S.A. Whitmore

2.0k total citations
26 papers, 1.3k citations indexed

About

S.A. Whitmore is a scholar working on Molecular Biology, Genetics and Oncology. According to data from OpenAlex, S.A. Whitmore has authored 26 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 9 papers in Genetics and 4 papers in Oncology. Recurrent topics in S.A. Whitmore's work include Genomic variations and chromosomal abnormalities (6 papers), Genomics and Chromatin Dynamics (6 papers) and Genetics and Neurodevelopmental Disorders (5 papers). S.A. Whitmore is often cited by papers focused on Genomic variations and chromosomal abnormalities (6 papers), Genomics and Chromatin Dynamics (6 papers) and Genetics and Neurodevelopmental Disorders (5 papers). S.A. Whitmore collaborates with scholars based in Australia, United States and United Kingdom. S.A. Whitmore's co-authors include David F. Callen, Corinne Antignac, Olivier Gribouval, Gillian P. Bates, Marlene Attard, G Jean, Stéphanie Cherqui, William van’t Hoff, Margaret Town and M. Broyer and has published in prestigious journals such as The Lancet, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

S.A. Whitmore

26 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
S.A. Whitmore Australia 18 536 418 327 291 255 26 1.3k
Céline J. Rocca United States 14 288 0.5× 232 0.6× 184 0.6× 144 0.5× 65 0.3× 24 698
Wayne S. Stanley United States 18 685 1.3× 57 0.1× 94 0.3× 85 0.3× 150 0.6× 41 954
Lídia Feliubadaló Spain 15 463 0.9× 249 0.6× 73 0.2× 373 1.3× 375 1.5× 42 1.0k
G. Levan Sweden 25 1.1k 2.1× 103 0.2× 105 0.3× 32 0.1× 588 2.3× 99 1.8k
Marie Trudel Canada 30 1.7k 3.3× 289 0.7× 195 0.6× 30 0.1× 1.1k 4.5× 71 2.5k
Katharina Hopp United States 24 1.4k 2.7× 505 1.2× 293 0.9× 32 0.1× 1.6k 6.1× 40 2.2k
Efraim H. Rosenberg Netherlands 15 413 0.8× 96 0.2× 56 0.2× 62 0.2× 130 0.5× 27 1.1k
Hitoshi Kanno Japan 24 528 1.0× 33 0.1× 341 1.0× 59 0.2× 271 1.1× 131 1.5k
Dominique Cottreau France 18 468 0.9× 50 0.1× 210 0.6× 70 0.2× 110 0.4× 47 929
Mitchell Hoffman United States 9 441 0.8× 92 0.2× 43 0.1× 44 0.2× 355 1.4× 19 1.0k

Countries citing papers authored by S.A. Whitmore

Since Specialization
Citations

This map shows the geographic impact of S.A. Whitmore's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S.A. Whitmore with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S.A. Whitmore more than expected).

Fields of papers citing papers by S.A. Whitmore

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S.A. Whitmore. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S.A. Whitmore. The network helps show where S.A. Whitmore may publish in the future.

