Abir A. Rahman
- Molecular Biology
- Neurology top 10%
- Physiology
- Cellular and Molecular Neuroscience
- Developmental Neuroscience top 10%
- Co-authors
- Emmanuel PinteauxGregory BixNarayanappa AmrutaBrad E. MorrisonN. Joseph EspatW. Scott HeltonA. A. KurlandM. Andreína Pacheco
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (4 papers)Acute Ischemic Stroke Management (3 papers)Nuclear Receptors and Signaling (3 papers)
- Journals
- StrokeScientific ReportsNeuroscience
- Partner nations
- United StatesUnited KingdomTürkiye
In The Last Decade
Abir A. Rahman
13 papers receiving 399 citations
Peers
Comparison fields: 5 of 76
- Molecular Biology 157
- Neurology 106
- Physiology 72
- Cellular and Molecular Neuroscience 64
- Developmental Neuroscience 60
Countries citing papers authored by Abir A. Rahman
This map shows the geographic impact of Abir A. Rahman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abir A. Rahman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abir A. Rahman more than expected).
Fields of papers citing papers by Abir A. Rahman
This network shows the impact of papers produced by Abir A. Rahman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abir A. Rahman. The network helps show where Abir A. Rahman may publish in the future.
Co-authorship network of co-authors of Abir A. Rahman
This figure shows the co-authorship network connecting the top 25 collaborators of Abir A. Rahman. A scholar is included among the top collaborators of Abir A. Rahman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abir A. Rahman. Abir A. Rahman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 1 | |
| 3 | 27 | |
| 4 | 14 | |
| 5 | 108 | |
| 6 | 40 | |
| 7 | 35 | |
| 8 | 3 | |
| 9 | Nuclear uptake of an amino-terminal fragment of apolipoprotein E4 promotes cell death and localizes within microglia of the Alzheimer's disease brain. | 18 |
| 10 | 28 | |
| 11 | 47 | |
| 12 | 60 | |
| 13 | 17 |
About Abir A. Rahman
Abir A. Rahman is a scholar working on Developmental Neuroscience, Neurology and Cellular and Molecular Neuroscience, having authored 13 papers that have together received 405 indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Acute Ischemic Stroke Management (3 papers) and Nuclear Receptors and Signaling (3 papers). The work is most often cited by research in Developmental Neuroscience (60 citations), Neurology (106 citations) and Cellular and Molecular Neuroscience (64 citations). Abir A. Rahman has collaborated with scholars based in United States, United Kingdom and Türkiye. Frequent co-authors include Emmanuel Pinteaux, Gregory Bix, Narayanappa Amruta, Brad E. Morrison, N. Joseph Espat, W. Scott Helton, A. A. Kurland, M. Andreína Pacheco, William E. Collins and Ananías A. Escalante. Their work appears in journals such as Stroke, Scientific Reports and Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.