William E. Samsa

725 total citations
14 papers, 580 citations indexed

About

William E. Samsa is a scholar working on Molecular Biology, Oncology and Endocrine and Autonomic Systems. According to data from OpenAlex, William E. Samsa has authored 14 papers receiving a total of 580 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 4 papers in Oncology and 3 papers in Endocrine and Autonomic Systems. Recurrent topics in William E. Samsa's work include DNA Repair Mechanisms (4 papers), PARP inhibition in cancer therapy (4 papers) and Ubiquitin and proteasome pathways (3 papers). William E. Samsa is often cited by papers focused on DNA Repair Mechanisms (4 papers), PARP inhibition in cancer therapy (4 papers) and Ubiquitin and proteasome pathways (3 papers). William E. Samsa collaborates with scholars based in United States. William E. Samsa's co-authors include Roman V. Kondratov, Guang Zhou, Véronique Lefèbvre, Ronald J. Midura, Amit Vasanji, Xin Zhou, Murali K. Mamidi, Brendan Lee, Zhen Zhang and Zihua Gong and has published in prestigious journals such as Nucleic Acids Research, Oncogene and Journal of Cellular Physiology.

In The Last Decade

William E. Samsa

13 papers receiving 579 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
William E. Samsa United States	9	257	206	181	117	89	14	580
Ayumi Kodama Japan	8	121 0.5×	138 0.7×	110 0.6×	20 0.2×	34 0.4×	14	348
Rebecca R. Majewski United States	9	111 0.4×	439 2.1×	111 0.6×	31 0.3×	10 0.1×	11	697
Alanna B. Chan United States	11	307 1.2×	304 1.5×	222 1.2×	96 0.8×	3 0.0×	13	633
Amanda L. Allred United States	7	225 0.9×	210 1.0×	209 1.2×	54 0.5×	3 0.0×	8	583
Antonio J. Jiménez‐Caliani Spain	10	141 0.5×	467 2.3×	70 0.4×	14 0.1×	21 0.2×	12	696
Koichi Fujikawa Japan	13	58 0.2×	174 0.8×	87 0.5×	17 0.1×	31 0.3×	34	442
Klaas Romanino Switzerland	8	27 0.1×	889 4.3×	377 2.1×	75 0.6×	24 0.3×	8	1.1k
Sabrina Friedline United States	4	83 0.3×	291 1.4×	202 1.1×	236 2.0×	7 0.1×	4	592
Naoko Ikematsu Japan	9	46 0.2×	658 3.2×	65 0.4×	12 0.1×	33 0.4×	10	843
Hiroshi Ooka Japan	13	32 0.1×	178 0.9×	200 1.1×	104 0.9×	11 0.1×	22	523

Countries citing papers authored by William E. Samsa

Since Specialization

Citations

This map shows the geographic impact of William E. Samsa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William E. Samsa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William E. Samsa more than expected).

Fields of papers citing papers by William E. Samsa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William E. Samsa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William E. Samsa. The network helps show where William E. Samsa may publish in the future.

Co-authorship network of co-authors of William E. Samsa

This figure shows the co-authorship network connecting the top 25 collaborators of William E. Samsa. A scholar is included among the top collaborators of William E. Samsa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William E. Samsa. William E. Samsa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Zhang, Zhen, William E. Samsa, & Zihua Gong. (2024). NBS1 dePARylation by NUDT16 is critical for DNA double-strand break repair. Molecular and Cellular Biochemistry. 480(4). 2595–2609. 1 indexed citations

Zhang, Zhen, William E. Samsa, & Zihua Gong. (2024). NUDT16 regulates CtIP PARylation to dictate homologous recombination repair. Nucleic Acids Research. 52(7). 3761–3777. 9 indexed citations

Samsa, William E., Zhen Zhang, & Zihua Gong. (2024). CBFβ Regulates RUNX3 ADP‐Ribosylation to Mediate Homologous Recombination Repair. Journal of Cellular Physiology. 240(1). e31503–e31503.

Zhang, Zhen, William E. Samsa, Fan Zhang, et al.. (2023). HDGFRP3 interaction with 53BP1 promotes DNA double-strand break repair. Nucleic Acids Research. 51(5). 2238–2256. 9 indexed citations

Mamidi, Murali K., William E. Samsa, Yuqing Chen, et al.. (2021). The transcriptional cofactor Jab1/Cops5 is crucial for BMP‐mediated mouse chondrocyte differentiation by repressing p53 activity. Journal of Cellular Physiology. 236(8). 5686–5697. 4 indexed citations

Mamidi, Murali K., William E. Samsa, David Danielpour, Ricky Chan, & Guang Zhou. (2021). The transcription co-factor JAB1/COPS5, serves as a potential oncogenic hub of human chondrosarcoma cells in vitro.. American Journal of Cancer Research. 11(10). 5063–5075. 2 indexed citations

Samsa, William E., et al.. (2020). The master developmental regulator Jab1/Cops5/Csn5 is essential for proper bone growth and survival in mice. Bone. 143. 115733–115733. 3 indexed citations

Samsa, William E., Murali K. Mamidi, Alexander Miron, et al.. (2020). The crucial p53-dependent oncogenic role of JAB1 in osteosarcoma in vivo. Oncogene. 39(23). 4581–4591. 22 indexed citations

Liang, Bojian, Murali K. Mamidi, William E. Samsa, et al.. (2020). Targeted and sustained Sox9 expression in mouse hypertrophic chondrocytes causes severe and spontaneous osteoarthritis by perturbing cartilage homeostasis.. PubMed. 12(3). 1056–1069. 16 indexed citations

10.

Samsa, William E., Amit Vasanji, Ronald J. Midura, & Roman V. Kondratov. (2016). Deficiency of circadian clock protein BMAL1 in mice results in a low bone mass phenotype. Bone. 84. 194–203. 102 indexed citations

11.

Samsa, William E., et al.. (2016). Transcriptional control of chondrocyte specification and differentiation. Seminars in Cell and Developmental Biology. 62. 34–49. 118 indexed citations

12.

Samsa, William E., Xin Zhou, & Guang Zhou. (2016). Signaling pathways regulating cartilage growth plate formation and activity. Seminars in Cell and Developmental Biology. 62. 3–15. 65 indexed citations

13.

Samsa, William E., et al.. (2010). Deficiency of circadian protein CLOCK reduces lifespan and increases age-related cataract development in mice. Aging. 2(12). 936–944. 152 indexed citations

14.

Samsa, William E., et al.. (2010). Circadian regulation of cell cycle: Molecular connections between aging and the circadian clock. Annals of Medicine. 42(6). 404–415. 77 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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