HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors.
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doi.org/w41627931 →Countries where authors are citing HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors.
This map shows the geographic impact of HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors.. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors. with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors. more than expected).
Fields of papers citing HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors.
This network shows the impact of HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors.. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the HIF-1-dependent regulation of hypoxic induction of the cell death factors BNIP3 and NIX in human tumors..
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/w41627931.