William P. Miller
- Molecular Biology
- Cardiology and Cardiovascular Medicine top 10%
- Physiology top 10%
- Ophthalmology top 5%
- Surgery
- Co-authors
- Michael D. DennisRichard L. MossAllyson L. ToroAlistair J. BarberS. H. NellisPolly A. HofmannA. J. LiedtkeScot R. Kimball
- Topics
- Retinal Diseases and Treatments (11 papers)Cardiac Imaging and Diagnostics (9 papers)Cardiovascular Function and Risk Factors (8 papers)
- Partner nations
- United StatesUnited KingdomSouth Korea
In The Last Decade
William P. Miller
43 papers receiving 873 citations
Peers
Comparison fields: 5 of 88
- Molecular Biology 309
- Cardiology and Cardiovascular Medicine 245
- Physiology 195
- Ophthalmology 147
- Surgery 117
Countries citing papers authored by William P. Miller
This map shows the geographic impact of William P. Miller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William P. Miller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William P. Miller more than expected).
Fields of papers citing papers by William P. Miller
This network shows the impact of papers produced by William P. Miller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William P. Miller. The network helps show where William P. Miller may publish in the future.
Co-authorship network of co-authors of William P. Miller
This figure shows the co-authorship network connecting the top 25 collaborators of William P. Miller. A scholar is included among the top collaborators of William P. Miller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William P. Miller. William P. Miller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 2 | |
| 4 | 63 | |
| 5 | 14 | |
| 6 | 27 | |
| 7 | 14 | |
| 8 | 65 | |
| 9 | 22 | |
| 10 | 17 | |
| 11 | 20 | |
| 12 | 24 | |
| 13 | A successful field-based pharmacy program. | 1 |
| 14 | 2 | |
| 15 | 3 | |
| 16 | 1 | |
| 17 | 15 | |
| 18 | 4 | |
| 19 | 7 | |
| 20 | 7 |
About William P. Miller
William P. Miller is a scholar working on Ophthalmology, Clinical Biochemistry and Cardiology and Cardiovascular Medicine, having authored 45 papers that have together received 892 indexed citations. Recurring topics across this work include Retinal Diseases and Treatments (11 papers), Cardiac Imaging and Diagnostics (9 papers) and Cardiovascular Function and Risk Factors (8 papers). The work is most often cited by research in Ophthalmology (147 citations), Endocrine and Autonomic Systems (109 citations) and Cardiology and Cardiovascular Medicine (245 citations). William P. Miller has collaborated with scholars based in United States, United Kingdom and South Korea. Frequent co-authors include Michael D. Dennis, Richard L. Moss, Allyson L. Toro, Alistair J. Barber, S. H. Nellis, Polly A. Hofmann, A. J. Liedtke, Scot R. Kimball, Kerry S. McDonald and Joshua Moore. Their work appears in journals such as Journal of Biological Chemistry, Circulation and Circulation Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.