Xianpeng Ge

497 total citations
12 papers, 393 citations indexed

About

Xianpeng Ge is a scholar working on Molecular Biology, Rheumatology and Cell Biology. According to data from OpenAlex, Xianpeng Ge has authored 12 papers receiving a total of 393 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Rheumatology and 3 papers in Cell Biology. Recurrent topics in Xianpeng Ge's work include Osteoarthritis Treatment and Mechanisms (3 papers), Ubiquitin and proteasome pathways (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). Xianpeng Ge is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (3 papers), Ubiquitin and proteasome pathways (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). Xianpeng Ge collaborates with scholars based in China, United States and Japan. Xianpeng Ge's co-authors include Xuchen Ma, Antonios O. Aliprantis, Julia F. Charles, Juanhong Meng, Kangtao Ma, Yukiko Maeda, William M. O’Brien, Jing Yan, Ellen M. Gravallese and Chunyan Zhou and has published in prestigious journals such as Nature Communications, PLoS ONE and eLife.

In The Last Decade

Xianpeng Ge

11 papers receiving 387 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Xianpeng Ge China 9 205 153 72 70 59 12 393
Karolina Kania United Kingdom 6 163 0.8× 209 1.4× 57 0.8× 40 0.6× 37 0.6× 8 412
Menno ter Huurne Netherlands 10 271 1.3× 285 1.9× 65 0.9× 49 0.7× 54 0.9× 15 605
Antonietta Fazio Italy 10 216 1.1× 141 0.9× 48 0.7× 43 0.6× 35 0.6× 23 458
Menglei Xu China 10 193 0.9× 90 0.6× 81 1.1× 55 0.8× 24 0.4× 34 365
Chun-Wai Suen China 12 176 0.9× 159 1.0× 106 1.5× 85 1.2× 80 1.4× 13 525
Camille Huser United Kingdom 9 185 0.9× 115 0.8× 38 0.5× 51 0.7× 36 0.6× 15 353
Olfa Ghali France 7 209 1.0× 66 0.4× 67 0.9× 72 1.0× 39 0.7× 7 440
Mari Nakagawa Japan 7 263 1.3× 79 0.5× 62 0.9× 172 2.5× 33 0.6× 12 570
Holly Dupuis Canada 10 223 1.1× 155 1.0× 59 0.8× 76 1.1× 20 0.3× 15 414
Azusa Maeda United States 10 300 1.5× 87 0.6× 45 0.6× 41 0.6× 43 0.7× 11 445

Countries citing papers authored by Xianpeng Ge

Since Specialization
Citations

This map shows the geographic impact of Xianpeng Ge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xianpeng Ge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xianpeng Ge more than expected).

Fields of papers citing papers by Xianpeng Ge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xianpeng Ge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xianpeng Ge. The network helps show where Xianpeng Ge may publish in the future.

Co-authorship network of co-authors of Xianpeng Ge

This figure shows the co-authorship network connecting the top 25 collaborators of Xianpeng Ge. A scholar is included among the top collaborators of Xianpeng Ge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xianpeng Ge. Xianpeng Ge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
2.
Zhang, Fan, Yuan Wang, Chunjie Wang, et al.. (2024). The ESCRT protein CHMP5 restricts bone formation by controlling endolysosome-mitochondrion-mediated cell senescence. eLife. 13. 1 indexed citations
3.
Zhang, Fan, Yuanyuan Wang, Ying Zhao, et al.. (2023). NFATc1 marks articular cartilage progenitors and negatively determines articular chondrocyte differentiation. eLife. 12. 8 indexed citations
5.
Kim, Jung‐Min, Kwang Hwan Park, Xianpeng Ge, et al.. (2020). A RUNX2 stabilization pathway mediates physiologic and pathologic bone formation. Nature Communications. 11(1). 2289–2289. 70 indexed citations
6.
Ge, Xianpeng, Ruirui Shi, & Xuchen Ma. (2017). The secreted protein WNT5A regulates condylar chondrocyte proliferation, hypertrophy and migration. Archives of Oral Biology. 82. 171–179. 16 indexed citations
7.
Ge, Xianpeng, Kelly Tsang, Lizhi He, et al.. (2016). NFAT restricts osteochondroma formation from entheseal progenitors. JCI Insight. 1(4). e86254–e86254. 11 indexed citations
8.
Ge, Xianpeng, Susan Y. Ritter, Kelly Tsang, et al.. (2016). Sex-Specific Protection of Osteoarthritis by Deleting Cartilage Acid Protein 1. PLoS ONE. 11(7). e0159157–e0159157. 21 indexed citations
9.
O’Brien, William M., Yukiko Maeda, Jing Yan, et al.. (2016). RANK‐Independent Osteoclast Formation and Bone Erosion in Inflammatory Arthritis. Arthritis & Rheumatology. 68(12). 2889–2900. 116 indexed citations
11.
Ge, Xianpeng, Xuchen Ma, Juanhong Meng, et al.. (2009). Role of Wnt‐5A in interleukin‐1β–induced matrix metalloproteinase expression in rabbit temporomandibular joint condylar chondrocytes. Arthritis & Rheumatism. 60(9). 2714–2722. 54 indexed citations
12.
Li, Jing, Ram Sharma, Mary Artero, et al.. (1997). Inhibitory effect of Tripterygium wilfordii multiglycoside on increased glomerular albumin permeability in vitro. Nephrology Dialysis Transplantation. 12(10). 2064–2068. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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