Xiaofan Qiu

573 total citations
18 papers, 435 citations indexed

About

Xiaofan Qiu is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Epidemiology. According to data from OpenAlex, Xiaofan Qiu has authored 18 papers receiving a total of 435 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Cellular and Molecular Neuroscience and 4 papers in Epidemiology. Recurrent topics in Xiaofan Qiu's work include Mitochondrial Function and Pathology (7 papers), Genetic Neurodegenerative Diseases (7 papers) and Receptor Mechanisms and Signaling (4 papers). Xiaofan Qiu is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), Genetic Neurodegenerative Diseases (7 papers) and Receptor Mechanisms and Signaling (4 papers). Xiaofan Qiu collaborates with scholars based in Canada, United States and Denmark. Xiaofan Qiu's co-authors include Michael R. Hayden, Dagmar E. Ehrnhoefer, Ujendra Kumar, R.K. Somvanshi, Sonia Franciosi, Niels H. Skotte, Safia Ladha, Nicholas S. Caron, Ying Wang and Kuljeet Vaid and has published in prestigious journals such as Endocrinology, Human Molecular Genetics and Cell Death and Differentiation.

In The Last Decade

Xiaofan Qiu

17 papers receiving 431 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Xiaofan Qiu Canada 13 298 176 68 62 61 18 435
Tomoo Tokime Japan 9 269 0.9× 144 0.8× 51 0.8× 38 0.6× 61 1.0× 12 465
Zhenfeng Xu United States 11 276 0.9× 115 0.7× 62 0.9× 33 0.5× 49 0.8× 11 527
Simona S. Ghanem United States 16 225 0.8× 75 0.4× 104 1.5× 98 1.6× 94 1.5× 30 474
Theresa L. Wellman United States 10 275 0.9× 59 0.3× 25 0.4× 50 0.8× 40 0.7× 11 454
Alicia Gates United States 9 189 0.6× 85 0.5× 53 0.8× 33 0.5× 63 1.0× 10 381
Suqin Jin China 12 243 0.8× 112 0.6× 55 0.8× 48 0.8× 30 0.5× 17 363
Katie Mayne United Kingdom 8 164 0.6× 91 0.5× 19 0.3× 65 1.0× 56 0.9× 10 397
Stephanie Kleinle Germany 17 587 2.0× 117 0.7× 28 0.4× 38 0.6× 39 0.6× 27 772
Maria Grazia Rambotti Italy 15 368 1.2× 113 0.6× 20 0.3× 50 0.8× 59 1.0× 34 522
Michelle D. Ashley United States 8 229 0.8× 118 0.7× 51 0.8× 28 0.5× 20 0.3× 10 444

Countries citing papers authored by Xiaofan Qiu

Since Specialization
Citations

This map shows the geographic impact of Xiaofan Qiu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiaofan Qiu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiaofan Qiu more than expected).

Fields of papers citing papers by Xiaofan Qiu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiaofan Qiu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiaofan Qiu. The network helps show where Xiaofan Qiu may publish in the future.

