Adrienne Samani

526 total citations
8 papers, 365 citations indexed

About

Adrienne Samani is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Adrienne Samani has authored 8 papers receiving a total of 365 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Cell Biology and 2 papers in Genetics. Recurrent topics in Adrienne Samani's work include Muscle Physiology and Disorders (6 papers), Genetics and Neurodevelopmental Disorders (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Adrienne Samani is often cited by papers focused on Muscle Physiology and Disorders (6 papers), Genetics and Neurodevelopmental Disorders (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Adrienne Samani collaborates with scholars based in United States and United Kingdom. Adrienne Samani's co-authors include Jason D. Doles, Dong Seong Cho, Runqing Huang, Eduardo N. Chini, Guilherme C. de Oliveira, João F. Passos, Claudia C.S. Chini, Kyaw Zaw Hein, James Boslett and Gina M. Warner and has published in prestigious journals such as SHILAP Revista de lepidopterología, Cell Metabolism and The FASEB Journal.

In The Last Decade

Adrienne Samani

8 papers receiving 358 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Adrienne Samani United States	6	135	128	111	94	68	8	365
Thais Peclat United States	11	61 0.5×	129 1.0×	88 0.8×	63 0.7×	52 0.8×	21	362
Alex Parker Canada	5	165 1.2×	58 0.5×	190 1.7×	98 1.0×	22 0.3×	6	404
John Wizeman United States	7	134 1.0×	33 0.3×	118 1.1×	58 0.6×	15 0.2×	8	319
Xinghua Hou China	8	275 2.0×	37 0.3×	256 2.3×	91 1.0×	142 2.1×	10	515
Ryan A. Doan United States	4	185 1.4×	64 0.5×	95 0.9×	72 0.8×	114 1.7×	5	441
Dara Watkins United States	7	172 1.3×	19 0.1×	20 0.2×	40 0.4×	37 0.5×	8	287
Keyi Geng China	11	173 1.3×	11 0.1×	73 0.7×	50 0.5×	20 0.3×	14	328
Ofir Zmira Israel	6	138 1.0×	30 0.2×	14 0.1×	72 0.8×	17 0.3×	7	369
Alisia Carnemolla United Kingdom	9	268 2.0×	8 0.1×	20 0.2×	58 0.6×	42 0.6×	9	384
Changyong Hu Singapore	10	151 1.1×	15 0.1×	12 0.1×	112 1.2×	51 0.8×	17	360

Countries citing papers authored by Adrienne Samani

Since Specialization

Citations

This map shows the geographic impact of Adrienne Samani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adrienne Samani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adrienne Samani more than expected).

Fields of papers citing papers by Adrienne Samani

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Adrienne Samani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adrienne Samani. The network helps show where Adrienne Samani may publish in the future.

Co-authorship network of co-authors of Adrienne Samani

This figure shows the co-authorship network connecting the top 25 collaborators of Adrienne Samani. A scholar is included among the top collaborators of Adrienne Samani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Adrienne Samani. Adrienne Samani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Samani, Adrienne, et al.. (2023). DOCK3 regulates normal skeletal muscle regeneration and glucose metabolism. The FASEB Journal. 37(10). e23198–e23198. 3 indexed citations

2.

Samani, Adrienne, Michael A. Lopez, Michael J. Conklin, et al.. (2022). miR-486 is essential for muscle function and suppresses a dystrophic transcriptome. Life Science Alliance. 5(9). e202101215–e202101215. 17 indexed citations

3.

Samani, Adrienne, Michael A. Lopez, Camille L. Birch, et al.. (2022). DOCKopathies: A systematic review of the clinical pathologies associated with human DOCK pathogenic variants. Human Mutation. 43(9). 1149–1161. 7 indexed citations

4.

Samani, Adrienne, Gérald Coulis, S. Armando Villalta, et al.. (2022). Next-Generation SINE Compound KPT−8602 Ameliorates Dystrophic Pathology in Zebrafish and Mouse Models of DMD. Biomedicines. 10(10). 2400–2400. 4 indexed citations

5.

Wang, Yimin, Adrienne Samani, Michael A. Lopez, et al.. (2020). DOCK3 is a dosage-sensitive regulator of skeletal muscle and Duchenne muscular dystrophy-associated pathologies. Human Molecular Genetics. 29(17). 2855–2871. 9 indexed citations

6.

Hogan, Kelly A., et al.. (2020). The wasting‐associated metabolite succinate disrupts myogenesis and impairs skeletal muscle regeneration. SHILAP Revista de lepidopterología. 3(2). 56–69. 14 indexed citations

7.

Tarragó, Mariana G., Claudia C.S. Chini, Karina S. Kanamori, et al.. (2018). A Potent and Specific CD38 Inhibitor Ameliorates Age-Related Metabolic Dysfunction by Reversing Tissue NAD+ Decline. Cell Metabolism. 27(5). 1081–1095.e10. 271 indexed citations

8.

Hogan, Kelly A., et al.. (2017). Tumor-derived cytokines impair myogenesis and alter the skeletal muscle immune microenvironment. Cytokine. 107. 9–17. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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