James M. Woods

3.6k total citations
44 papers, 2.9k citations indexed

About

James M. Woods is a scholar working on Oncology, Immunology and Rheumatology. According to data from OpenAlex, James M. Woods has authored 44 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Oncology, 16 papers in Immunology and 11 papers in Rheumatology. Recurrent topics in James M. Woods's work include Chemokine receptors and signaling (11 papers), Cell Adhesion Molecules Research (9 papers) and Rheumatoid Arthritis Research and Therapies (7 papers). James M. Woods is often cited by papers focused on Chemokine receptors and signaling (11 papers), Cell Adhesion Molecules Research (9 papers) and Rheumatoid Arthritis Research and Therapies (7 papers). James M. Woods collaborates with scholars based in United States, United Kingdom and Japan. James M. Woods's co-authors include Alisa E. Koch, Michael V. Volin, G. Kenneth Haines, Walter C. Prozialeck, Joshua Edwards, M. Asif Amin, Christy C. Park, Lisa A. Harlow, Jeffrey H. Ruth and Jacques Morel and has published in prestigious journals such as Journal of Biological Chemistry, Nature Immunology and The Journal of Immunology.

In The Last Decade

James M. Woods

42 papers receiving 2.8k citations

Peers

James M. Woods

IMMUN

ONCOL

RHEUM

HTM

Keigo Nishida Japan

Françis Frankenne Belgium

Satoru Yamasaki Japan

Youichiro Wada Japan

Patricia Ruíz Germany

Shuxia Wang China

Diana Patricia Portales‐Pérez Mexico

Christopher D. Kontos United States

Paula H. Stern United States

Christer T. Jansén Finland

Keigo Nishida Japan

James M. Woods

1.4k ×1.3

IMMUN

824 ×0.9

ONCOL

2.0k ×2.7

193 ×0.3

RHEUM

498 ×1.4

HTM

Citations per year, relative to James M. Woods James M. Woods (= 1×) peers Keigo Nishida

Countries citing papers authored by James M. Woods

Since Specialization

Citations

This map shows the geographic impact of James M. Woods's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James M. Woods with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James M. Woods more than expected).

Fields of papers citing papers by James M. Woods

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James M. Woods. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James M. Woods. The network helps show where James M. Woods may publish in the future.

Co-authorship network of co-authors of James M. Woods

This figure shows the co-authorship network connecting the top 25 collaborators of James M. Woods. A scholar is included among the top collaborators of James M. Woods based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James M. Woods. James M. Woods is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ilyashenko, Gennadiy, et al.. (2013). Enantioselective Synthesis and Application to the Allylic Imidate Rearrangement of Amine‐Coordinated Palladacycle Catalysts of Cobalt Sandwich Complexes. Chemistry - A European Journal. 19(52). 17951–17962. 15 indexed citations

Volin, Michael V., et al.. (2010). Fractalkine-Induced Endothelial Cell Migration Requires MAP Kinase Signaling. Pathobiology. 77(1). 7–16. 29 indexed citations

Woods, James M., et al.. (2008). Gene Therapy for Rheumatoid Arthritis: Recent Advances. Current Gene Therapy. 8(1). 24–41. 14 indexed citations

Prozialeck, Walter C., Joshua Edwards, Daniel W. Nebert, et al.. (2007). The Vascular System as a Target of Metal Toxicity. Toxicological Sciences. 102(2). 207–218. 251 indexed citations

Volin, Michael V., Shiva Shahrara, G. Kenneth Haines, James M. Woods, & Alisa E. Koch. (2007). Expression of mucin 3 and mucin 5AC in arthritic synovial tissue. Arthritis & Rheumatism. 58(1). 46–52. 13 indexed citations

Haas, Christian S., M. Asif Amin, Jeffrey H. Ruth, et al.. (2007). In vivo inhibition of angiogenesis by interleukin‐13 gene therapy in a rat model of rheumatoid arthritis. Arthritis & Rheumatism. 56(8). 2535–2548. 47 indexed citations

