Ainhoa Letamendı́a
- Molecular Biology top 10%
- Pulmonary and Respiratory Medicine top 10%
- Oncology top 10%
- Genetics top 5%
- Cancer Research top 10%
- Co-authors
- Liliana AttisanoÉtienne LabbéCarmelo BernabéuPedro LastresCarmen LangaUlla RaabMercedes Guerrero-EsteoTilman Sánchez-Elsner
- Topics
- TGF-β signaling in diseases (5 papers)Vascular Anomalies and Treatments (4 papers)Peptidase Inhibition and Analysis (3 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryThe Journal of Cell Biology
- Partner nations
- SpainCanadaUnited States
In The Last Decade
Ainhoa Letamendı́a
16 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 91
- Molecular Biology 1.1k
- Pulmonary and Respiratory Medicine 264
- Oncology 262
- Genetics 219
- Cancer Research 203
Countries citing papers authored by Ainhoa Letamendı́a
This map shows the geographic impact of Ainhoa Letamendı́a's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ainhoa Letamendı́a with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ainhoa Letamendı́a more than expected).
Fields of papers citing papers by Ainhoa Letamendı́a
This network shows the impact of papers produced by Ainhoa Letamendı́a. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ainhoa Letamendı́a. The network helps show where Ainhoa Letamendı́a may publish in the future.
Co-authorship network of co-authors of Ainhoa Letamendı́a
This figure shows the co-authorship network connecting the top 25 collaborators of Ainhoa Letamendı́a. A scholar is included among the top collaborators of Ainhoa Letamendı́a based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ainhoa Letamendı́a. Ainhoa Letamendı́a is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 31 | |
| 3 | 76 | |
| 4 | 1 | |
| 5 | 138 | |
| 6 | 97 | |
| 7 | 184 | |
| 8 | 97 | |
| 9 | 355 | |
| 10 | 15 | |
| 11 | 86 | |
| 12 | 36 | |
| 13 | 192 | |
| 14 | 31 | |
| 15 | 10 | |
| 16 | 262 |
About Ainhoa Letamendı́a
Ainhoa Letamendı́a is a scholar working on Genetics, Immunology and Allergy and Cell Biology, having authored 16 papers that have together received 1.6k indexed citations. Recurring topics across this work include TGF-β signaling in diseases (5 papers), Vascular Anomalies and Treatments (4 papers) and Peptidase Inhibition and Analysis (3 papers). The work is most often cited by research in Genetics (219 citations), Immunology and Allergy (109 citations) and Molecular Biology (1.1k citations). Ainhoa Letamendı́a has collaborated with scholars based in Spain, Canada and United States. Frequent co-authors include Liliana Attisano, Étienne Labbé, Carmelo Bernabéu, Pedro Lastres, Carmen Langa, Ulla Raab, Mercedes Guerrero-Esteo, Tilman Sánchez-Elsner, Àngels Fabra and Michelle Letarte. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.