Aiko Sawa

427 citations
12 papers · 373 indexed · h-index 8
Topics
Phosphodiesterase function and regulation (7 papers)Cholinesterase and Neurodegenerative Diseases (4 papers)Neuropeptides and Animal Physiology (4 papers)
Partner nations
JapanUnited States

In The Last Decade

Aiko Sawa

12 papers receiving 349 citations

Peers

Aiko Sawa
Comparison fields: 5 of 61
  • Molecular Biology 207
  • Cellular and Molecular Neuroscience 170
  • Pharmacology 121
  • Clinical Psychology 63
  • Cognitive Neuroscience 53
Replace Taiichiro Imanishi with:
Taiichiro Imanishi Japan
Ros R. Brett United Kingdom
E. S. Gershon United States
J.Brent Bedingfield United States
Ryan Ting‐A‐Kee Canada
Claire Allison United Kingdom
Bartley G. Hoebel United States
Carine Dias Canada
Valérie Fauchey France
Alberto Casti Italy
Aiko Sawa relative to Taiichiro Imanishi Japan Taiichiro Imanishi's profile →
Citations per field
00.5×1.6×
Taiichiro Imanishi · 1×
Citations per year

Countries citing papers authored by Aiko Sawa

Since Specialization
Citations

This map shows the geographic impact of Aiko Sawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aiko Sawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aiko Sawa more than expected).

Fields of papers citing papers by Aiko Sawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aiko Sawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aiko Sawa. The network helps show where Aiko Sawa may publish in the future.

Co-authorship network of co-authors of Aiko Sawa

This figure shows the co-authorship network connecting the top 25 collaborators of Aiko Sawa. A scholar is included among the top collaborators of Aiko Sawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aiko Sawa. Aiko Sawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
#WorkIndexed citations
1 7
2 16
3 22
4 24
5 115
6 1
7 7
8 5
9 144
10 18
11 3
12 11

About Aiko Sawa

Aiko Sawa is a scholar working on Cellular and Molecular Neuroscience, Pharmacology and Physiology, having authored 12 papers that have together received 373 indexed citations. Recurring topics across this work include Phosphodiesterase function and regulation (7 papers), Cholinesterase and Neurodegenerative Diseases (4 papers) and Neuropeptides and Animal Physiology (4 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (170 citations), Pharmacology (121 citations) and Behavioral Neuroscience (20 citations). Aiko Sawa has collaborated with scholars based in Japan and United States. Frequent co-authors include Yasuyuki Ichimaru, Takashi Egawa, Taiichiro Imanishi, Nobuyuki Yamashita, Shinsuke Kato, Showa Ueki, Tsuneyuki Yamamoto, Akiko Hayashi, Miki Yamauchi and William B. Stavinoha. Their work appears in journals such as European Journal of Pharmacology, Developmental Brain Research and Brain Research Bulletin.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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