Yi‐Shing Ma

1.3k total citations
24 papers, 1.0k citations indexed

About

Yi‐Shing Ma is a scholar working on Molecular Biology, Clinical Biochemistry and Physiology. According to data from OpenAlex, Yi‐Shing Ma has authored 24 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 12 papers in Clinical Biochemistry and 5 papers in Physiology. Recurrent topics in Yi‐Shing Ma's work include Mitochondrial Function and Pathology (20 papers), Metabolism and Genetic Disorders (12 papers) and ATP Synthase and ATPases Research (6 papers). Yi‐Shing Ma is often cited by papers focused on Mitochondrial Function and Pathology (20 papers), Metabolism and Genetic Disorders (12 papers) and ATP Synthase and ATPases Research (6 papers). Yi‐Shing Ma collaborates with scholars based in Taiwan, Canada and Japan. Yi‐Shing Ma's co-authors include Yau‐Huei Wei, Cheng‐Yoong Pang, Hsin‐Chen Lee, Chong‐Kuei Lii, Chen‐Ling Kuo, Haw‐Wen Chen, Chin‐San Liu, Shi‐Bei Wu, Yuting Wu and Chin‐Chang Huang and has published in prestigious journals such as Biochemical Journal, Annals of the New York Academy of Sciences and Archives of Biochemistry and Biophysics.

In The Last Decade

Yi‐Shing Ma

23 papers receiving 985 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yi‐Shing Ma Taiwan 15 709 236 180 95 87 24 1.0k
Paola Mirra Italy 19 423 0.6× 243 1.0× 235 1.3× 173 1.8× 98 1.1× 31 981
Sherine S.L. Chan United States 22 1.0k 1.5× 447 1.9× 117 0.7× 58 0.6× 64 0.7× 35 1.4k
Pascaline Clerc United States 8 789 1.1× 199 0.8× 214 1.2× 65 0.7× 129 1.5× 10 1.0k
Pavel Ješina Czechia 23 803 1.1× 405 1.7× 213 1.2× 51 0.5× 57 0.7× 53 1.3k
Orian Shirihai United States 8 884 1.2× 148 0.6× 415 2.3× 112 1.2× 228 2.6× 10 1.3k
Vicky N. Jackson United Kingdom 16 783 1.1× 393 1.7× 369 2.0× 102 1.1× 64 0.7× 21 1.2k
Lisa Tilokani United Kingdom 6 941 1.3× 198 0.8× 205 1.1× 93 1.0× 225 2.6× 6 1.2k
Cynthia G. Van Horn United States 14 636 0.9× 108 0.5× 309 1.7× 107 1.1× 140 1.6× 15 1.0k
Marta Frigeni United States 7 365 0.5× 231 1.0× 187 1.0× 52 0.5× 68 0.8× 11 769
Changlin Zhen China 9 509 0.7× 98 0.4× 173 1.0× 71 0.7× 122 1.4× 12 906

Countries citing papers authored by Yi‐Shing Ma

Since Specialization
Citations

This map shows the geographic impact of Yi‐Shing Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yi‐Shing Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yi‐Shing Ma more than expected).

Fields of papers citing papers by Yi‐Shing Ma

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yi‐Shing Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yi‐Shing Ma. The network helps show where Yi‐Shing Ma may publish in the future.