Co-authorship network of co-authors of S.A. Whitmore

This figure shows the co-authorship network connecting the top 25 collaborators of S.A. Whitmore. A scholar is included among the top collaborators of S.A. Whitmore based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S.A. Whitmore. S.A. Whitmore is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Savino, Michelangelo, Maria D’Apolito, Marta Centra, et al.. (1999). Characterization of Copine VII, a New Member of the Copine Family, and Its Exclusion as a Candidate in Sporadic Breast Cancers with Loss of Heterozygosity at 16q24.3. Genomics. 61(2). 219–226. 19 indexed citations
3.
Crawford, Joanna, Leonarda Ianzano, Michelangelo Savino, et al.. (1999). ThePISSLREGene: Structure, Exon Skipping, and Exclusion as Tumor Suppressor in Breast Cancer. Genomics. 56(1). 90–97. 35 indexed citations
4.
Bhalla, Kavita, Helen J. Eyre, S.A. Whitmore, Grant R. Sutherland, & David F. Callen. (1999). C16orf5, a novel proline-rich gene at 16p13.3, is highly expressed in the brain. Journal of Human Genetics. 44(6). 383–387. 4 indexed citations
5.
Town, Margaret, G Jean, Stéphanie Cherqui, et al.. (1998). A novel gene encoding an integral membrane protein is mutated in nephropathic cystinosis. Nature Genetics. 18(4). 319–324. 447 indexed citations
6.
Cavanaugh, Juleen A., David F. Callen, Susan R. Wilson, et al.. (1998). Analysis of Australian Crohn's disease pedigrees refines the localization for susceptibility to inflammatory bowel disease on chromosome 16. Annals of Human Genetics. 62(4). 291–298. 83 indexed citations
7.
Whitmore, S.A., Joanna Crawford, Sinoula Apostolou, et al.. (1998). Construction of a High-Resolution Physical and Transcription Map of Chromosome 16q24.3: A Region of Frequent Loss of Heterozygosity in Sporadic Breast Cancer. Genomics. 50(1). 1–8. 27 indexed citations
8.
Whitmore, S.A., Chatri Settasatian, Joanna Crawford, et al.. (1998). Characterization and Screening for Mutations of the Growth Arrest-Specific 11 (GAS11) andC16orf3Genes at 16q24.3 in Breast Cancer. Genomics. 52(3). 325–331. 44 indexed citations
9.
Ianzano, Leonarda, Maria D’Apolito, Marta Centra, et al.. (1997). The Genomic Organization of the Fanconi Anemia Group A (FAA) Gene. Genomics. 41(3). 309–314. 41 indexed citations
10.
Wang, Zhaoyi, Qingqing Qiu, Wolfgang Seufert, et al.. (1996). Molecular Cloning of the cDNA and Chromosome Localization of the Gene for Human Ubiquitin-conjugating Enzyme 9. Journal of Biological Chemistry. 271(40). 24811–24816. 82 indexed citations
11.
Callen, David F., Sharon Lane, H.M. Kozman, et al.. (1995). Integration of Transcript and Genetic Maps of Chromosome 16 at Near-1-Mb Resolution: Demonstration of a “Hot Spot” for Recombination at 16p12. Genomics. 29(2). 503–511. 30 indexed citations
12.
Kuss, Bryone J., Helen J. Eyre, Sharon Lane, et al.. (1994). Deletion of gene for multidrug resistance in acute myeloid leukaemia with inversion in chromosome 16: prognostic implications. The Lancet. 343(8912). 1531–1534. 88 indexed citations
13.
14.
Samani, Nilesh J., S.A. Whitmore, Michael Kaiser, et al.. (1994). Chromosomal Assignment of the Human SA Gene to 16p13.11 and Demonstration of Its Expression in the Kidney. Biochemical and Biophysical Research Communications. 199(2). 862–868. 10 indexed citations
15.
Stallings, Raymond L., S.A. Whitmore, Norman A. Doggett, & David F. Callen. (1993). Refined physical mapping of chromosome 16-specific low-abundance repetitive DNA sequences. Cytogenetic and Genome Research. 63(2). 97–101. 9 indexed citations
16.
Callen, David F., Norman A. Doggett, Raymond L. Stallings, et al.. (1992). High-resolution cytogenetic-based physical map of human chromosome 16. Genomics. 13(4). 1178–1185. 44 indexed citations
17.
Stallings, Raymond L., Norman A. Doggett, David F. Callen, et al.. (1992). Evaluation of a cosmid contig physical map of human chromosome 16. Genomics. 13(4). 1031–1039. 36 indexed citations
18.
Kozman, H.M., Ági K. Gedeon, S.A. Whitmore, et al.. (1991). Addition of MT, D16S10, D16S4, and D16S91 to the linkage map within 16q12.1-q22.1. Genomics. 11(3). 756–759. 3 indexed citations
19.
Harris, Peter C., Sinoula Apostolou, Elizabeth Baker, et al.. (1991). A refined physical map of the long arm of human chromosome 16. Genomics. 10(2). 308–312. 13 indexed citations
20.
Pritchard, Melanie, Elizabeth Baker, S.A. Whitmore, et al.. (1991). The interleukin-4 receptor gene (IL4R) maps to 16p11.2–16p12.1 in human and to the distal region of mouse chromosome 7. Genomics. 10(3). 801–806. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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