Akiko Nakai

807 citations
20 papers · 569 indexed · h-index 9
Topics
Circadian rhythm and melatonin (6 papers)Receptor Mechanisms and Signaling (4 papers)T-cell and B-cell Immunology (3 papers)
Partner nations
JapanAustraliaGermany

In The Last Decade

Akiko Nakai

19 papers receiving 557 citations

Peers

Akiko Nakai
Comparison fields: 5 of 90
  • Molecular Biology 185
  • Endocrine and Autonomic Systems 119
  • Physiology 110
  • Neurology 98
  • Immunology 93
Replace Julie A. Wixey with:
Julie A. Wixey Australia
Kevin G. Burfeind United States
Christine Molinaro United States
Jennifer A. Stokes United States
Susumu Take Japan
Luitpold E. Miller Germany
Alejandro Villarreal Argentina
Marjolein A. van Maanen Netherlands
Jodie C.E. Hall United States
Michael J Cole Australia
Akiko Nakai relative to Julie A. Wixey Australia Julie A. Wixey's profile →
Citations per field
00.5×1.5×2.0×
Julie A. Wixey · 1×
Citations per year

Countries citing papers authored by Akiko Nakai

Since Specialization
Citations

This map shows the geographic impact of Akiko Nakai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Akiko Nakai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Akiko Nakai more than expected).

Fields of papers citing papers by Akiko Nakai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Akiko Nakai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Akiko Nakai. The network helps show where Akiko Nakai may publish in the future.

Co-authorship network of co-authors of Akiko Nakai

This figure shows the co-authorship network connecting the top 25 collaborators of Akiko Nakai. A scholar is included among the top collaborators of Akiko Nakai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Akiko Nakai. Akiko Nakai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 4
3 4
4 25
5 14
6 1
7 144
8 2
9 222
10 1
11 20
12 39
13 7
14 1
15 12
16 51
17 1
18 13
19 7
20 1

About Akiko Nakai

Akiko Nakai is a scholar working on Endocrine and Autonomic Systems, Biological Psychiatry and Behavioral Neuroscience, having authored 20 papers that have together received 569 indexed citations. Recurring topics across this work include Circadian rhythm and melatonin (6 papers), Receptor Mechanisms and Signaling (4 papers) and T-cell and B-cell Immunology (3 papers). The work is most often cited by research in Endocrine and Autonomic Systems (119 citations), Behavioral Neuroscience (43 citations) and Neurology (98 citations). Akiko Nakai has collaborated with scholars based in Japan, Australia and Germany. Frequent co-authors include Kazuhiro Suzuki, Masaki Noda, Yuki Hayano, Katsumi Iwase, Masaki Shimizu, Yoshihito Kasahara, Hidetoshi Seki, Rikio Shinohara, Kohei Uchimura and Shoichi Koizumi. Their work appears in journals such as The Journal of Experimental Medicine, Gastroenterology and Kidney International.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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