José M. López‐Novoa

15.8k total citations · 3 hit papers
362 papers, 12.9k citations indexed

About

José M. López‐Novoa is a scholar working on Nephrology, Molecular Biology and Physiology. According to data from OpenAlex, José M. López‐Novoa has authored 362 papers receiving a total of 12.9k indexed citations (citations by other indexed papers that have themselves been cited), including 108 papers in Nephrology, 101 papers in Molecular Biology and 74 papers in Physiology. Recurrent topics in José M. López‐Novoa's work include Nitric Oxide and Endothelin Effects (57 papers), Chronic Kidney Disease and Diabetes (44 papers) and Liver Disease and Transplantation (39 papers). José M. López‐Novoa is often cited by papers focused on Nitric Oxide and Endothelin Effects (57 papers), Chronic Kidney Disease and Diabetes (44 papers) and Liver Disease and Transplantation (39 papers). José M. López‐Novoa collaborates with scholars based in Spain, France and United States. José M. López‐Novoa's co-authors include Francisco J. López‐Hernández, Carmelo Bernabéu, Ana I. Morales, M. Ángela Nieto, Carlos Martı́nez-Salgado, María T. Grande, Fernando Pérez‐Barriocanal, Miguel Arévalo, Alicia Rodríguez‐Barbero and Yaremi Quirós and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Journal of Clinical Investigation.

In The Last Decade

José M. López‐Novoa

356 papers receiving 12.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Snail1-induced partial epithelial-to-mesenchymal transition drives renal fibrosis in mice and can be targeted to reverse established disease

2015 622 citations María T. Grande, Berta Sanchez‐Laorden et al. Nature Medicine profile →
New insights into the mechanism of aminoglycoside nephrotoxicity: an integrative point of view

2010 562 citations José M. López‐Novoa et al. Kidney International profile →
Inflammation and EMT: an alliance towards organ fibrosis and cancer progression

2009 542 citations José M. López‐Novoa, M. Ángela Nieto profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
José M. López‐Novoa Spain	59	4.5k	2.7k	1.7k	1.4k	1.4k	362	12.9k
Hidenori Koyama Japan	68	5.9k 1.3×	1.8k 0.7×	1.3k 0.8×	1.6k 1.1×	1.7k 1.2×	414	15.1k
Gary E. Striker United States	58	2.2k 0.5×	3.0k 1.1×	1.2k 0.7×	1.8k 1.2×	1.3k 0.9×	150	13.5k
Fuad N. Ziyadeh United States	66	6.3k 1.4×	6.8k 2.5×	1.2k 0.7×	1.5k 1.0×	1.6k 1.1×	161	15.7k
Toshimitsu Niwa Japan	60	4.1k 0.9×	5.1k 1.9×	818 0.5×	1.9k 1.3×	1.2k 0.8×	311	13.0k
Karl A. Nath United States	72	7.0k 1.5×	3.8k 1.4×	1.9k 1.1×	1.7k 1.1×	1.8k 1.3×	219	15.7k
Anupam Agarwal United States	65	7.0k 1.5×	1.9k 0.7×	1.2k 0.7×	1.0k 0.7×	1.3k 0.9×	250	13.4k
Masakazu Haneda Japan	67	4.8k 1.1×	3.5k 1.3×	827 0.5×	2.3k 1.6×	2.7k 1.9×	292	16.1k
Glenda C. Gobé Australia	51	4.0k 0.9×	2.0k 0.8×	1.3k 0.7×	929 0.6×	1.0k 0.7×	297	11.3k
Carla Zoja Italy	69	3.4k 0.8×	4.8k 1.8×	1.2k 0.7×	2.0k 1.4×	2.0k 1.4×	192	13.3k
Takeshi Sugaya Japan	61	3.7k 0.8×	3.4k 1.3×	731 0.4×	821 0.6×	1.5k 1.0×	229	10.7k

Countries citing papers authored by José M. López‐Novoa

Since Specialization

Citations

This map shows the geographic impact of José M. López‐Novoa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José M. López‐Novoa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José M. López‐Novoa more than expected).

Fields of papers citing papers by José M. López‐Novoa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José M. López‐Novoa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José M. López‐Novoa. The network helps show where José M. López‐Novoa may publish in the future.

Co-authorship network of co-authors of José M. López‐Novoa

This figure shows the co-authorship network connecting the top 25 collaborators of José M. López‐Novoa. A scholar is included among the top collaborators of José M. López‐Novoa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José M. López‐Novoa. José M. López‐Novoa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Rodríguez‐Barbero, Alicia, et al.. (2020). Pregnancy-Induced High Plasma Levels of Soluble Endoglin in Mice Lead to Preeclampsia Symptoms and Placental Abnormalities. International Journal of Molecular Sciences. 22(1). 165–165. 21 indexed citations

2.

Gallardo‐Vara, Eunate, José Luis Bartha, Irene García‐Palmero, et al.. (2020). Potential Role of Circulating Endoglin in Hypertension via the Upregulated Expression of BMP4. Cells. 9(4). 988–988. 25 indexed citations

3.

