Yan-Wei Sha

858 total citations
29 papers, 570 citations indexed

About

Yan-Wei Sha is a scholar working on Reproductive Medicine, Genetics and Molecular Biology. According to data from OpenAlex, Yan-Wei Sha has authored 29 papers receiving a total of 570 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Reproductive Medicine, 16 papers in Genetics and 11 papers in Molecular Biology. Recurrent topics in Yan-Wei Sha's work include Sperm and Testicular Function (15 papers), Reproductive Biology and Fertility (10 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (9 papers). Yan-Wei Sha is often cited by papers focused on Sperm and Testicular Function (15 papers), Reproductive Biology and Fertility (10 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (9 papers). Yan-Wei Sha collaborates with scholars based in China, United States and United Kingdom. Yan-Wei Sha's co-authors include Libin Mei, Lin Li, Zhiying Su, Ping Li, Xiaohui Xu, Lu Ding, Zhiyong Ji, Pingping Qiu, Ping Li and Xiaoqin He and has published in prestigious journals such as Development, Gene and Biology of Reproduction.

In The Last Decade

Yan-Wei Sha

28 papers receiving 565 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yan-Wei Sha China 13 385 353 283 213 69 29 570
Mariem Ben Khelifa France 7 435 1.1× 347 1.0× 351 1.2× 249 1.2× 68 1.0× 12 626
Sandra Yassine France 7 517 1.3× 291 0.8× 473 1.7× 268 1.3× 54 0.8× 7 728
Thomas Karaouzène France 9 572 1.5× 346 1.0× 519 1.8× 285 1.3× 73 1.1× 10 790
Zhiyong Ji China 15 491 1.3× 439 1.2× 360 1.3× 290 1.4× 73 1.1× 40 730
Mingrong Lv China 16 336 0.9× 231 0.7× 299 1.1× 239 1.1× 100 1.4× 37 587
Libin Mei China 17 611 1.6× 562 1.6× 475 1.7× 322 1.5× 97 1.4× 33 872
Caroline Cazin France 10 312 0.8× 278 0.8× 258 0.9× 190 0.9× 51 0.7× 20 481
Yanwei Sha China 18 608 1.6× 554 1.6× 468 1.7× 323 1.5× 123 1.8× 43 911
Can Dai China 11 209 0.5× 122 0.3× 310 1.1× 234 1.1× 94 1.4× 22 446
Shoji Kuretake Japan 8 548 1.4× 321 0.9× 591 2.1× 274 1.3× 68 1.0× 9 784

Countries citing papers authored by Yan-Wei Sha

Since Specialization
Citations

This map shows the geographic impact of Yan-Wei Sha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yan-Wei Sha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yan-Wei Sha more than expected).

Fields of papers citing papers by Yan-Wei Sha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yan-Wei Sha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yan-Wei Sha. The network helps show where Yan-Wei Sha may publish in the future.

