Alan J. Lewis
- Molecular Biology top 10%
- Immunology top 5%
- Pharmacology top 2%
- Physiology top 5%
- Organic Chemistry top 5%
- Co-authors
- Joseph ChangRichard P. CarlsonArend BouhuysP. EyreD.H. BrownJames S. DouglasChris PlumptonFilippo Volpe
- Topics
- Asthma and respiratory diseases (29 papers)Inflammatory mediators and NSAID effects (25 papers)Respiratory and Cough-Related Research (12 papers)
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Alan J. Lewis
134 papers receiving 2.8k citations
Peers
Comparison fields: 5 of 170
- Molecular Biology 844
- Immunology 653
- Pharmacology 538
- Physiology 520
- Organic Chemistry 336
Countries citing papers authored by Alan J. Lewis
This map shows the geographic impact of Alan J. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alan J. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alan J. Lewis more than expected).
Fields of papers citing papers by Alan J. Lewis
This network shows the impact of papers produced by Alan J. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alan J. Lewis. The network helps show where Alan J. Lewis may publish in the future.
Co-authorship network of co-authors of Alan J. Lewis
This figure shows the co-authorship network connecting the top 25 collaborators of Alan J. Lewis. A scholar is included among the top collaborators of Alan J. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alan J. Lewis. Alan J. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 4 | |
| 3 | 4 | |
| 4 | 2 | |
| 5 | 66 | |
| 6 | 7 | |
| 7 | 4 | |
| 8 | Pharmacological methods in the control of inflammation | 157 |
| 9 | Nonsteroidal anti-inflammatory drugs : mechanisms and clinical use | 41 |
| 10 | 7 | |
| 11 | 6 | |
| 12 | 0 | |
| 13 | Modulation of mouse ear edema by cyclooxygenase and lipoxygenase inhibitors | 1 |
| 14 | 27 | |
| 15 | 2 | |
| 16 | 4 | |
| 17 | 58 | |
| 18 | 5 | |
| 19 | An Empirical Assessment of Tax Mentality | 57 |
| 20 | 8 |
About Alan J. Lewis
Alan J. Lewis is a scholar working on Immunology and Allergy, Pharmacology and Physiology, having authored 139 papers that have together received 3.1k indexed citations. Recurring topics across this work include Asthma and respiratory diseases (29 papers), Inflammatory mediators and NSAID effects (25 papers) and Respiratory and Cough-Related Research (12 papers). The work is most often cited by research in Pharmacology (538 citations), Immunology (653 citations) and Immunology and Allergy (185 citations). Alan J. Lewis has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Joseph Chang, Richard P. Carlson, Arend Bouhuys, P. Eyre, D.H. Brown, James S. Douglas, Chris Plumpton, Filippo Volpe, Fabio Martinon and Barbara Maschera. Their work appears in journals such as Nature, Contemporary Sociology A Journal of Reviews and Nature Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.