Haas, Christian S., M. Asif Amin, Jeffrey H. Ruth, et al.. (2006). Inhibition of angiogenesis by interleukin‐4 gene therapy in rat adjuvant‐induced arthritis. Arthritis & Rheumatism. 54(8). 2402–2414. 47 indexed citations

Prozialeck, Walter C., Joshua Edwards, & James M. Woods. (2006). The vascular endothelium as a target of cadmium toxicity. Life Sciences. 79(16). 1493–1506. 201 indexed citations

Shahrara, Shiva, M. Asif Amin, James M. Woods, G. Kenneth Haines, & Alisa E. Koch. (2003). Chemokine receptor expression and in vivo signaling pathways in the joints of rats with adjuvant‐induced arthritis. Arthritis & Rheumatism. 48(12). 3568–3583. 61 indexed citations

10.

Hu, Xiaoyu, Carmen Herrero, Taras T. Antoniv, et al.. (2002). Sensitization of IFN-γ Jak-STAT signaling during macrophage activation. Nature Immunology. 3(9). 859–866. 199 indexed citations

11.

Perlman, Harris, Hongtao Liu, Constantinos Georganas, et al.. (2002). Modifications in adenoviral coat fiber proteins and transcriptional regulatory sequences enhance transgene expression.. PubMed. 29(8). 1593–600. 10 indexed citations

12.

Woods, James M., Kenneth J. Katschke, Michael V. Volin, et al.. (2001). IL-4 Adenoviral Gene Therapy Reduces Inflammation, Proinflammatory Cytokines, Vascularization, and Bony Destruction in Rat Adjuvant-Induced Arthritis. The Journal of Immunology. 166(2). 1214–1222. 122 indexed citations

13.

Koch, A E, Michael V. Volin, James M. Woods, et al.. (2001). Regulation of angiogenesis by the C-X-C chemokines interleukin-8 and epithelial neutrophil activating peptide 78 in the rheumatoid joint. Arthritis & Rheumatism. 44(1). 31–40. 121 indexed citations

14.

Halloran, Margaret M., William W. Carley, Peter J. Polverini, et al.. (2000). Ley/H: An Endothelial-Selective, Cytokine-Inducible, Angiogenic Mediator. The Journal of Immunology. 164(9). 4868–4877. 56 indexed citations

15.

Woods, James M., Kenneth J. Katschke, Michihide Tokuhira, et al.. (2000). Reduction of Inflammatory Cytokines and Prostaglandin E2 by IL-13 Gene Therapy in Rheumatoid Arthritis Synovium. The Journal of Immunology. 165(5). 2755–2763. 42 indexed citations

16.

Szekanecz, Zoltán, Margaret M. Halloran, Michael V. Volin, et al.. (2000). Temporal expression of inflammatory cytokines and chemokines in rat adjuvant‐induced arthritis. Arthritis & Rheumatism. 43(6). 1266–1277. 149 indexed citations

17.

Halloran, Margaret M., James M. Woods, Robert M. Strieter, et al.. (1999). The Role of an Epithelial Neutrophil-Activating Peptide-78-Like Protein in Rat Adjuvant-Induced Arthritis. The Journal of Immunology. 162(12). 7492–7500. 73 indexed citations

18.

Volin, Michael V., Manisha R. Shah, Michihide Tokuhira, et al.. (1998). RANTES Expression and Contribution to Monocyte Chemotaxis in Arthritis. Clinical Immunology and Immunopathology. 89(1). 44–53. 105 indexed citations

19.

Woods, James M. & Mary J. Druse. (1996). Effects of Chronic Ethanol Consumption and Aging on Dopamine, Serotonin, and Metabolites. Journal of Neurochemistry. 66(5). 2168–2178. 29 indexed citations

20.

Woods, James M., et al.. (1993). Effects of Chronic Ethanol Consumption on G Proteins in Brain Areas Associated with the Nigrostriatal and Mesolimbic Dopamine Systems. Alcoholism Clinical and Experimental Research. 17(6). 1247–1253. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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