Díaz‐Rodríguez, Elena, et al.. (2020). Continuous endoglin (CD105) overexpression disrupts angiogenesis and facilitates tumor cell metastasis. Angiogenesis. 23(2). 231–247. 30 indexed citations

4.

Olmos, Gemma, et al.. (2017). Impaired erythropoietin synthesis in chronic kidney disease is caused by alterations in extracellular matrix composition. Journal of Cellular and Molecular Medicine. 22(1). 302–314. 22 indexed citations

5.

Němečková, Ivana, Jana Rathouska, Carmelo Bernabéu, et al.. (2015). High Soluble Endoglin Levels Do Not Induce Endothelial Dysfunction in Mouse Aorta. PLoS ONE. 10(3). e0119665–e0119665. 20 indexed citations

6.

Grande, María T., Berta Sanchez‐Laorden, Cristina López-Blau, et al.. (2015). Snail1-induced partial epithelial-to-mesenchymal transition drives renal fibrosis in mice and can be targeted to reverse established disease. Nature Medicine. 21(9). 989–997. 622 indexed citations breakdown →

7.

López‐Novoa, José M., et al.. (2014). Soluble endoglin: a biomarker or a protagonist in the pathogenesis of preeclampsia?. Portuguese National Funding Agency for Science, Research and Technology (RCAAP Project by FCT). 28(3). 185–192. 3 indexed citations

8.

Tornavaca, Olga, Gloria Pascual, M. L. Barreiro, et al.. (2009). Kidney Androgen-Regulated Protein Transgenic Mice Show Hypertension and Renal Alterations Mediated by Oxidative Stress. Circulation. 119(14). 1908–1917. 26 indexed citations

9.

García‐Criado, Francisco Javier, et al.. (2009). Protective Effect of New Nitrosothiols on the Early Inflammatory Response to Kidney Ischemia/Reperfusion and Transplantation in Rats. Journal of Interferon & Cytokine Research. 29(8). 441–450. 8 indexed citations

10.

Eleno, N., et al.. (2006). Reduced Tumor Growth and Angiogenesis in Endoglin-Haploinsufficient Mice. Tumor Biology. 28(1). 1–8. 58 indexed citations

11.

Rodríguez-Peña, Ana B., Eugenio Santos, Miguel Arévalo, & José M. López‐Novoa. (2005). Activation of small GTPase Ras and renal fibrosis. Journal of Nephrology. 18(3). 341–349. 19 indexed citations

12.

Criado, Manuel, Ángel Sánchez‐Rodríguez, F. Hidalgo, et al.. (2005). Relationships between NOS2 and HO-1 in liver of rats with chronic bile duct ligation. Hepatology Research. 32(1). 58–65. 7 indexed citations

13.

Schanstra, Joost P., Eric Neau, José M. López‐Novoa, et al.. (2002). In vivo bradykinin B2 receptor activation reduces renal fibrosis. Journal of Clinical Investigation. 110(3). 371–379. 113 indexed citations

14.

Schanstra, Joost P., Eric Neau, José M. López‐Novoa, et al.. (2002). In vivo bradykinin B2 receptor activation reduces renal fibrosis. Journal of Clinical Investigation. 110(3). 371–379. 10 indexed citations

15.

Ortíz, M. Clara, Lourdes A. Fortepiani, Carlos Martı́nez-Salgado, et al.. (2001). Efectos renales de la inhibición crónica de la síntesis de óxido nítrico en ratas cirróticas con ascitis. Nefrología. 21(6). 556–564. 1 indexed citations

16.

Rodriquez, A. A., et al.. (1999). Renal Function in Compensated Hepatic Cirrhosis: Effects of an Amino Acid Infusion and Relationship with Nitric Acid. Digestive Diseases. 17(4). 235–240. 6 indexed citations

17.

Peiró, Concepción, Ana M. Rodrı́guez-López, Javier Angulo, et al.. (1998). Endogenous Angiotensin II and Cell Hypertrophy in Vascular Smooth Muscle Cultures from Hypertensive Ren-2 Transgenic Rats. Cellular Physiology and Biochemistry. 8(1-2). 106–116. 6 indexed citations

18.

Rodrı́guez-López, Ana M., et al.. (1998). Glomerular cell proliferation and apoptosis in uninephrectomized spontaneously hypertensive rats. Kidney International. 54. S36–S40. 40 indexed citations

19.

Gómez‐Garré, Dulcenombre, Antonio López‐Farré, N. Eleno, & José M. López‐Novoa. (1996). Comparative Effects of Dopexamine and Dopamine on Glycerol-Induced Acute Renal Failure in Rats. Renal Failure. 18(1). 59–68. 8 indexed citations

20.

Pintado, José, et al.. (1990). Renal catabolism of human glucagon-like peptides 1 and 2. Canadian Journal of Physiology and Pharmacology. 68(12). 1568–1573. 63 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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