Abigail Buchwalter
- Aging top 10%
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- Nuclear Structure and Function 9
- RNA Research and Splicing 5
- Genomics and Chromatin Dynamics 4
- DNA Repair Mechanisms 3
- CRISPR and Genetic Engineering 2
- Mitochondrial Function and Pathology 2
-
- Cellular transport and secretion 3
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- Force Microscopy Techniques and Applications 1
- Co-authors
- Martin W. HetzerYun Xiang LiangJames E. HuettnerKevin M. KaltenbronnCraig A. DoupnikKendall BlumerRyan M. DrenanMaurine E. Linder
- Cited by
- AgingMolecular BiologyCell Biology
- Journals
- Molecular Biology of the Cell (2 papers)Nature Cell Biology (1 paper)The Journal of Cell Biology (1 paper)
- Partner nations
- United StatesBelgiumGermany
In The Last Decade
Abigail Buchwalter
13 papers receiving 532 citations
Peers
Comparison fields: 5 of 58
- Aging 22
- Molecular Biology 463
- Cell Biology 93
- Cellular and Molecular Neuroscience 45
- Physiology 43
Countries citing papers authored by Abigail Buchwalter
This map shows the geographic impact of Abigail Buchwalter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abigail Buchwalter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abigail Buchwalter more than expected).
Fields of papers citing papers by Abigail Buchwalter
This network shows the impact of papers produced by Abigail Buchwalter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abigail Buchwalter. The network helps show where Abigail Buchwalter may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Abigail Buchwalter, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2026 | 0 | |
| 2 | 2025 | 1 | |
| 3 | 2025 | 2 | |
| 4 | 2024 | 0 | |
| 5 | 2023 | 8 | |
| 6 | 2023 | 12 | |
| 7 | 2023 | 1 | |
| 8 | 2023 | 5 | |
| 9 | 2020 | 6 | |
| 10 | 2019 | 32 | |
| 11 | 2018 | 136 | |
| 12 | 2017 | 184 | |
| 13 | 2015 | 33 | |
| 14 | 2014 | 54 | |
| 15 | 2006 | 63 |
About Abigail Buchwalter
Abigail Buchwalter is a scholar working on Cell Biology, Molecular Biology and Cellular and Molecular Neuroscience, having authored 15 papers that have together received 537 indexed citations. Recurring topics across this work include Nuclear Structure and Function (9 papers), RNA Research and Splicing (5 papers), Genomics and Chromatin Dynamics (4 papers), DNA Repair Mechanisms (3 papers), Cellular transport and secretion (3 papers), CRISPR and Genetic Engineering (2 papers), Mitochondrial Function and Pathology (2 papers) and Force Microscopy Techniques and Applications (1 paper). The work is most often cited by research in Aging (22 citations), Molecular Biology (463 citations) and Cell Biology (93 citations). Abigail Buchwalter has collaborated with scholars based in United States, Belgium and Germany. Frequent co-authors include Martin W. Hetzer, Yun Xiang Liang, James E. Huettner, Kevin M. Kaltenbronn, Craig A. Doupnik, Kendall Blumer, Ryan M. Drenan, Maurine E. Linder, Muralidharan Jayaraman and Roberta Schulte. Their work appears in journals such as Molecular Biology of the Cell, Nature Cell Biology, The Journal of Cell Biology, Current Opinion in Cell Biology and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.