A E Koch

7.7k total citations · 2 hit papers
47 papers, 6.5k citations indexed

About

A E Koch is a scholar working on Rheumatology, Immunology and Allergy and Oncology. According to data from OpenAlex, A E Koch has authored 47 papers receiving a total of 6.5k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Rheumatology, 22 papers in Immunology and Allergy and 12 papers in Oncology. Recurrent topics in A E Koch's work include Cell Adhesion Molecules Research (22 papers), Rheumatoid Arthritis Research and Therapies (10 papers) and Systemic Lupus Erythematosus Research (9 papers). A E Koch is often cited by papers focused on Cell Adhesion Molecules Research (22 papers), Rheumatoid Arthritis Research and Therapies (10 papers) and Systemic Lupus Erythematosus Research (9 papers). A E Koch collaborates with scholars based in United States, Philippines and Hungary. A E Koch's co-authors include Richard M. Pope, G. Kenneth Haines, Lisa A. Harlow, Steven L. Kunkel, Robert M. Strieter, James B. Rottman, G. Kenneth Haines, Patricia N. Myers, Marcel Loetscher and Nasim Kassam and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

A E Koch

47 papers receiving 6.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The chemokine receptors CXCR3 and CCR5 mark subsets of T cells associated with certain inflammatory reactions.

1998 1.2k citations Suofu Qin, James B. Rottman et al. Journal of Clinical Investigation profile →
Enhanced production of monocyte chemoattractant protein-1 in rheumatoid arthritis.

1992 543 citations A E Koch, Steven L. Kunkel et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
A E Koch United States	33	2.7k	2.4k	1.7k	1.5k	1.5k	47	6.5k
G. Kenneth Haines United States	43	2.3k 0.9×	2.2k 0.9×	1.6k 1.0×	2.1k 1.3×	1.1k 0.7×	92	6.6k
Lisa A. Harlow United States	25	1.9k 0.7×	1.7k 0.7×	1.0k 0.6×	1.8k 1.1×	1.1k 0.8×	35	5.3k
Edward P. Amento United States	43	1.8k 0.7×	1.2k 0.5×	1.2k 0.7×	2.3k 1.5×	664 0.5×	78	8.2k
Ewa Paleolog United Kingdom	46	1.5k 0.6×	1.2k 0.5×	1.6k 1.0×	2.3k 1.5×	583 0.4×	123	6.6k
Itsuo Iwamoto Japan	51	4.4k 1.7×	1.1k 0.4×	1.1k 0.7×	1.1k 0.7×	997 0.7×	167	7.7k
Margaret L. Hibbs Australia	49	4.3k 1.6×	1.3k 0.6×	583 0.3×	2.8k 1.8×	1.6k 1.1×	128	8.2k
Kazuhiko Takehara Japan	69	3.8k 1.4×	1.4k 0.6×	2.9k 1.7×	4.3k 2.8×	1.2k 0.8×	375	15.9k
Andrew Filer United Kingdom	49	2.5k 0.9×	1.1k 0.4×	2.7k 1.6×	1.9k 1.3×	399 0.3×	176	7.2k
Michael V. Volin United States	39	1.6k 0.6×	1.4k 0.6×	1.2k 0.7×	1.7k 1.1×	497 0.3×	69	4.9k
David Chantry United States	35	3.0k 1.1×	1.8k 0.8×	870 0.5×	1.1k 0.7×	636 0.4×	74	5.5k

Countries citing papers authored by A E Koch

Since Specialization

Citations

This map shows the geographic impact of A E Koch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A E Koch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A E Koch more than expected).

Fields of papers citing papers by A E Koch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A E Koch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A E Koch. The network helps show where A E Koch may publish in the future.

Co-authorship network of co-authors of A E Koch

This figure shows the co-authorship network connecting the top 25 collaborators of A E Koch. A scholar is included among the top collaborators of A E Koch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A E Koch. A E Koch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Duvvuri, Bhargavi, Ernst R. Dow, A E Koch, et al.. (2024). Neutrophil Activation Markers and Rheumatoid Arthritis Treatment Response to the JAK1/2 Inhibitor Baricitinib. Arthritis & Rheumatology. 77(4). 395–404. 4 indexed citations

Prossin, Alan R., A E Koch, Phillip L. Campbell, et al.. (2016). Experimental sadness induces relevant interactions between central endogenous opioid activation and plasma IL-18 concentrations in depressed volunteers. Molecular Psychiatry. 21(2). 151–151. 7 indexed citations

Prossin, Alan R., et al.. (2015). Acute experimental changes in mood state regulate immune function in relation to central opioid neurotransmission: a model of human CNS-peripheral inflammatory interaction. Molecular Psychiatry. 21(2). 243–251. 33 indexed citations

