Ainhoa Letamendı́a

1.9k total citations
16 papers, 1.6k citations indexed

About

Ainhoa Letamendı́a is a scholar working on Molecular Biology, Genetics and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Ainhoa Letamendı́a has authored 16 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Ainhoa Letamendı́a's work include TGF-β signaling in diseases (5 papers), Vascular Anomalies and Treatments (4 papers) and Peptidase Inhibition and Analysis (3 papers). Ainhoa Letamendı́a is often cited by papers focused on TGF-β signaling in diseases (5 papers), Vascular Anomalies and Treatments (4 papers) and Peptidase Inhibition and Analysis (3 papers). Ainhoa Letamendı́a collaborates with scholars based in Spain, Canada and United States. Ainhoa Letamendı́a's co-authors include Liliana Attisano, Étienne Labbé, Carmelo Bernabéu, Pedro Lastres, Carmen Langa, Ulla Raab, Mercedes Guerrero-Esteo, Tilman Sánchez-Elsner, Àngels Fabra and Michelle Letarte and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.

In The Last Decade

Ainhoa Letamendı́a

16 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ainhoa Letamendı́a Spain 13 1.1k 264 262 219 203 16 1.6k
Dianne Mitchell United States 17 814 0.7× 146 0.6× 192 0.7× 252 1.2× 178 0.9× 28 1.4k
Richard Charlton United Kingdom 16 854 0.8× 395 1.5× 248 0.9× 289 1.3× 204 1.0× 24 1.5k
Keiko Funa Sweden 20 716 0.6× 222 0.8× 609 2.3× 118 0.5× 215 1.1× 37 1.6k
Linda Chang Canada 15 1.0k 0.9× 204 0.8× 229 0.9× 87 0.4× 263 1.3× 19 1.4k
Dan Hicklin United States 10 1.3k 1.2× 204 0.8× 473 1.8× 131 0.6× 423 2.1× 21 1.9k
Gregg P. Solar United States 8 760 0.7× 130 0.5× 268 1.0× 288 1.3× 159 0.8× 8 1.7k
Beatriz Velasco Spain 14 685 0.6× 199 0.8× 196 0.7× 188 0.9× 284 1.4× 24 1.2k
Yuan Zhu Germany 19 802 0.7× 284 1.1× 320 1.2× 166 0.8× 155 0.8× 48 2.0k
Rachel Krasich United States 5 941 0.8× 166 0.6× 223 0.9× 278 1.3× 202 1.0× 5 1.2k
Frances Cousins United Kingdom 10 914 0.8× 127 0.5× 171 0.7× 89 0.4× 144 0.7× 14 1.4k

Countries citing papers authored by Ainhoa Letamendı́a

Since Specialization
Citations

This map shows the geographic impact of Ainhoa Letamendı́a's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ainhoa Letamendı́a with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ainhoa Letamendı́a more than expected).

Fields of papers citing papers by Ainhoa Letamendı́a

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ainhoa Letamendı́a. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ainhoa Letamendı́a. The network helps show where Ainhoa Letamendı́a may publish in the future.

