Yukio Akaneya
- Cellular and Molecular Neuroscience top 2%
- Molecular Biology
- Developmental Neuroscience top 2%
- Neurology top 10%
- Cognitive Neuroscience top 10%
- Co-authors
- Tadaharu TsumotoHiroshi HatanakaBin JiangMitsuo TakahashiH. HatanakaYoshio HataYasushi EnokidoEdward B. Ziff
- Topics
- Neuroscience and Neuropharmacology Research (14 papers)Nerve injury and regeneration (9 papers)Neurogenesis and neuroplasticity mechanisms (8 papers)
- Journals
- Journal of NeuroscienceSHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- JapanUnited StatesRussia
In The Last Decade
Yukio Akaneya
25 papers receiving 981 citations
Peers
Comparison fields: 5 of 86
- Cellular and Molecular Neuroscience 678
- Molecular Biology 364
- Developmental Neuroscience 307
- Neurology 132
- Cognitive Neuroscience 117
Countries citing papers authored by Yukio Akaneya
This map shows the geographic impact of Yukio Akaneya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yukio Akaneya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yukio Akaneya more than expected).
Fields of papers citing papers by Yukio Akaneya
This network shows the impact of papers produced by Yukio Akaneya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yukio Akaneya. The network helps show where Yukio Akaneya may publish in the future.
Co-authorship network of co-authors of Yukio Akaneya
This figure shows the co-authorship network connecting the top 25 collaborators of Yukio Akaneya. A scholar is included among the top collaborators of Yukio Akaneya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yukio Akaneya. Yukio Akaneya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 63 | |
| 3 | 12 | |
| 4 | 9 | |
| 5 | 41 | |
| 6 | 93 | |
| 7 | 13 | |
| 8 | 39 | |
| 9 | 43 | |
| 10 | 40 | |
| 11 | 38 | |
| 12 | 4 | |
| 13 | 13 | |
| 14 | 48 | |
| 15 | 100 | |
| 16 | 6 | |
| 17 | 4 | |
| 18 | 8 | |
| 19 | 34 | |
| 20 | 51 |
About Yukio Akaneya
Yukio Akaneya is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Cognitive Neuroscience, having authored 25 papers that have together received 1.0k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (14 papers), Nerve injury and regeneration (9 papers) and Neurogenesis and neuroplasticity mechanisms (8 papers). The work is most often cited by research in Developmental Neuroscience (307 citations), Cellular and Molecular Neuroscience (678 citations) and Neurology (132 citations). Yukio Akaneya has collaborated with scholars based in Japan, United States and Russia. Frequent co-authors include Tadaharu Tsumoto, Hiroshi Hatanaka, Bin Jiang, Mitsuo Takahashi, H. Hatanaka, Yoshio Hata, Yasushi Enokido, Edward B. Ziff, Akihiko Kitamura and Kazuhiro Sohya. Their work appears in journals such as Journal of Neuroscience, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.