Co-authorship network of co-authors of Yi‐Shing Ma

This figure shows the co-authorship network connecting the top 25 collaborators of Yi‐Shing Ma. A scholar is included among the top collaborators of Yi‐Shing Ma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yi‐Shing Ma. Yi‐Shing Ma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Wu, Yuting, et al.. (2023). Mitochondrial impairment and synaptic dysfunction are associated with neurological defects in iPSCs-derived cortical neurons of MERRF patients. Journal of Biomedical Science. 30(1). 70–70. 11 indexed citations
3.
Ma, Yi‐Shing, et al.. (2018). Promoter analysis and transcriptional regulation of human carbonic anhydrase VIII gene in a MERRF disease cell model. Archives of Biochemistry and Biophysics. 641. 50–61. 5 indexed citations
4.
Chen, Zhen, Yue Wu, Yi‐Shing Ma, et al.. (2017). Profiling of cardiolipins and their hydroperoxides in HepG2 cells by LC/MS. Analytical and Bioanalytical Chemistry. 409(24). 5735–5745. 17 indexed citations
5.
Ma, Yi‐Shing, S. Yoshida, Yu Kobayashi, et al.. (2016). Improvement of Mitochondrial Function and Lipid Utilization by 3,5-dihydroxy-4-methoxybenzyl Alcohol, an Oyster-derived polyphenol, in Oleate-loaded C2C12 Myotubes. Journal of food and nutrition research. 4(8). 498–507. 1 indexed citations
6.
Ma, Yi‐Shing, et al.. (2013). High Glucose Modulates Antiproliferative Effect and Cytotoxicity of 5-Fluorouracil in Human Colon Cancer Cells. DNA and Cell Biology. 33(2). 64–72. 54 indexed citations
7.
Chen, Chin‐Yi, Ting‐Hui Lin, Yi‐Shing Ma, et al.. (2012). The protective roles of phosphorylated heat shock protein 27 in human cells harboring myoclonus epilepsy with ragged‐red fibers A8344G mtDNA mutation. FEBS Journal. 279(16). 2987–3001. 7 indexed citations
8.
Chen, Chin‐Yi, et al.. (2011). Decreased heat shock protein 27 expression and altered autophagy in human cells harboring A8344G mitochondrial DNA mutation. Mitochondrion. 11(5). 739–749. 19 indexed citations
9.
10.
Ma, Yi‐Shing, et al.. (2009). Response to the increase of oxidative stress and mutation of mitochondrial DNA in aging. Biochimica et Biophysica Acta (BBA) - General Subjects. 1790(10). 1021–1029. 77 indexed citations
11.
Cheng, Wen‐Ling, et al.. (2006). Alteration in the copy number of mitochondrial DNA in leukocytes of patients with mitochondrial encephalomyopathies. Acta Neurologica Scandinavica. 0(0). 2502536978–2502536978. 53 indexed citations
12.
Ma, Yi‐Shing, et al.. (2005). Upregulation of Matrix Metalloproteinase 1 and Disruption of Mitochondrial Network in Skin Fibroblasts of Patients with MERRF Syndrome. Annals of the New York Academy of Sciences. 1042(1). 55–63. 27 indexed citations
13.
Thajeb, Peterus, Yi‐Shing Ma, Chin‐Yuan Tzen, et al.. (2005). Oculopharyngeal somatic myopathy in a patient with a novel large-scale 3399bp deletion and a homoplasmic T5814C transition of the mitochondrial DNA. Clinical Neurology and Neurosurgery. 108(4). 407–410. 3 indexed citations
14.
Cheng, Wen‐Ling, et al.. (2005). High Prevalence of the COII/tRNALys Intergenic 9‐bp Deletion in Mitochondrial DNA of Taiwanese Patients with MELAS or MERRF Syndrome. Annals of the New York Academy of Sciences. 1042(1). 82–87. 9 indexed citations
15.
Liu, Chin‐San, Chen‐Ling Kuo, Haw‐Wen Chen, et al.. (2003). Oxidative Stress-related Alteration of the Copy Number of Mitochondrial DNA in Human Leukocytes. Free Radical Research. 37(12). 1307–1317. 237 indexed citations
16.
Huang, Chin‐Chang, et al.. (2002). Clinical Phenotype, Prognosis and Mitochondrial DNA Mutation Load in Mitochondrial Encephalomyopathies. Journal of Biomedical Science. 9(6). 527–533. 23 indexed citations
17.
Lim, Paik‐Seong, et al.. (2002). Mitochondrial DNA Mutations and Oxidative Damage in Skeletal Muscle of Patients with Chronic Uremia. Journal of Biomedical Science. 9(6). 549–560. 26 indexed citations
18.
Lim, Paik‐Seong, et al.. (2002). Mitochondrial DNA mutations and oxidative damage in skeletal muscle of patients with chronic uremia. Journal of Biomedical Science. 9(6). 549–560. 22 indexed citations
19.
Wei, Yau‐Huei, et al.. (2001). Increases of Mitochondrial Mass and Mitochondrial Genome in Association with Enhanced Oxidative Stress in Human Cells Harboring 4,977 BP‐Deleted Mitochondrial DNA. Annals of the New York Academy of Sciences. 928(1). 97–112. 89 indexed citations
20.
Wei, Yau‐Huei, et al.. (1998). Oxidative Damage and Mutation to Mitochondrial DNA and Age‐dependent Decline of Mitochondrial Respiratory Functiona. Annals of the New York Academy of Sciences. 854(1). 155–170. 215 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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