Yogesh Dhungana
- Immunology top 2%
- Molecular Biology top 10%
- Oncology top 5%
- Cancer Research top 5%
- Genetics
- Co-authors
- Hongbo ChiPeter VogelNicole M. ChapmanLingyun LongJordy SaraviaCliff GuyHao ShiGeoffrey Neale
- Topics
- Immune Cell Function and Interaction (15 papers)T-cell and B-cell Immunology (11 papers)CAR-T cell therapy research (4 papers)
- Cited by
- ImmunologyCancer ResearchOncology
- Journals
- NatureCellNature Communications
- Partner nations
- United StatesChinaRussia
In The Last Decade
Yogesh Dhungana
27 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 80
- Immunology 1.2k
- Molecular Biology 773
- Oncology 584
- Cancer Research 357
- Genetics 143
Countries citing papers authored by Yogesh Dhungana
This map shows the geographic impact of Yogesh Dhungana's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yogesh Dhungana with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yogesh Dhungana more than expected).
Fields of papers citing papers by Yogesh Dhungana
This network shows the impact of papers produced by Yogesh Dhungana. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yogesh Dhungana. The network helps show where Yogesh Dhungana may publish in the future.
Co-authorship network of co-authors of Yogesh Dhungana
This figure shows the co-authorship network connecting the top 25 collaborators of Yogesh Dhungana. A scholar is included among the top collaborators of Yogesh Dhungana based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yogesh Dhungana. Yogesh Dhungana is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 8 | |
| 3 | SLC38A2 and glutamine signalling in cDC1s dictate anti-tumour immunitybreakdown → | 154 |
| 4 | 10 | |
| 5 | 8 | |
| 6 | Lipid signalling enforces functional specialization of Treg cells in tumoursbreakdown → | 286 |
| 7 | 95 | |
| 8 | 2 | |
| 9 | 61 | |
| 10 | 60 | |
| 11 | 39 | |
| 12 | 61 | |
| 13 | 53 | |
| 14 | 281 | |
| 15 | 62 | |
| 16 | 87 | |
| 17 | 35 | |
| 18 | Metabolic signaling directs the reciprocal lineage decisions of αβ and γδ T cells | 14 |
| 19 | 146 | |
| 20 | 132 |
About Yogesh Dhungana
Yogesh Dhungana is a scholar working on Immunology, Oncology and Cell Biology, having authored 27 papers that have together received 1.9k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (15 papers), T-cell and B-cell Immunology (11 papers) and CAR-T cell therapy research (4 papers). The work is most often cited by research in Immunology (1.2k citations), Cancer Research (357 citations) and Oncology (584 citations). Yogesh Dhungana has collaborated with scholars based in United States, China and Russia. Frequent co-authors include Hongbo Chi, Peter Vogel, Nicole M. Chapman, Lingyun Long, Jordy Saravia, Cliff Guy, Hao Shi, Geoffrey Neale, Seon Ah Lim and Jun Wei. Their work appears in journals such as Nature, Cell and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.