Co-authorship network of co-authors of Ainhoa Letamendı́a

This figure shows the co-authorship network connecting the top 25 collaborators of Ainhoa Letamendı́a. A scholar is included among the top collaborators of Ainhoa Letamendı́a based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ainhoa Letamendı́a. Ainhoa Letamendı́a is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Ball, Jonathan S., Maciej Trznadel, Manon Beekhuijzen, et al.. (2024). Biological variability hampers the use of skeletal staining methods in zebrafish embryo developmental toxicity assays. Reproductive Toxicology. 127. 108615–108615. 2 indexed citations
2.
Alday, Aintzane, et al.. (2014). Ionic channels underlying the ventricular action potential in zebrafish embryo. Pharmacological Research. 84. 26–31. 31 indexed citations
3.
Letamendı́a, Ainhoa, et al.. (2012). Development and Validation of an Automated High-Throughput System for Zebrafish In Vivo Screenings. PLoS ONE. 7(5). e36690–e36690. 76 indexed citations
4.
Letamendı́a, Ainhoa, et al.. (2010). Evaluation of zebrafish as a model to predict human hepatotoxicity. Toxicology Letters. 196. S214–S214. 1 indexed citations
5.
Labbé, Étienne, Lisa Lock, Ainhoa Letamendı́a, et al.. (2007). Transcriptional Cooperation between the Transforming Growth Factor-β and Wnt Pathways in Mammary and Intestinal Tumorigenesis. Cancer Research. 67(1). 75–84. 138 indexed citations
6.
Santibáñez, Juan F., Ainhoa Letamendı́a, Fernando Pérez‐Barriocanal, et al.. (2006). Endoglin increases eNOS expression by modulating Smad2 protein levels and Smad2‐dependent TGF‐β signaling. Journal of Cellular Physiology. 210(2). 456–468. 97 indexed citations
7.
Guerrero-Esteo, Mercedes, Tilman Sánchez-Elsner, Ainhoa Letamendı́a, & Carmelo Bernabéu. (2002). Extracellular and Cytoplasmic Domains of Endoglin Interact with the Transforming Growth Factor-β Receptors I and II. Journal of Biological Chemistry. 277(32). 29197–29209. 184 indexed citations
8.
Letamendı́a, Ainhoa, Étienne Labbé, & Liliana Attisano. (2001). Transcriptional Regulation by Smads: Crosstalk between the TGF-β and Wnt Pathways. Journal of Bone and Joint Surgery. 83(Pt 1). S1–31–S1–39. 97 indexed citations
9.
Labbé, Étienne, Ainhoa Letamendı́a, & Liliana Attisano. (2000). Association of Smads with lymphoid enhancer binding factor 1/T cell-specific factor mediates cooperative signaling by the transforming growth factor-β and Wnt pathways. Proceedings of the National Academy of Sciences. 97(15). 8358–8363. 355 indexed citations
10.
Velasco, Beatriz, Pedro Lastres, Ainhoa Letamendı́a, et al.. (1999). Expression of normal and truncated forms of human endoglin. Biochemical Journal. 339(3). 579–579. 15 indexed citations
11.
Guerrero-Esteo, Mercedes, Pedro Lastres, Ainhoa Letamendı́a, et al.. (1999). Endoglin overexpression modulates cellular morphology, migration, and adhesion of mouse fibroblasts. European Journal of Cell Biology. 78(9). 614–623. 86 indexed citations
12.
Raab, Ulla, Beatriz Velasco, Pedro Lastres, et al.. (1999). Expression of normal and truncated forms of human endoglin. Biochemical Journal. 339(3). 579–588. 36 indexed citations
13.
Letamendı́a, Ainhoa, Pedro Lastres, Luisa M. Botella, et al.. (1998). Role of Endoglin in Cellular Responses to Transforming Growth Factor-β. Journal of Biological Chemistry. 273(49). 33011–33019. 192 indexed citations
14.
Letamendı́a, Ainhoa, Pedro Lastres, Nuria Almendro, et al.. (1998). Endoglin, a component of the TGF-β receptor system, is a differentiation marker of human choriocarcinoma cells. International Journal of Cancer. 76(4). 541–546. 31 indexed citations
15.
Luque, Alfonso, Carlos Cabañas, Ulla Raab, et al.. (1997). The use of recombinant vaccinia virus to generate monoclonal antibodies against the cell‐surface glycoprotein endoglin. FEBS Letters. 413(2). 265–268. 10 indexed citations
16.
Lastres, Pedro, Ainhoa Letamendı́a, Carlos Rı́us, et al.. (1996). Endoglin modulates cellular responses to TGF-beta 1.. The Journal of Cell Biology. 133(5). 1109–1121. 262 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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