States and Spain. Alan Mackay's co-authors include Jorge S. Reis‐Filho, Rachael Natrajan, Alan Ashworth, Chris Jones, Felipe C. Geyer, Anita Grigoriadis, Alan Ashworth, Kay Savage, Maryou B. Lambros and Kerry Fenwick and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Alan Mackay

134 papers receiving 9.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

BRCA1 Basal-like Breast Cancers Originate from Luminal Epithelial Progenitors and Not from Basal Stem Cells

2010 557 citations Felipe C. Geyer, Howard Kendrick et al. profile →
FGFR1 Amplification Drives Endocrine Therapy Resistance and Is a Therapeutic Target in Breast Cancer

2010 544 citations Nicholas C. Turner, Alex Pearson et al. Cancer Research profile →
Histone H3F3A and HIST1H3B K27M mutations define two subgroups of diffuse intrinsic pontine gliomas with different prognosis and phenotypes

2015 426 citations Kathryn R. Taylor, Alan Mackay et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Alan Mackay United Kingdom	56	5.5k	3.9k	3.5k	1.6k	1.4k	134	10.1k
Shikha Bose United States	43	6.3k 1.1×	2.6k 0.7×	2.1k 0.6×	1.6k 1.0×	1.1k 0.8×	109	10.2k
Leif W. Ellisen United States	54	9.4k 1.7×	4.9k 1.3×	3.9k 1.1×	1.1k 0.7×	2.1k 1.5×	152	15.9k
Jiřina Bártková Denmark	62	10.5k 1.9×	7.8k 2.0×	2.8k 0.8×	1.3k 0.8×	1.8k 1.3×	136	15.4k
Cheryl Gillett United Kingdom	45	4.2k 0.8×	4.0k 1.0×	2.4k 0.7×	1.3k 0.8×	955 0.7×	123	8.5k
Robert L. Yauch United States	40	6.9k 1.3×	3.2k 0.8×	1.2k 0.3×	1.4k 0.9×	1.1k 0.8×	70	10.2k
José Palacios Spain	57	5.1k 0.9×	3.6k 0.9×	3.4k 1.0×	1.8k 1.1×	1.4k 1.0×	204	10.4k
Bryan D. Young United Kingdom	61	5.2k 0.9×	1.6k 0.4×	1.6k 0.5×	1.3k 0.8×	1.5k 1.1×	191	9.2k
Thierry Soussi France	62	7.5k 1.4×	7.9k 2.0×	3.2k 0.9×	1.6k 1.0×	1.1k 0.8×	174	13.1k
Cees J. Cornelisse Netherlands	55	5.1k 0.9×	3.5k 0.9×	4.4k 1.3×	2.4k 1.5×	2.6k 1.9×	201	12.4k
Poul H. Sorensen Canada	65	7.9k 1.4×	3.7k 1.0×	2.6k 0.7×	2.2k 1.4×	1.0k 0.7×	203	14.7k

Countries citing papers authored by Alan Mackay

Since Specialization

Citations

This map shows the geographic impact of Alan Mackay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alan Mackay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alan Mackay more than expected).

Fields of papers citing papers by Alan Mackay

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alan Mackay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alan Mackay. The network helps show where Alan Mackay may publish in the future.

Co-authorship network of co-authors of Alan Mackay

This figure shows the co-authorship network connecting the top 25 collaborators of Alan Mackay. A scholar is included among the top collaborators of Alan Mackay based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alan Mackay. Alan Mackay is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Mackay, Alan, Massimiliano Manzo, Ilaria Alborelli, et al.. (2021). The Long Non-Coding RNA H19 Drives the Proliferation of Diffuse Intrinsic Pontine Glioma with H3K27 Mutation. International Journal of Molecular Sciences. 22(17). 9165–9165. 6 indexed citations

Fons, Nathan R., Ranjini K. Sundaram, G. Breuer, et al.. (2019). PPM1D mutations silence NAPRT gene expression and confer NAMPT inhibitor sensitivity in glioma. Nature Communications. 10(1). 3790–3790. 51 indexed citations

Stewart, Grant D., Fiach C. O’Mahony, Alexander Laird, et al.. (2015). Sunitinib Treatment Exacerbates Intratumoral Heterogeneity in Metastatic Renal Cancer. Clinical Cancer Research. 21(18). 4212–4223. 29 indexed citations

Patani, Neill, Anita K. Dunbier, H Anderson, et al.. (2014). Differences in the Transcriptional Response to Fulvestrant and Estrogen Deprivation in ER-Positive Breast Cancer. Clinical Cancer Research. 20(15). 3962–3973. 16 indexed citations

