Yuichi Hattori

520 total citations
9 papers, 455 citations indexed

About

Yuichi Hattori is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Yuichi Hattori has authored 9 papers receiving a total of 455 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Cellular and Molecular Neuroscience and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Yuichi Hattori's work include Ion channel regulation and function (5 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Neuroscience and Neuropharmacology Research (4 papers). Yuichi Hattori is often cited by papers focused on Ion channel regulation and function (5 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Neuroscience and Neuropharmacology Research (4 papers). Yuichi Hattori collaborates with scholars based in Japan. Yuichi Hattori's co-authors include Morio Kanno, Morio Kanno, Ichiro Sakuma, Akira Kitabatake, Satoshi Gando, Naoyuki Matsuda, Toshiteru Ishitani, Junko Kimura, Atsushi Tamada and Mitsuhiro Fukao and has published in prestigious journals such as The Journal of Physiology, British Journal of Pharmacology and Biochemical Pharmacology.

In The Last Decade

Yuichi Hattori

9 papers receiving 445 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yuichi Hattori Japan 8 235 214 186 69 65 9 455
Morio Kanno Japan 9 213 0.9× 228 1.1× 185 1.0× 57 0.8× 64 1.0× 13 457
Sylvie Delaflotte France 14 164 0.7× 316 1.5× 188 1.0× 39 0.6× 49 0.8× 29 549
G. J. Dusting Australia 12 180 0.8× 282 1.3× 135 0.7× 51 0.7× 107 1.6× 18 540
J. A. Bellan United States 12 129 0.5× 231 1.1× 125 0.7× 32 0.5× 53 0.8× 22 449
Ingrid Heinroth‐Hoffmann Germany 15 297 1.3× 128 0.6× 286 1.5× 30 0.4× 44 0.7× 29 541
Klaus-Dieter Schultz Germany 5 145 0.6× 254 1.2× 193 1.0× 45 0.7× 37 0.6× 6 465
Kuniaki Iwasawa Japan 17 262 1.1× 154 0.7× 360 1.9× 39 0.6× 62 1.0× 37 696
Kazuhiro Hisaki Japan 10 233 1.0× 338 1.6× 123 0.7× 40 0.6× 59 0.9× 19 418
Marie-Jeanne Mathy Netherlands 15 303 1.3× 174 0.8× 290 1.6× 134 1.9× 57 0.9× 31 644
Belinda A. Ahlers United States 14 326 1.4× 167 0.8× 390 2.1× 34 0.5× 52 0.8× 19 623

Countries citing papers authored by Yuichi Hattori

Since Specialization
Citations

This map shows the geographic impact of Yuichi Hattori's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuichi Hattori with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuichi Hattori more than expected).

Fields of papers citing papers by Yuichi Hattori

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuichi Hattori. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuichi Hattori. The network helps show where Yuichi Hattori may publish in the future.

Co-authorship network of co-authors of Yuichi Hattori

This figure shows the co-authorship network connecting the top 25 collaborators of Yuichi Hattori. A scholar is included among the top collaborators of Yuichi Hattori based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuichi Hattori. Yuichi Hattori is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Hattori, Yuichi, Naoyuki Matsuda, Junko Kimura, et al.. (2000). Diminished function and expression of the cardiac Na+‐Ca2+ exchanger in diabetic rats: implication in Ca2+ overload. The Journal of Physiology. 527(1). 85–94. 112 indexed citations
2.
Matsuda, Naoyuki, et al.. (1999). Diabetes-induced down-regulation of β1-adrenoceptor mRNA expression in rat heart. Biochemical Pharmacology. 58(5). 881–885. 44 indexed citations
4.
Hattori, Yuichi, et al.. (1997). Alterations in endothelium‐dependent hyperpolarization and relaxation in mesenteric arteries from streptozotocin‐induced diabetic rats. British Journal of Pharmacology. 121(7). 1383–1391. 140 indexed citations
5.
Fukao, Mitsuhiro, et al.. (1995). Thapsigargin‐ and cyclopiazonic acid‐induced endothelium‐dependent hyperpolarization in rat mesenteric artery. British Journal of Pharmacology. 115(6). 987–992. 56 indexed citations
7.
Hattori, Yuichi, et al.. (1988). Cardiac versus vascular effects of a new dihydropyridine derivative, CV-4093. In vitro comparison with other calcium antagonists. European Journal of Pharmacology. 146(1). 35–43. 45 indexed citations
8.
Tohse, Noritsugu, Yuichi Hattori, Haruaki Nakaya, & Morio Kanno. (1987). Effects of α-adrenoceptor stimulation on electrophysiological properties and mechanics in rat papillary muscle. General Pharmacology The Vascular System. 18(5). 539–546. 26 indexed citations
9.
Hattori, Yuichi, et al.. (1980). Effects of Calcium Antagonists and Lidocaine on Conduction Delay Induced by Acute Myocardial Ischemia in Dogs. The Japanese Journal of Pharmacology. 30(5). 587–597. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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