Wolfgang Erl

4.4k total citations · 1 hit paper
40 papers, 3.7k citations indexed

About

Wolfgang Erl is a scholar working on Molecular Biology, Cancer Research and Immunology and Allergy. According to data from OpenAlex, Wolfgang Erl has authored 40 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 15 papers in Cancer Research and 14 papers in Immunology and Allergy. Recurrent topics in Wolfgang Erl's work include Cell Adhesion Molecules Research (14 papers), NF-κB Signaling Pathways (9 papers) and Inflammatory mediators and NSAID effects (7 papers). Wolfgang Erl is often cited by papers focused on Cell Adhesion Molecules Research (14 papers), NF-κB Signaling Pathways (9 papers) and Inflammatory mediators and NSAID effects (7 papers). Wolfgang Erl collaborates with scholars based in Germany, United States and Sweden. Wolfgang Erl's co-authors include Christian Weber, Mihail Hristov, Angelika Pietsch, Kim S. C. Weber, Stefan Linder, H.W.L. Ziegler-Heitbrock, M Ströbel, Göran K. Hansson, Rainer de Martin and Ulrich Danesch and has published in prestigious journals such as Circulation, Blood and The Journal of Immunology.

In The Last Decade

Wolfgang Erl

39 papers receiving 3.6k citations

Hit Papers

Endothelial Progenitor Cells 2003 2026 2010 2018 2003 100 200 300 400 500

Peers

Wolfgang Erl
Thomas O. McDonald United States
D P Via United States
Farhad Parhami United States
Nichola Figg United Kingdom
Peter Libby United States
Murray C.H. Clarke United Kingdom
Thomas O. McDonald United States
Wolfgang Erl
Citations per year, relative to Wolfgang Erl Wolfgang Erl (= 1×) peers Thomas O. McDonald

Countries citing papers authored by Wolfgang Erl

Since Specialization
Citations

This map shows the geographic impact of Wolfgang Erl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wolfgang Erl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wolfgang Erl more than expected).

Fields of papers citing papers by Wolfgang Erl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wolfgang Erl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wolfgang Erl. The network helps show where Wolfgang Erl may publish in the future.