Zvelebil, Marketa, Erik Oliemuller, Qiong Gao, et al.. (2013). Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers. Breast Cancer Research. 15(2). R25–R25. 51 indexed citations

Wetterskog, Daniel, Alan Mackay, Rachael Natrajan, et al.. (2013). Integrative molecular and functional profiling of ERBB2-amplified breast cancers identifies new genetic dependencies. Oncogene. 33(5). 619–631. 22 indexed citations

Bjerke, Lynn, Alan Mackay, Meera Nandhabalan, et al.. (2013). Histone H3.3 Mutations Drive Pediatric Glioblastoma through Upregulation of MYCN. Cancer Discovery. 3(5). 512–519. 226 indexed citations

Morandi, Andrea, Lesley‐Ann Martin, Qiong Gao, et al.. (2013). GDNF–RET Signaling in ER-Positive Breast Cancers Is a Key Determinant of Response and Resistance to Aromatase Inhibitors. Cancer Research. 73(12). 3783–3795. 83 indexed citations

Sharpe, Rachel, Alex Pearson, María Teresa Herrera-Abreu, et al.. (2011). FGFR Signaling Promotes the Growth of Triple-Negative and Basal-Like Breast Cancer Cell Lines Both In Vitro and In Vivo. Clinical Cancer Research. 17(16). 5275–5286. 144 indexed citations

10.

Patani, Neill, et al.. (2011). Direct evidence for concurrent morphological and genetic heterogeneity in an invasive ductal carcinoma of triple-negative phenotype. Journal of Clinical Pathology. 64(9). 822–828. 23 indexed citations

11.

Turner, Nicholas C., Alex Pearson, Rachel Sharpe, et al.. (2010). FGFR1 Amplification Drives Endocrine Therapy Resistance and Is a Therapeutic Target in Breast Cancer. Cancer Research. 70(5). 2085–2094. 544 indexed citations breakdown →

12.

Bax, Dorine A., Alan Mackay, Suzanne E. Little, et al.. (2010). A Distinct Spectrum of Copy Number Aberrations in Pediatric High-Grade Gliomas. Clinical Cancer Research. 16(13). 3368–3377. 110 indexed citations

13.

Geyer, Felipe C., Maryou B. Lambros, Rachael Natrajan, et al.. (2010). Genomic and immunohistochemical analysis of adenosquamous carcinoma of the breast. Modern Pathology. 23(7). 951–960. 41 indexed citations

14.

Mackay, Alan, Ander Urruticoechea, J. Michael Dixon, et al.. (2007). Molecular response to aromatase inhibitor treatment in primary breast cancer. Breast Cancer Research. 9(3). R37–R37. 97 indexed citations

15.

Reis‐Filho, Jorge S., Peter T. Simpson, Nicholas C. Turner, et al.. (2006). FGFR1 Emerges as a Potential Therapeutic Target for Lobular Breast Carcinomas. Clinical Cancer Research. 12(22). 6652–6662. 215 indexed citations

16.

Docquier, France, Dawn Farrar, Vivien D'Arcy, et al.. (2005). Heightened Expression of CTCF in Breast Cancer Cells Is Associated with Resistance to Apoptosis. Cancer Research. 65(12). 5112–5122. 83 indexed citations

17.

Jones, Chris, Alan Mackay, Anita Grigoriadis, et al.. (2004). Expression Profiling of Purified Normal Human Luminal and Myoepithelial Breast Cells. Cancer Research. 64(9). 3037–3045. 202 indexed citations

18.

Mackay, Alan, Chris Jones, Tim Dexter, et al.. (2003). cDNA microarray analysis of genes associated with ERBB2 (HER2/neu) overexpression in human mammary luminal epithelial cells. Oncogene. 22(17). 2680–2688. 135 indexed citations

19.

Jongeneel, C. Victor, Christian Iseli, Brian J. Stevenson, et al.. (2003). Comprehensive sampling of gene expression in human cell lines with massively parallel signature sequencing. Proceedings of the National Academy of Sciences. 100(8). 4702–4705. 87 indexed citations

20.

Jones, Carolyn, Alan Mackay, Anita Grigoriadis, et al.. (2003). Expression Profiling of Purified Normal Human Luminal and Myoepithelial Breast Cells: Identification of Novel Prognostic Markers for Breast Cancer. UCL Discovery (University College London). 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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