Genovese, M. C., Joel M. Kremer, Omid Zamani, et al.. (2015). OP0029 Baricitinib, An Oral Janus Kinase (JAK)1/JAK2 Inhibitor, in Patients with Active Rheumatoid Arthritis (RA) and an Inadequate Response to TNF Inhibitors: Results of the Phase 3 RA-Beacon Study. Annals of the Rheumatic Diseases. 74. 75–76. 20 indexed citations

Marotte, Hubert, Jeffrey H. Ruth, Phillip L. Campbell, A E Koch, & Salahuddin Ahmed. (2009). Green tea extract inhibits chemokine production, but up-regulates chemokine receptor expression, in rheumatoid arthritis synovial fibroblasts and rat adjuvant-induced arthritis. Lara D. Veeken. 49(3). 467–479. 50 indexed citations

Koch, A E. (2003). Angiogenesis as a target in rheumatoid arthritis. Annals of the Rheumatic Diseases. 62. ii60–ii67. 173 indexed citations

Koch, A E, Michael V. Volin, James M. Woods, et al.. (2001). Regulation of angiogenesis by the C-X-C chemokines interleukin-8 and epithelial neutrophil activating peptide 78 in the rheumatoid joint. Arthritis & Rheumatism. 44(1). 31–40. 121 indexed citations

Perlman, Harris, Lisa J. Pagliari, Hongtao Liu, et al.. (2001). Rheumatoid arthritis synovial macrophages express the Fas-associated death domain-like interleukin-1?-converting enzyme-inhibitory protein and are refractory to Fas-mediated apoptosis. Arthritis & Rheumatism. 44(1). 21–30. 85 indexed citations

Koch, A E. (2000). The role of angiogenesis in rheumatoid arthritis: recent developments. Annals of the Rheumatic Diseases. 59. i65–i71. 93 indexed citations

10.

Volin, Michael V., Zoltán Szekanecz, Margaret M. Halloran, et al.. (1999). PECAM-1 and Leukosialin (CD43) Expression Correlate with Heightened Inflammation in Rat Adjuvant-Induced Arthritis. Experimental and Molecular Pathology. 66(3). 211–219. 11 indexed citations

11.

Szekanecz, Zoltán, A E Koch, S L Kunkel, & Robert M. Strieter. (1998). Cytokines in Rheumatoid Arthritis. Drugs & Aging. 12(5). 377–390. 57 indexed citations

12.

Qin, Suofu, James B. Rottman, Patricia N. Myers, et al.. (1998). The chemokine receptors CXCR3 and CCR5 mark subsets of T cells associated with certain inflammatory reactions.. Journal of Clinical Investigation. 101(4). 746–754. 1178 indexed citations breakdown →

13.

Szekanecz, Zoltán, G. Kenneth Haines, Lisa A. Harlow, et al.. (1995). Increased Synovial Expression of Transforming Growth Factor (TGF)-β Receptor Endoglin and TGF-β1 in Rheumatoid Arthritis: Possible Interactions in the Pathogenesis of the Disease. Clinical Immunology and Immunopathology. 76(2). 187–194. 64 indexed citations

14.

Koch, A E, Steven L. Kunkel, Lisa A. Harlow, et al.. (1994). Macrophage inflammatory protein-1 alpha. A novel chemotactic cytokine for macrophages in rheumatoid arthritis.. Journal of Clinical Investigation. 93(3). 921–928. 261 indexed citations

15.

Leibovich, S. Joseph, et al.. (1994). Production of angiogenic activity by human monocytes requires an L-arginine/nitric oxide-synthase-dependent effector mechanism.. Proceedings of the National Academy of Sciences. 91(10). 4190–4194. 179 indexed citations

16.

Koch, A E, B J Nickoloff, Jan Holgersson, et al.. (1994). 4A11, a monoclonal antibody recognizing a novel antigen expressed on aberrant vascular endothelium. Upregulation in an in vivo model of contact dermatitis.. PubMed. 144(2). 244–59. 25 indexed citations

17.

Koch, A E, Steven L. Kunkel, W H Pearce, et al.. (1993). Enhanced production of the chemotactic cytokines interleukin-8 and monocyte chemoattractant protein-1 in human abdominal aortic aneurysms.. PubMed Central. 142(5). 1423–31. 216 indexed citations

18.

Koch, A E, et al.. (1993). Soluble E-Selectin in Arthritis. Clinical Immunology and Immunopathology. 69(1). 29–35. 50 indexed citations

19.

Johnson, B. A., G. Kenneth Haines, Lisa A. Harlow, & A E Koch. (1993). Adhesion molecule expression in human synovial tissue. Arthritis & Rheumatism. 36(2). 137–146. 180 indexed citations

20.

Koch, A E, Steven L. Kunkel, Lisa A. Harlow, et al.. (1992). Enhanced production of monocyte chemoattractant protein-1 in rheumatoid arthritis.. Journal of Clinical Investigation. 90(3). 772–779. 543 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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