Xing Deng

1.4k total citations
21 papers, 1.1k citations indexed

About

Xing Deng is a scholar working on Hepatology, Epidemiology and Molecular Biology. According to data from OpenAlex, Xing Deng has authored 21 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Hepatology, 9 papers in Epidemiology and 6 papers in Molecular Biology. Recurrent topics in Xing Deng's work include Liver physiology and pathology (10 papers), Liver Disease Diagnosis and Treatment (7 papers) and Liver Diseases and Immunity (3 papers). Xing Deng is often cited by papers focused on Liver physiology and pathology (10 papers), Liver Disease Diagnosis and Treatment (7 papers) and Liver Diseases and Immunity (3 papers). Xing Deng collaborates with scholars based in China and United States. Xing Deng's co-authors include Wei‐Fen Xie, Chuan Yin, Junping Zhang, Hongyang Wang, Jin Ding, Lijian Hui, Weiping Li, Yue-Xiang Chen, Xin Zeng and Beifang Ning and has published in prestigious journals such as The Journal of Cell Biology, Gastroenterology and Hepatology.

In The Last Decade

Xing Deng

19 papers receiving 1.1k citations

Peers

Xing Deng

HEPAT

EPIDE

SURGE

E. Zamara Italy

Saswati Hazra United States

Delia Blaya Spain

Mar Coll Spain

Steve F. Bronk United States

Seishi Nagamori Japan

Motoi Kondo Japan

Simon Weonsang Ro South Korea

Sebastião N. Martins-Filho Canada

E. Zamara Italy

Xing Deng

331 ×0.7

569 ×1.2

HEPAT

477 ×1.5

EPIDE

275 ×1.0

SURGE

212 ×1.1

Citations per year, relative to Xing Deng Xing Deng (= 1×) peers E. Zamara

Countries citing papers authored by Xing Deng

Since Specialization

Citations

This map shows the geographic impact of Xing Deng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xing Deng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xing Deng more than expected).

Fields of papers citing papers by Xing Deng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xing Deng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xing Deng. The network helps show where Xing Deng may publish in the future.

Co-authorship network of co-authors of Xing Deng

This figure shows the co-authorship network connecting the top 25 collaborators of Xing Deng. A scholar is included among the top collaborators of Xing Deng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xing Deng. Xing Deng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Liu, Shuqing, Chuan Yin, Xing Deng, et al.. (2025). SOX9 promotes hepatocyte proliferation via upregulating TGF-α expression during liver regeneration. Hepatology International.

Liu, Qingqing, Zhou Lan, Q. Kevin Fang, et al.. (2025). Nitrogen-doped porous biochar with p-π conjugated structure for efficient visible-light photocatalytic amine oxidation integrated with CO2 reduction. Applied Catalysis B: Environmental. 378. 125587–125587. 2 indexed citations

Zheng, Jianfei, et al.. (2025). TMEDs mediate versatile cargo transport in vesicle-dependent unconventional secretion. The Journal of Cell Biology. 225(1).

Chen, Wei, Xing Deng, Hong Zhang, et al.. (2024). Bile acid profiling as an effective biomarker for staging in pediatric inflammatory bowel disease. Gut Microbes. 16(1). 2323231–2323231. 14 indexed citations

Liu, Shu, et al.. (2023). Depletion of Tgfbr2 in hepatocytes alleviates liver fibrosis and restores hepatic function in fibrotic mice. Journal of Digestive Diseases. 24(1). 39–50. 3 indexed citations

Zhang, Fang, Ping Zhou, Yingping Tian, et al.. (2023). Anisodamine hydrobromide in the treatment of critically ill patients with septic shock: a multicenter randomized controlled trial. Annals of Medicine. 55(2). 2264318–2264318. 5 indexed citations