Co-authorship network of co-authors of Wolfgang Erl

This figure shows the co-authorship network connecting the top 25 collaborators of Wolfgang Erl. A scholar is included among the top collaborators of Wolfgang Erl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wolfgang Erl. Wolfgang Erl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Weber, Georg F., Florian Gaertner, Wolfgang Erl, et al.. (2006). IL-22-Mediated Tumor Growth Reduction Correlates with Inhibition of ERK1/2 and AKT Phosphorylation and Induction of Cell Cycle Arrest in the G2-M Phase. The Journal of Immunology. 177(11). 8266–8272. 70 indexed citations
2.
Erl, Wolfgang, et al.. (2006). Docosahexaenoic acid induces ciap1 mRNA and protects human endothelial cells from stress-induced apoptosis. American Journal of Physiology-Heart and Circulatory Physiology. 290(6). H2178–H2186. 19 indexed citations
3.
Erl, Wolfgang. (2005). Statin-induced Vascular Smooth Muscle Cell Apoptosis: A Possible Role in the Prevention of Restenosis?. PubMed. 5(2). 135–144. 29 indexed citations
4.
Kim, Hyo Jung, et al.. (2005). Docosahexaenoic acid induces apoptosis in proliferating human endothelial cells. Journal of Cellular Physiology. 204(3). 881–888. 49 indexed citations
5.
Zeiffer, Ute, Andreas Schober, Michael Lietz, et al.. (2004). Neointimal Smooth Muscle Cells Display a Proinflammatory Phenotype Resulting in Increased Leukocyte Recruitment Mediated by P-Selectin and Chemokines. Circulation Research. 94(6). 776–784. 92 indexed citations
6.
Erl, Wolfgang, et al.. (2003). Cyclopentenone prostaglandins induce endothelial cell apoptosis independent of the peroxisome proliferator‐activated receptor‐γ. European Journal of Immunology. 34(1). 241–250. 18 indexed citations
7.
Erl, Wolfgang. (2003). HMG-CoA reductase inhibitors induce apoptosis in neointima-derived vascular smooth muscle cells. Atherosclerosis. 169(2). 251–258. 48 indexed citations
8.
Erl, Wolfgang, et al.. (2003). Structural requirements of cyclopentenone prostaglandins to induce endothelial cell apoptosis. Biochemical and Biophysical Research Communications. 307(2). 322–326. 16 indexed citations
9.
Weber, Tobias, Min Lu, Ladislav Anděra, et al.. (2002). Vitamin E succinate is a potent novel antineoplastic agent with high selectivity and cooperativity with tumor necrosis factor-related apoptosis-inducing ligand (Apo2 ligand) in vivo.. PubMed. 8(3). 863–9. 164 indexed citations
10.
Weber, Christian & Wolfgang Erl. (2001). Modulation of vascular cell activation, function, and apoptosis: Role of antioxidants and nuclear factor-κB. Current topics in cellular regulation. 36. 217–235. 25 indexed citations
11.
Erl, Wolfgang. (2001). Transient increase of monocyte adhesion after a single dose of simvastatin. Atherosclerosis. 158(2). 491–493. 2 indexed citations
12.
Weber, Christian, Wolfgang Erl, & Kim S. C. Weber. (1999). Effects of Oxidized Low Density Lipoprotein, Lipid Mediators and Statins on Vascular Cell Interactions. Clinical Chemistry and Laboratory Medicine (CCLM). 37(3). 243–254. 40 indexed citations
13.
Erl, Wolfgang & Christian Weber. (1998). Monocytic cell adhesion to endothelial cells stimulated by oxidized low density lipoprotein is mediated by distinct endothelial ligands. Atherosclerosis. 136(2). 297–303. 79 indexed citations
14.
Weber, Christian, Wolfgang Erl, & Kim S. C. Weber. (1997). HMG-CoA Reductase Inhibitors Decrease CD11b Expression and CD11b-Dependent Adhesion of Monocytes to Endothelium and Reduce Increased Adhesiveness of Monocytes Isolated From Patients With Hypercholesterolemia. Journal of the American College of Cardiology. 30(5). 1212–1217. 342 indexed citations
15.
Pietsch, Angelika, Wolfgang Erl, & Reinhard Lorenz. (1996). Lovastatin reduces expression of the combined adhesion and scavenger receptor CD36 in human monocytic cells. Biochemical Pharmacology. 52(3). 433–439. 74 indexed citations
16.
Erl, Wolfgang. (1995). Adhesion properties of Mono Mac 6, a monocytic cell line with characteristics of mature human monocytes. Atherosclerosis. 113(1). 99–107. 29 indexed citations
17.
Weber, Christian, Emil V. Negrescu, Wolfgang Erl, et al.. (1995). Inhibitors of protein tyrosine kinase suppress TNF-stimulated induction of endothelial cell adhesion molecules. The Journal of Immunology. 155(1). 445–451. 116 indexed citations
18.
Weber, Christian & Wolfgang Erl. (1995). Enhancement of Monocyte Adhesion to Endothelial Cells by Oxidatively Modified Low-Density Lipoprotein Is Mediated by Activation of Cd11B. Biochemical and Biophysical Research Communications. 206(2). 621–628. 61 indexed citations
19.
Weber, Christian & Wolfgang Erl. (1995). Lovastatin induces differentiation of Mono Mac 6 cells. Cell Biochemistry and Function. 13(4). 273–277. 19 indexed citations
20.
Weber, Christian, et al.. (1993). Platelet Activating Factor Enhances Receptor-Operated Ca2+-Influx and Subsequent Prostacyclin Synthesis in Human Endothelial Cells. Biochemical and Biophysical Research Communications. 195(2). 874–880. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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