E. Zamara

1.4k total citations
18 papers, 1.2k citations indexed

About

E. Zamara is a scholar working on Hepatology, Epidemiology and Molecular Biology. According to data from OpenAlex, E. Zamara has authored 18 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Hepatology, 10 papers in Epidemiology and 5 papers in Molecular Biology. Recurrent topics in E. Zamara's work include Liver physiology and pathology (11 papers), Liver Disease Diagnosis and Treatment (10 papers) and Cancer, Hypoxia, and Metabolism (3 papers). E. Zamara is often cited by papers focused on Liver physiology and pathology (11 papers), Liver Disease Diagnosis and Treatment (10 papers) and Cancer, Hypoxia, and Metabolism (3 papers). E. Zamara collaborates with scholars based in Italy, United Kingdom and France. E. Zamara's co-authors include Maurizio Parola, Erica Novo, Stefania Cannito, L. Valfrè di Bonzo, Massimo Pinzani, Fabio Marra, Sebastiano Colombatto, Carlo Cravanzola, Alessandra Caligiuri and Alessandra Compagnone and has published in prestigious journals such as Journal of Biological Chemistry, Gut and Journal of Hepatology.

In The Last Decade

E. Zamara

18 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E. Zamara Italy 10 569 477 331 275 212 18 1.2k
Claudia Paternostro Italy 17 459 0.8× 525 1.1× 475 1.4× 147 0.5× 289 1.4× 23 1.3k
Elmar R. Burchardt Germany 12 630 1.1× 442 0.9× 312 0.9× 381 1.4× 77 0.4× 21 1.1k
Takahiro Ueki Japan 15 832 1.5× 323 0.7× 705 2.1× 371 1.3× 160 0.8× 23 1.7k
Mingyi Xu China 21 410 0.7× 511 1.1× 627 1.9× 163 0.6× 393 1.9× 56 1.3k
Masaharu Yamazaki Japan 22 469 0.8× 436 0.9× 453 1.4× 110 0.4× 310 1.5× 57 1.3k
Yifeng He China 20 577 1.0× 338 0.7× 445 1.3× 411 1.5× 303 1.4× 53 1.4k
Ryuichiro Sakata Japan 14 494 0.9× 321 0.7× 251 0.8× 222 0.8× 101 0.5× 23 925
Hajime Sunagozaka Japan 20 457 0.8× 401 0.8× 296 0.9× 196 0.7× 138 0.7× 42 1.1k
Xing Deng China 12 469 0.8× 326 0.7× 508 1.5× 284 1.0× 195 0.9× 21 1.1k
Hiroto Kumemura Japan 15 436 0.8× 454 1.0× 392 1.2× 115 0.4× 98 0.5× 21 1.1k

Countries citing papers authored by E. Zamara

Since Specialization
Citations

This map shows the geographic impact of E. Zamara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Zamara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Zamara more than expected).

Fields of papers citing papers by E. Zamara

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Zamara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Zamara. The network helps show where E. Zamara may publish in the future.