Qiu, Qiang, Minghai Tang, Fang Wang, et al.. (2019). Purinostat Mesylate Is a Uniquely Potent and Selective Inhibitor of HDACs for the Treatment of BCR-ABL –Induced B-Cell Acute Lymphoblastic Leukemia. Clinical Cancer Research. 25(24). 7527–7539. 16 indexed citations

Li, Weiping, Liguang Yang, Qiang He, et al.. (2019). A Homeostatic Arid1a-Dependent Permissive Chromatin State Licenses Hepatocyte Responsiveness to Liver-Injury-Associated YAP Signaling. Cell stem cell. 25(1). 54–68.e5. 88 indexed citations

Deng, Xing, Xin Zhang, Weiping Li, et al.. (2018). Chronic Liver Injury Induces Conversion of Biliary Epithelial Cells into Hepatocytes. Cell stem cell. 23(1). 114–122.e3. 213 indexed citations

10.

Xiang, Daimin, Wen Sun, Beifang Ning, et al.. (2017). The HLF/IL-6/STAT3 feedforward circuit drives hepatic stellate cell activation to promote liver fibrosis. Gut. 67(9). 1704–1715. 181 indexed citations

11.

Qian, Hui, Xing Deng, Chen‐Hong Ding, et al.. (2015). An HNF1α-regulated feedback circuit modulates hepatic fibrogenesis via the crosstalk between hepatocytes and hepatic stellate cells. Cell Research. 25(8). 930–945. 64 indexed citations

12.

Zhang, Jianmei, et al.. (2015). Mechanism of sophocarpine in treating experimental colitis in mice. China Journal of Chinese Materia Medica. 40(15). 3081–7. 4 indexed citations

13.

Li, Hongguang, et al.. (2015). Protection of Sophocarpine on Colonic Barrier in DSS-induced Acute Colitis in Mice by Increasing Expression of HNF4α. Chinese Herbal Medicines. 7(3). 261–266. 3 indexed citations

14.

Ning, Beifang, Jin Ding, Jiao Liu, et al.. (2014). Hepatocyte nuclear factor 4α-nuclear factor-κB feedback circuit modulates liver cancer progression. Hepatology. 60(5). 1607–1619. 87 indexed citations

15.

Xu, Wei‐Heng, Honggang Hu, Yuan Tian, et al.. (2014). Bioactive compound reveals a novel function for ribosomal protein S5 in hepatic stellate cell activation and hepatic fibrosis. Hepatology. 60(2). 648–660. 48 indexed citations

16.

Li, Jie, Xingxia Li, Wei‐Heng Xu, et al.. (2014). Antifibrotic effects of luteolin on hepatic stellate cells and liver fibrosis by targeting AKT/mTOR/p70S6K and TGFβ/Smad signalling pathways. Liver International. 35(4). 1222–1233. 106 indexed citations

17.

Sun, Wen, Jin Ding, Kun Wu, et al.. (2011). Gankyrin-mediated dedifferentiation facilitates the tumorigenicity of rat hepatocytes and hepatoma cells. Hepatology. 54(4). 1259–1272. 48 indexed citations

18.

Deng, Xing, Xin Zhang, Chuan Yin, et al.. (2008). Hepatic stellate cells modulate the differentiation of bone marrow mesenchymal stem cells into hepatocyte‐like cells. Journal of Cellular Physiology. 217(1). 138–144. 30 indexed citations

19.

Yin, Chuan, Yong Lin, Xin Zhang, et al.. (2008). Differentiation therapy of hepatocellular carcinoma in mice with recombinant adenovirus carrying hepatocyte nuclear factor-4α gene. Hepatology. 48(5). 1528–1539. 172 indexed citations

20.

Deng, Xing, Wei‐Fen Xie, Yue-Xiang Chen, & Xin Zhang. (2008). 687 Hepatic Stellate Cells Modulate the Differentiation of Bone Marrow Mesenchymal Stem Cells Into Hepatocyte-Like Cells. Gastroenterology. 134(4). A–769. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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