Michelle D. Ashley
- Molecular Biology
- Pathology and Forensic Medicine top 10%
- Cellular and Molecular Neuroscience
- Developmental Neuroscience top 5%
- Cancer Research
- Co-authors
- Robert MellerRoger P. SimonSimon ThompsonKōichi InoueGiuseppe PignataroDavid C. HenshallPhilip R. GafkenTheresa A. Lusardi
- Topics
- Neuroscience and Neuropharmacology Research (5 papers)Mitochondrial Function and Pathology (3 papers)Ubiquitin and proteasome pathways (3 papers)
- Cited by
- Developmental NeurosciencePathology and Forensic MedicineCellular and Molecular Neuroscience
- Partner nations
- United StatesIrelandIndia
In The Last Decade
Michelle D. Ashley
10 papers receiving 436 citations
Peers
Comparison fields: 5 of 67
- Molecular Biology 229
- Pathology and Forensic Medicine 153
- Cellular and Molecular Neuroscience 118
- Developmental Neuroscience 111
- Cancer Research 52
Countries citing papers authored by Michelle D. Ashley
This map shows the geographic impact of Michelle D. Ashley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michelle D. Ashley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michelle D. Ashley more than expected).
Fields of papers citing papers by Michelle D. Ashley
This network shows the impact of papers produced by Michelle D. Ashley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michelle D. Ashley. The network helps show where Michelle D. Ashley may publish in the future.
Co-authorship network of co-authors of Michelle D. Ashley
This figure shows the co-authorship network connecting the top 25 collaborators of Michelle D. Ashley. A scholar is included among the top collaborators of Michelle D. Ashley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michelle D. Ashley. Michelle D. Ashley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Investigating the role of the actin regulating complex ARP2/3 in rapid ischemic tolerance induced neuro-protection. | 5 |
| 2 | 50 | |
| 3 | Suppression of TNF receptor-1 signaling in an in vitro model of epileptic tolerance. | 9 |
| 4 | Rapid ischemic tolerance induced by adenosine preconditioning results in Bcl-2 interacting mediator of cell death (Bim) degradation by the proteasome. | 7 |
| 5 | 32 | |
| 6 | 9 | |
| 7 | 75 | |
| 8 | 14 | |
| 9 | 71 | |
| 10 | 172 |
About Michelle D. Ashley
Michelle D. Ashley is a scholar working on Cellular and Molecular Neuroscience, Neurology and Developmental Neuroscience, having authored 10 papers that have together received 444 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (5 papers), Mitochondrial Function and Pathology (3 papers) and Ubiquitin and proteasome pathways (3 papers). The work is most often cited by research in Developmental Neuroscience (111 citations), Pathology and Forensic Medicine (153 citations) and Cellular and Molecular Neuroscience (118 citations). Michelle D. Ashley has collaborated with scholars based in United States, Ireland and India. Frequent co-authors include Robert Meller, Roger P. Simon, Simon Thompson, Kōichi Inoue, Giuseppe Pignataro, David C. Henshall, Philip R. Gafken, Theresa A. Lusardi, Zhi-Gang Xiong and Julie A. Saugstad. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.