Co-authorship network of co-authors of E. Zamara

This figure shows the co-authorship network connecting the top 25 collaborators of E. Zamara. A scholar is included among the top collaborators of E. Zamara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. Zamara. E. Zamara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Galastri, S., E. Zamara, Stefano Milani, et al.. (2012). Lack of CC chemokine ligand 2 differentially affects inflammation and fibrosis according to the genetic background in a murine model of steatohepatitis. Clinical Science. 123(7). 459–471. 53 indexed citations
2.
Vizzutti, Francesco, A. Provenzano, S. Galastri, et al.. (2009). Curcumin limits the fibrogenic evolution of experimental steatohepatitis. Laboratory Investigation. 90(1). 104–115. 84 indexed citations
3.
Cannito, Stefania, Erica Novo, L. Valfrè di Bonzo, et al.. (2008). Multiple molecular mechanisms sustain hypoxia - dependent epithelial - mesenchymal transition and increased invasiveness in human cancer cells. American Journal Of Pathology. 173. 1 indexed citations
4.
Bandino, Andrea, Alessandra Compagnone, Carlo Cravanzola, et al.. (2008). ß-Catenin triggers nuclear factor ?B-dependent up-regulation of hepatocyte inducible nitric oxide synthase. The International Journal of Biochemistry & Cell Biology. 40(9). 1861–1871. 11 indexed citations
5.
Cannito, Stefania, Erica Novo, Alessandra Compagnone, et al.. (2008). Redox mechanisms switch on hypoxia-dependent epithelial–mesenchymal transition in cancer cells. Carcinogenesis. 29(12). 2267–2278. 252 indexed citations
6.
Cannito, Stefania, Erica Novo, L. Valfrè di Bonzo, et al.. (2008). 136 REDOX-SENSITIVE GSK-3BETA INHIBITION AND LATE VEGF RELEASE MEDIATE HYPOXIA-DEPENDENT EPITHELIAL—MESENCHYMAL TRANSITION AND INCREASED INVASIVENESS IN HUMAN HEPG2 CELLS. Journal of Hepatology. 48. S58–S59. 1 indexed citations
7.
Novo, Erica, Stefania Cannito, E. Zamara, et al.. (2007). Proangiogenic Cytokines as Hypoxia-Dependent Factors Stimulating Migration of Human Hepatic Stellate Cells. American Journal Of Pathology. 170(6). 1942–1953. 170 indexed citations
8.
Bonzo, L. Valfrè di, Ivana Ferrero, Carlo Cravanzola, et al.. (2007). Human mesenchymal stem cells as a two-edged sword in hepatic regenerative medicine: engraftment and hepatocyte differentiation versus profibrogenic potential. Gut. 57(2). 223–231. 225 indexed citations
9.
Zamara, E., S. Galastri, Sara Aleffi, et al.. (2006). Prevention of severe toxic liver injury and oxidative stress in MCP-1-deficient mice. Journal of Hepatology. 46(2). 230–238. 84 indexed citations
10.
Novo, Erica, Fabio Marra, E. Zamara, et al.. (2006). Overexpression of Bcl-2 by activated human hepatic stellate cells: resistance to apoptosis as a mechanism of progressive hepatic fibrogenesis in humans. Gut. 55(8). 1174–1182. 132 indexed citations
11.
Novo, Erica, E. Zamara, L. Valfrè di Bonzo, et al.. (2005). Migration of human activated hepatic stellate cells induced by superoxide anion: a Ras/Erk – dependent event that does not affect proliferation. United European Gastroenterology Journal. 1 indexed citations
12.
Novo, Erica, Fabio Marra, E. Zamara, et al.. (2004). 321 High levels of BCL-2, C-AKT and TRK-A in activated human hepatic stellate cells: Possible mechanisms for survival to apoptosis induction. Journal of Hepatology. 40. 98–98. 1 indexed citations
13.
Zamara, E., Erica Novo, Fabio Marra, et al.. (2003). 4-Hydroxynonenal as a selective pro-fibrogenic stimulus for activated human hepatic stellate cells. Journal of Hepatology. 40(1). 60–68. 103 indexed citations
14.
Zamara, E., Andrea Bonacchi, Erica Novo, et al.. (2003). Activated human hepatic stellate cells are resistant to classic pro-apoptotic stimuli. Journal of Hepatology. 38. 21–21. 2 indexed citations
15.
Zamara, E., Erica Novo, Gaia Robino, et al.. (2002). 4-Hydroxynonenal selectively up-regulates timp-1 gene expression inhuman hepatic stellate cells. Journal of Hepatology. 36. 76–76. 1 indexed citations
16.
Robino, Gaia, E. Zamara, Erica Novo, Umberto Dianzani, & Maurizio Parola. (2001). 4‐Hydroxy‐2,3‐alkenals as signal molecules modulating proliferative and adaptative cell responses. BioFactors. 15(2-4). 103–106. 7 indexed citations
17.
Robino, Gaia, Maurizio Parola, Fabio Marra, et al.. (2000). Interaction between 4-Hydroxy-2,3-alkenals and the Platelet-derived Growth Factor-β Receptor. Journal of Biological Chemistry. 275(51). 40561–40567. 53 indexed citations
18.
Robino, Gaia, Maurizio Parola, Fabio Marra, et al.. (2000). 4-hydroxy-2,3-alkenals impair tyrosine phosphorylation and downstream signaling of the PDGF-β receptor in human hepatic stellate cells. Journal of Hepatology. 32. 35–35. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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