Wolfgang Seufert
- Molecular Biology top 2%
- Cell Biology top 0.5%
- Oncology top 5%
- Plant Science top 5%
- Genetics top 10%
- Co-authors
- Stefan JentschMichael SchwabBruce FutcherKim NasmythWolfgang ZachariaeA. B. FutcherBrandt L. SchneiderBarbara Steiner
- Topics
- Fungal and yeast genetics research (16 papers)Ubiquitin and proteasome pathways (13 papers)RNA and protein synthesis mechanisms (10 papers)
- Cited by
- Cell BiologyMolecular BiologyAging
- Partner nations
- GermanyUnited StatesAustria
In The Last Decade
Wolfgang Seufert
58 papers receiving 3.9k citations
Peers
Comparison fields: 5 of 114
- Molecular Biology 3.6k
- Cell Biology 1.6k
- Oncology 572
- Plant Science 446
- Genetics 326
Countries citing papers authored by Wolfgang Seufert
This map shows the geographic impact of Wolfgang Seufert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wolfgang Seufert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wolfgang Seufert more than expected).
Fields of papers citing papers by Wolfgang Seufert
This network shows the impact of papers produced by Wolfgang Seufert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wolfgang Seufert. The network helps show where Wolfgang Seufert may publish in the future.
Co-authorship network of co-authors of Wolfgang Seufert
This figure shows the co-authorship network connecting the top 25 collaborators of Wolfgang Seufert. A scholar is included among the top collaborators of Wolfgang Seufert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wolfgang Seufert. Wolfgang Seufert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 5 | |
| 4 | 9 | |
| 5 | 15 | |
| 6 | 1 | |
| 7 | 27 | |
| 8 | 32 | |
| 9 | 70 | |
| 10 | 21 | |
| 11 | 19 | |
| 12 | 14 | |
| 13 | 30 | |
| 14 | 49 | |
| 15 | Informations- und Kommunikationswirtschaft räumlich stark konzentriert | 6 |
| 16 | Unternehmensorientierte Dienstleister: Noch Nachholbedarf bei der Anwendung moderner Informations- und Kommunikationstechnik | 1 |
| 17 | 43 | |
| 18 | 82 | |
| 19 | 5 | |
| 20 | 19 |
About Wolfgang Seufert
Wolfgang Seufert is a scholar working on Cell Biology, Molecular Biology and Pharmacology, having authored 68 papers that have together received 3.9k indexed citations. Recurring topics across this work include Fungal and yeast genetics research (16 papers), Ubiquitin and proteasome pathways (13 papers) and RNA and protein synthesis mechanisms (10 papers). The work is most often cited by research in Cell Biology (1.6k citations), Molecular Biology (3.6k citations) and Aging (54 citations). Wolfgang Seufert has collaborated with scholars based in Germany, United States and Austria. Frequent co-authors include Stefan Jentsch, Michael Schwab, Bruce Futcher, Kim Nasmyth, Wolfgang Zachariae, A. B. Futcher, Brandt L. Schneider, Barbara Steiner, Walter Messer and Qingmei Yang. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.