Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS
Abstract
loading...
About
In The Last Decade
doi.org/10.1038/nn.2660 →Countries where authors are citing Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS
This map shows the geographic impact of Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS more than expected).
Fields of papers citing Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS
This network shows the impact of Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/nn.2660.