William Wierda

987 citations
11 papers · 599 indexed · h-index 8
Topics
Chronic Lymphocytic Leukemia Research (8 papers)Chronic Myeloid Leukemia Treatments (7 papers)Eosinophilic Disorders and Syndromes (3 papers)
Partner nations
United StatesSpainJapan

In The Last Decade

William Wierda

11 papers receiving 587 citations

Peers

William Wierda
Comparison fields: 5 of 46
  • Hematology 424
  • Genetics 359
  • Rheumatology 187
  • Public Health, Environmental and Occupational Health 159
  • Pathology and Forensic Medicine 115
Replace Hagop M. Kantarjian with:
Hagop M. Kantarjian United States
Rainer Krahl Germany
Beata Stella‐Hołowiecka Poland
Antônio Vaz de Macedo Brazil
Luis Felipe Casado Spain
D Inverardi Italy
Sabine Fürst France
M. Hoffknecht Germany
Haefaa Alchalby Germany
Guru Subramanian Guru Murthy United States
William Wierda relative to Hagop M. Kantarjian United States Hagop M. Kantarjian's profile →
Citations per field
00.5×1.5×2.4×
Hagop M. Kantarjian · 1×
Citations per year

Countries citing papers authored by William Wierda

Since Specialization
Citations

This map shows the geographic impact of William Wierda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William Wierda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William Wierda more than expected).

Fields of papers citing papers by William Wierda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William Wierda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William Wierda. The network helps show where William Wierda may publish in the future.

Co-authorship network of co-authors of William Wierda

This figure shows the co-authorship network connecting the top 25 collaborators of William Wierda. A scholar is included among the top collaborators of William Wierda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William Wierda. William Wierda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
#WorkIndexed citations
1 1
2 57
3 38
4 9
5 135
6 1
7 52
8 2
9 93
10 58
11 153

About William Wierda

William Wierda is a scholar working on Genetics, Hematology and Rheumatology, having authored 11 papers that have together received 599 indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (8 papers), Chronic Myeloid Leukemia Treatments (7 papers) and Eosinophilic Disorders and Syndromes (3 papers). The work is most often cited by research in Hematology (424 citations), Genetics (359 citations) and Rheumatology (187 citations). William Wierda has collaborated with scholars based in United States, Spain and Japan. Frequent co-authors include Jörge E. Cortes, Hagop M. Kantarjian, Stefan Faderl, Francis J. Giles, Susan O’Brien, Deborah A. Thomas, Mary Beth Rios, Guillermo Garcia‐Manero, Moshe Talpaz and Michael J. Keating. Their work appears in journals such as Journal of Clinical Oncology, Blood and Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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