Co-authorship network of co-authors of Xiaofan Qiu

This figure shows the co-authorship network connecting the top 25 collaborators of Xiaofan Qiu. A scholar is included among the top collaborators of Xiaofan Qiu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiaofan Qiu. Xiaofan Qiu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Yen, Ernest Ting-Ta, Bichoy Bahr, Yu Yao, et al.. (2022). Programmable Oscillator Implementation Using 2.5 GHz Mirror-encapsulated BAW Resonator to Achieve $\pm 20$ PPM Overall Stability. 2022 IEEE International Ultrasonics Symposium (IUS). 1–4.
2.
Skotte, Niels H., Mahmoud A. Pouladi, Dagmar E. Ehrnhoefer, et al.. (2020). Compromised IGF signaling causes caspase-6 activation in Huntington disease. Experimental Neurology. 332. 113396–113396. 8 indexed citations
3.
4.
Ehrnhoefer, Dagmar E., Dale D. O. Martin, Mandi E. Schmidt, et al.. (2018). Preventing mutant huntingtin proteolysis and intermittent fasting promote autophagy in models of Huntington disease. Acta Neuropathologica Communications. 6(1). 16–16. 56 indexed citations
5.
Ehrnhoefer, Dagmar E., Amber L. Southwell, Xiaofan Qiu, et al.. (2017). HACE1 is essential for astrocyte mitochondrial function and influences Huntington disease phenotypes in vivo. Human Molecular Genetics. 27(2). 239–253. 29 indexed citations
6.
Skotte, Niels H., Shaun S. Sanders, Roshni R. Singaraja, et al.. (2016). Palmitoylation of caspase-6 by HIP14 regulates its activation. Cell Death and Differentiation. 24(3). 433–444. 36 indexed citations
7.
Ehrnhoefer, Dagmar E., et al.. (2016). Laquinimod decreases Bax expression and reduces caspase-6 activation in neurons. Experimental Neurology. 283(Pt A). 121–128. 27 indexed citations
8.
Wong, Bibiana K. Y., Dagmar E. Ehrnhoefer, Rona K. Graham, et al.. (2015). Partial rescue of some features of Huntington Disease in the genetic absence of caspase-6 in YAC128 mice. Neurobiology of Disease. 76. 24–36. 41 indexed citations
9.
Ehrnhoefer, Dagmar E., et al.. (2015). A Huntingtin-based peptide inhibitor of caspase-6 provides protection from mutant Huntingtin-induced motor and behavioral deficits. Human Molecular Genetics. 24(9). 2604–2614. 49 indexed citations
10.
Somvanshi, R.K., Shenglong Zou, Xiaofan Qiu, & Ujendra Kumar. (2014). Somatostatin receptor-2 negatively regulates β-adrenergic receptor mediated Ca2+ dependent signaling pathways in H9c2 cells. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1843(4). 735–745. 7 indexed citations
11.
Ehrnhoefer, Dagmar E., Niels H. Skotte, Safia Ladha, et al.. (2013). p53 increases caspase-6 expression and activation in muscle tissue expressing mutant huntingtin. Human Molecular Genetics. 23(3). 717–729. 38 indexed citations
12.
Somvanshi, R.K., Xiaofan Qiu, & Ujendra Kumar. (2012). Isoproterenol induced hypertrophy and associated signaling pathways are modulated by Somatostatin in H9c2 cells. International Journal of Cardiology. 167(3). 1012–1022. 16 indexed citations
13.
Somvanshi, R.K., et al.. (2011). Receptor specific crosstalk and modulation of signaling upon heterodimerization between β1-adrenergic receptor and somatostatin receptor-5. Cellular Signalling. 23(5). 794–811. 18 indexed citations
14.
Kharmate, Geetanjali, Padmesh S. Rajput, Heather L. Watt, et al.. (2011). Role of somatostatin receptor 1 and 5 on epidermal growth factor receptor mediated signaling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1813(6). 1172–1189. 17 indexed citations
16.
Kharmate, Geetanjali, Padmesh S. Rajput, Heather L. Watt, et al.. (2010). Dissociation of Epidermal Growth Factor Receptor and ErbB2 Heterodimers in the Presence of Somatostatin Receptor 5 Modulate Signaling Pathways. Endocrinology. 152(3). 931–945. 16 indexed citations
17.
Desai, M.D., et al.. (2008). On the Multihop Performance of Synchronization Mechanisms in High Propagation Delay Networks. IEEE Transactions on Computers. 58(5). 577–590. 11 indexed citations
18.
Wang, Ying, et al.. (2005). Endothelin-1 expression in vascular adventitial fibroblasts. American Journal of Physiology-Heart and Circulatory Physiology. 290(2). H700–H708. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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