William Charles Kreisl
- Biological Psychiatry top 1%
- Neurology top 0.5%
- Neuroinflammation and Neurodegeneration Mechanisms 11
- Psychiatry and Mental health top 2%
- Dementia and Cognitive Impairment Research 15
- Epilepsy research and treatment 7
- Physiology top 2%
- Alzheimer's disease research and treatments 20
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- Neuroscience and Neuropharmacology Research 5
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- Drug Transport and Resistance Mechanisms 14
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- Pharmacological Effects and Toxicity Studies 7
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- S100 Proteins and Annexins 6
William Charles Kreisl
55 papers receiving 3.0k citations
Peers
Comparison fields: 5 of 107
- Biological Psychiatry 423
- Neurology 1.1k
- Psychiatry and Mental health 617
- Physiology 886
- Cellular and Molecular Neuroscience 515
Countries citing papers authored by William Charles Kreisl
This map shows the geographic impact of William Charles Kreisl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William Charles Kreisl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William Charles Kreisl more than expected).
Fields of papers citing papers by William Charles Kreisl
This network shows the impact of papers produced by William Charles Kreisl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William Charles Kreisl. The network helps show where William Charles Kreisl may publish in the future.
Co-authorship network
The 25 scholars most cited alongside William Charles Kreisl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 0 | |
| 3 | 2023 | 1 | |
| 4 | 2021 | 19 | |
| 5 | 2021 | 4 | |
| 6 | 2021 | 41 | |
| 7 | 2021 | 16 | |
| 8 | 2020 | 52 | |
| 9 | 2019 | 34 | |
| 10 | 2019 | 9 | |
| 11 | 2017 | 25 | |
| 12 | 2017 | 234 | |
| 13 | 2017 | 18 | |
| 14 | 2016 | 37 | |
| 15 | 2016 | 132 | |
| 16 | 2013 | 274 | |
| 17 | 2010 | 295 | |
| 18 | 2009 | 17 | |
| 19 | 2009 | 42 | |
| 20 | 2008 | 47 |
About William Charles Kreisl
William Charles Kreisl is a scholar working on Biological Psychiatry, Neurology and Psychiatry and Mental health, having authored 58 papers that have together received 3.0k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (20 papers), Dementia and Cognitive Impairment Research (15 papers), Drug Transport and Resistance Mechanisms (14 papers), Neuroinflammation and Neurodegeneration Mechanisms (11 papers), Epilepsy research and treatment (7 papers), Pharmacological Effects and Toxicity Studies (7 papers), S100 Proteins and Annexins (6 papers) and Neuroscience and Neuropharmacology Research (5 papers). The work is most often cited by research in Biological Psychiatry (423 citations), Neurology (1.1k citations) and Psychiatry and Mental health (617 citations). William Charles Kreisl has collaborated with scholars based in United States, United Kingdom and Sweden. Frequent co-authors include Robert B. Innis, Victor W. Pike, Sami S. Zoghbi, Cheryl L. Morse, Martin Schain, Masahiro Fujita, Kimberly J. Jenko, Chul Hyoung Lyoo, Ioline D. Henter and Min-Jeong Kim. Their work appears in journals such as Proceedings of the National Academy of Sciences, JAMA and NeuroImage.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.