Co-authorship network of co-authors of Yan-Wei Sha

This figure shows the co-authorship network connecting the top 25 collaborators of Yan-Wei Sha. A scholar is included among the top collaborators of Yan-Wei Sha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yan-Wei Sha. Yan-Wei Sha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Sha, Yan-Wei, et al.. (2023). Eif4enif1 haploinsufficiency disrupts oocyte mitochondrial dynamics and leads to subfertility. Development. 150(23). 11 indexed citations
3.
Li, Na, Wensheng Liu, Yan-Wei Sha, et al.. (2022). Biallelic mutations in spermatogenesis and centriole-associated 1 like (SPATC1L) cause acephalic spermatozoa syndrome and male infertility. Asian Journal of Andrology. 24(1). 67–72. 13 indexed citations
4.
Sha, Yan-Wei, Libin Mei, Zhiyong Ji, et al.. (2018). Two cases of complex balanced autosomal translocations associated with severe oligozoospermia. Gene. 663. 126–130. 3 indexed citations
5.
Sha, Yan-Wei, Xiong Wang, Zhiying Su, et al.. (2018). TDRD6 is associated with oligoasthenoteratozoospermia by sequencing the patient from a consanguineous family. Gene. 659. 84–88. 28 indexed citations
6.
Jin, Baofang, Zhiying Su, Libin Mei, et al.. (2018). Identification of a novel mutation in FGFR1 gene in patients with Kallmann syndrome by high throughput sequencing. Systems Biology in Reproductive Medicine. 64(3). 202–206. 4 indexed citations
7.
8.
Sha, Yan-Wei, Xiaohui Xu, Zhiyong Ji, et al.. (2018). Genetic contribution of SUN5 mutations to acephalic spermatozoa in Fujian China. Gene. 647. 221–225. 34 indexed citations
9.
Li, Lin, Yan-Wei Sha, Xiaohui Xu, et al.. (2018). DNAH6is a novel candidate gene associated with sperm head anomaly. Andrologia. 50(4). e12953–e12953. 46 indexed citations
10.
Jin, Lin, Yan-Wei Sha, Pingping Qiu, et al.. (2017). Effects of hepatitis B virus infection in women with different ovarian reserve on outcomes of in vitro fertilization and embryo transfer. 37(2). 106–110. 1 indexed citations
11.
Li, Lin, Yan-Wei Sha, Zhiying Su, et al.. (2017). A novel mutation in HAUS7 results in severe oligozoospermia in two brothers. Gene. 639. 106–110. 16 indexed citations
12.
Sha, Yan-Wei, Libin Mei, Xianjing Huang, et al.. (2017). Use of targeted sequence capture and high-throughput sequencing identifies a novel PKD1 mutation involved in adult polycystic kidney disease. Gene. 634. 1–4. 4 indexed citations
13.
Sha, Yan-Wei, Xiaohui Xu, Libin Mei, et al.. (2017). A homozygous CEP135 mutation is associated with multiple morphological abnormalities of the sperm flagella (MMAF). Gene. 633. 48–53. 100 indexed citations
14.
Li, Ping, et al.. (2016). Genetic factors contributing to human primary ciliary dyskinesia and male infertility. Asian Journal of Andrology. 19(5). 515–515. 20 indexed citations
15.
Li, Ping, Yan-Wei Sha, & Lu Ding. (2014). Management of primary ciliary dyskinesia/Kartagener′s syndrome in infertile male patients and current progress in defining the underlying genetic mechanism. Asian Journal of Andrology. 16(1). 101–101. 42 indexed citations
16.
Zou, Shasha, Zheng Li, Yanan Wang, et al.. (2014). Association Study Between Polymorphisms of PRMT6, PEX10, SOX5, and Nonobstructive Azoospermia in the Han Chinese Population1. Biology of Reproduction. 90(5). 96–96. 17 indexed citations
17.
Zong, Chen, Yan-Wei Sha, Huifen Xiang, et al.. (2014). Glutathione S-transferase A1 polymorphism and the risk of recurrent spontaneous abortion in Chinese Han population. Journal of Assisted Reproduction and Genetics. 31(3). 379–382. 8 indexed citations
18.
Li, Ping, et al.. (2013). Non-chimerism and chimerism pseudo dicentric Y chromosome: two case reports about azoospermia and cytogenetic/molecular genetic analysis in the Chinese population. Journal of Assisted Reproduction and Genetics. 30(4). 539–546. 3 indexed citations
19.
Sha, Yan-Wei, et al.. (2012). [Ring 22 chromosome syndrome induced azoospermia: a case report and literature review].. PubMed. 18(12). 1111–4. 1 indexed citations
20.
Wang, Wenbo, et al.. (2012). [Multiplex ligation-dependent probe amplification for detecting AZF microdeletions on the Y chromosome in infertile men with azoospermia or severe oligozoospermia].. PubMed. 18(2). 115–21. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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