Yee‐Man Lau
Impact in
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- Cardiomyopathy and Myosin Studies
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- Neuroscience and Neural Engineering
Papers in
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- Nuclear Structure and Function 3
- RNA Research and Splicing 3
- Congenital heart defects research 2
- Pluripotent Stem Cells Research 2
- Mitochondrial Function and Pathology 2
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- Cardiomyopathy and Myosin Studies 4
- Cardiovascular Effects of Exercise 2
- Co-authors
- Wing‐Hon Lai (12 shared papers)Chung‐Wah Siu (11 shared papers)Kwong‐Man Ng (11 shared papers)Yee-Ki Lee (8 shared papers)Hung‐Fat Tse (8 shared papers)Yau-Chi Chan (2 shared papers)Hung‐Fat Tse (3 shared papers)Qizhou Lian (1 shared paper)
In The Last Decade
Yee‐Man Lau
15 papers receiving 628 citations
Peers
Comparison fields: 5 of 77
- Cardiology and Cardiovascular Medicine 216
- Cellular and Molecular Neuroscience 147
- Molecular Biology 476
- Cell Biology 50
- Aging 5
Countries citing papers authored by Yee‐Man Lau
This map shows the geographic impact of Yee‐Man Lau's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yee‐Man Lau with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yee‐Man Lau more than expected).
Fields of papers citing papers by Yee‐Man Lau
This network shows the impact of papers produced by Yee‐Man Lau. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yee‐Man Lau. The network helps show where Yee‐Man Lau may publish in the future.
Co-authors
The 25 scholars most cited alongside Yee‐Man Lau, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 124 | |
| 2 | 2012 | 107 | |
| 3 | 2018 | 64 | |
| 4 | 2000 | 55 | |
| 5 | 2021 | 53 | |
| 6 | 2017 | 50 | |
| 7 | 2013 | 40 | |
| 8 | 2016 | 39 | |
| 9 | 2001 | 34 | |
| 10 | 2015 | 30 | |
| 11 | 2019 | 13 | |
| 12 | 2023 | 10 | |
| 13 | 2016 | 8 | |
| 14 | 2016 | 7 | |
| 15 | 2022 | 4 |
About Yee‐Man Lau
Yee‐Man Lau is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience, Cell Biology and Surgery, having authored 15 papers that have together received 638 indexed citations. Recurring topics across this work include Cardiomyopathy and Myosin Studies (4 papers), Nuclear Structure and Function (3 papers), RNA Research and Splicing (3 papers), Genetic Neurodegenerative Diseases (2 papers), Cardiovascular Effects of Exercise (2 papers), Congenital heart defects research (2 papers), Pluripotent Stem Cells Research (2 papers) and Mitochondrial Function and Pathology (2 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (216 citations), Cellular and Molecular Neuroscience (147 citations), Molecular Biology (476 citations), Cell Biology (50 citations) and Aging (5 citations). Yee‐Man Lau has collaborated with scholars based in Hong Kong, China and Singapore. Frequent co-authors include Wing‐Hon Lai, Chung‐Wah Siu, Kwong‐Man Ng, Yee-Ki Lee, Hung‐Fat Tse, Yau-Chi Chan, Hung‐Fat Tse, Qizhou Lian, Jenny Chung-Yee Ho and Ka‐Wing Au. Their work appears in journals such as Scientific Reports, Journal of Cellular Biochemistry, International Journal of Cardiology, Pflügers Archiv - European Journal of Physiology and Aging.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.